What is he best known for?
The fields of study he is best known for:
His main research concerns Pathology, Molecular biology, Biochemistry, Alpha-synuclein and Phosphorylation.
His Molecular biology study integrates concerns from other disciplines, such as Epitope, Exon, Cytoplasmic inclusion and Proteinase K.
His Biochemistry research includes elements of Tau protein and Cell biology.
His studies deal with areas such as Fibril, Dementia with Lewy bodies, Human brain, Genetically modified mouse and Atrophy as well as Alpha-synuclein.
His Dementia with Lewy bodies research includes themes of Immunoelectron microscopy and Synuclein.
In his study, Activator and Downregulation and upregulation is strongly linked to Kinase, which falls under the umbrella field of Phosphorylation.
His most cited work include:
- α-Synuclein in filamentous inclusions of Lewy bodies from Parkinson’s disease and dementia with Lewy bodies (2244 citations)
- TDP-43 is a component of ubiquitin-positive tau-negative inclusions in frontotemporal lobar degeneration and amyotrophic lateral sclerosis (1717 citations)
- Assembly of microtubule-associated protein tau into Alzheimer-like filaments induced by sulphated glycosaminoglycans (755 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Pathology, Cell biology, Phosphorylation, Frontotemporal lobar degeneration and Biochemistry.
His study in Pathology concentrates on Tauopathy, Corticobasal degeneration, Progressive supranuclear palsy, Dementia and Dementia with Lewy bodies.
His Dementia with Lewy bodies research incorporates themes from α synuclein, Synucleinopathies and Atrophy.
He combines subjects such as Fibril, In vitro, Ubiquitin and Alpha-synuclein with his study of Cell biology.
His Phosphorylation research incorporates elements of Molecular biology and Antibody.
His Frontotemporal lobar degeneration study also includes
- Amyotrophic lateral sclerosis and related Cytoplasmic inclusion,
- Pathological which intersects with area such as Disease.
He most often published in these fields:
- Pathology (37.14%)
- Cell biology (26.35%)
- Phosphorylation (19.68%)
What were the highlights of his more recent work (between 2017-2021)?
- Cell biology (26.35%)
- Pathology (37.14%)
- Tauopathy (12.38%)
In recent papers he was focusing on the following fields of study:
Masato Hasegawa mainly focuses on Cell biology, Pathology, Tauopathy, Neuroscience and Neurodegeneration.
His research integrates issues of In vitro, α synuclein, Pathogenesis and Alpha-synuclein in his study of Cell biology.
His Pathology study is mostly concerned with Frontotemporal lobar degeneration, Autopsy, Pathological, Substantia nigra and Gliosis.
His studies in Tauopathy integrate themes in fields like Progressive supranuclear palsy, Corticobasal degeneration, Dementia, Parkinsonism and Gene isoform.
His study explores the link between Neuroscience and topics such as Amyotrophic lateral sclerosis that cross with problems in Cerebellum and Tau pathology.
His Protein aggregation research is multidisciplinary, relying on both Fibril, Ubiquitin and Phosphorylation.
Between 2017 and 2021, his most popular works were:
- Reconsideration of Amyloid Hypothesis and Tau Hypothesis in Alzheimer's Disease (248 citations)
- Novel tau filament fold in corticobasal degeneration (100 citations)
- Structures of α-synuclein filaments from multiple system atrophy. (88 citations)
In his most recent research, the most cited papers focused on:
Cell biology, Neurodegeneration, Protein aggregation, In vitro and Neuroscience are his primary areas of study.
His Cell biology research is multidisciplinary, incorporating perspectives in Cell culture, Dopaminergic, Neurotransmission and Alpha-synuclein.
His research in Neurodegeneration tackles topics such as Phenotype which are related to areas like Axoplasmic transport, Frontotemporal lobar degeneration, Amyloid, Amyotrophic lateral sclerosis and Parkinson's disease.
His In vitro research includes themes of Fibril and In vivo.
His research integrates issues of Ubiquitin, Amyloid β, Disease, Biochemistry of Alzheimer's disease and Amyloidosis in his study of Neuroscience.
Dementia is a subfield of Pathology that Masato Hasegawa explores.
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