What is he best known for?
The fields of study he is best known for:
Dieter Edbauer spends much of his time researching C9orf72, C9orf72 Protein, Amyotrophic lateral sclerosis, Neurodegeneration and Molecular biology.
As a part of the same scientific family, Dieter Edbauer mostly works in the field of C9orf72, focusing on Genetics and, on occasion, DNA Repeat Expansion and Frontotemporal lobar degeneration.
Dieter Edbauer regularly links together related areas like Frontotemporal dementia in his Amyotrophic lateral sclerosis studies.
His Neurodegeneration research is multidisciplinary, incorporating elements of Microtubule-associated protein, Loss function and Signal transducing adaptor protein.
Dieter Edbauer combines subjects such as Gene and Presenilin with his study of Molecular biology.
His work in Presenilin addresses issues such as Protein structure, which are connected to fields such as Cell biology.
His most cited work include:
- The C9orf72 GGGGCC Repeat Is Translated into Aggregating Dipeptide-Repeat Proteins in FTLD/ALS (815 citations)
- The C9orf72 GGGGCC Repeat Is Translated into Aggregating Dipeptide-Repeat Proteins in FTLD/ALS (815 citations)
- Reconstitution of γ-secretase activity (783 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in C9orf72, Cell biology, Amyotrophic lateral sclerosis, Neurodegeneration and C9orf72 Protein.
His work deals with themes such as DNA Repeat Expansion, Molecular biology and Genetics, which intersect with C9orf72.
His Cell biology study integrates concerns from other disciplines, such as Biochemistry, Gene knockdown and Downregulation and upregulation.
The Amyotrophic lateral sclerosis study which covers Frontotemporal lobar degeneration that intersects with Cancer research.
His work investigates the relationship between Neurodegeneration and topics such as Neuroscience that intersect with problems in miR-132 and Epigenetics.
Dieter Edbauer interconnects RNA and Haploinsufficiency in the investigation of issues within C9orf72 Protein.
He most often published in these fields:
- C9orf72 (72.59%)
- Cell biology (54.07%)
- Amyotrophic lateral sclerosis (47.41%)
What were the highlights of his more recent work (between 2017-2021)?
- C9orf72 (72.59%)
- Cell biology (54.07%)
- Amyotrophic lateral sclerosis (47.41%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in C9orf72, Cell biology, Amyotrophic lateral sclerosis, Neurodegeneration and Frontotemporal dementia.
His studies deal with areas such as Pathogenesis, Mutation, Hsp70, Axon and C9orf72 Protein as well as C9orf72.
His studies examine the connections between Cell biology and genetics, as well as such issues in Translation, with regards to Ribosome, Nucleolus, Protein biosynthesis and Ribosome biogenesis.
His Amyotrophic lateral sclerosis study incorporates themes from RNA, Transgene and Heat shock protein.
The various areas that he examines in his Neurodegeneration study include Endocrinology, Skeletal muscle, Ubiquitin, Neuroinflammation and Antibody.
His Frontotemporal dementia research includes themes of Treatment strategy, Motor neuron, Neurite and Neurotransmission.
Between 2017 and 2021, his most popular works were:
- In Situ Structure of Neuronal C9orf72 Poly-GA Aggregates Reveals Proteasome Recruitment. (157 citations)
- In Situ Structure of Neuronal C9orf72 Poly-GA Aggregates Reveals Proteasome Recruitment. (157 citations)
- Loss of TREM2 function increases amyloid seeding but reduces plaque-associated ApoE. (140 citations)
In his most recent research, the most cited papers focused on:
Dieter Edbauer mostly deals with Cell biology, C9orf72, Translation, Mutation and Cytoplasm.
His Cell biology research is multidisciplinary, incorporating perspectives in Stress granule, Downregulation and upregulation and Gene knockdown.
His C9orf72 research includes elements of Frontotemporal lobar degeneration and Whole genome sequencing.
His research integrates issues of Protein aggregation, Frontotemporal dementia, Amyotrophic lateral sclerosis, Proteasome and Proteostasis in his study of Translation.
His Mutation research incorporates themes from Protein isoform, Protein family, Haploinsufficiency, C9orf72 Protein and Gene isoform.
Many of his research projects under Cytoplasm are closely connected to NPM1 with NPM1, tying the diverse disciplines of science together.
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