D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Molecular Biology D-index 60 Citations 20,482 114 World Ranking 1287 National Ranking 660

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • DNA
  • Gene expression

Jennifer L. Martindale mostly deals with Molecular biology, Cell biology, RNA-binding protein, AU-rich element and Protein biosynthesis. Her biological study deals with issues like Translation, which deal with fields such as Polysome. Her studies deal with areas such as Apoptosis, Programmed cell death and microRNA as well as Cell biology.

She interconnects Oxidative stress, Reactive oxygen species and Signal transduction in the investigation of issues within Programmed cell death. Jennifer L. Martindale has included themes like Regulation of gene expression, Ubiquitin, Transcription and Untranslated region in her RNA-binding protein study. Her work in AU-rich element covers topics such as Gene expression which are related to areas like Effector, Adipocyte and Endocrinology.

Her most cited work include:

  • Cellular response to oxidative stress: signaling for suicide and survival. (1849 citations)
  • Gadd153 Sensitizes Cells to Endoplasmic Reticulum Stress by Down-Regulating Bcl2 and Perturbing the Cellular Redox State (1530 citations)
  • The cellular response to oxidative stress: influences of mitogen-activated protein kinase signalling pathways on cell survival (685 citations)

What are the main themes of her work throughout her whole career to date?

Jennifer L. Martindale mainly focuses on Cell biology, Molecular biology, RNA-binding protein, Messenger RNA and Gene silencing. Her Cell biology study combines topics from a wide range of disciplines, such as RNA, Cell and microRNA. Jennifer L. Martindale focuses mostly in the field of RNA, narrowing it down to topics relating to Mitochondrion and, in certain cases, Oxidative stress.

Her work deals with themes such as Translation, Three prime untranslated region, Untranslated region and Polysome, which intersect with Molecular biology. Her study in RNA-binding protein is interdisciplinary in nature, drawing from both Regulation of gene expression, Gene expression and RNA interference. Her Messenger RNA research is multidisciplinary, incorporating elements of RNA splicing and Bioinformatics.

She most often published in these fields:

  • Cell biology (51.82%)
  • Molecular biology (50.91%)
  • RNA-binding protein (37.27%)

What were the highlights of her more recent work (between 2017-2021)?

  • Cell biology (51.82%)
  • Gene silencing (25.45%)
  • Gene expression (19.09%)

In recent papers she was focusing on the following fields of study:

Her scientific interests lie mostly in Cell biology, Gene silencing, Gene expression, RNA-binding protein and Messenger RNA. Her biological study spans a wide range of topics, including Cell, Transcription factor, microRNA and Cell growth. Her microRNA study also includes

  • Polyadenylation which connect with Protein biosynthesis,
  • Three prime untranslated region that connect with fields like Cellular differentiation.

Her research integrates issues of CCL2, Immune system, Antigen and Tumor necrosis factor alpha in her study of Gene silencing. Her RNA-binding protein research incorporates elements of Translation, Insulin and Autophagy, Autophagosome formation. Her Messenger RNA research includes elements of Biophysics, Distribution and Polysome.

Between 2017 and 2021, her most popular works were:

  • SCAMP4 enhances the senescent cell secretome. (18 citations)
  • circSamd4 represses myogenic transcriptional activity of PUR proteins (16 citations)
  • A small protein encoded by a putative lncRNA regulates apoptosis and tumorigenicity in human colorectal cancer cells. (10 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Gene expression

Jennifer L. Martindale spends much of her time researching Cell biology, RNA, Gene silencing, Transcription and Phenotype. Her work carried out in the field of Cell biology brings together such families of science as Polyadenylation and Oxidative stress. While the research belongs to areas of Polyadenylation, Jennifer L. Martindale spends her time largely on the problem of Protein biosynthesis, intersecting her research to questions surrounding Untranslated region.

The various areas that Jennifer L. Martindale examines in her Oxidative stress study include DNA damage, Reactive oxygen species, Proinflammatory cytokine, PI3K/AKT/mTOR pathway and Mitochondrion. Her Phenotype research is multidisciplinary, incorporating perspectives in Interleukin 6, Secretion, Interleukin 8 and Chemokine, CXCL1. Jennifer L. Martindale works mostly in the field of Endoplasmic reticulum, limiting it down to topics relating to Polysome and, in certain cases, Gene expression and Carcinogenesis, as a part of the same area of interest.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Cellular response to oxidative stress: signaling for suicide and survival.

Jennifer L. Martindale;Nikki J. Holbrook;Nikki J. Holbrook.
Journal of Cellular Physiology (2002)

3152 Citations

Gadd153 Sensitizes Cells to Endoplasmic Reticulum Stress by Down-Regulating Bcl2 and Perturbing the Cellular Redox State

Karen D. McCullough;Jennifer L. Martindale;Lars-Oliver Klotz;Tak-Yee Aw.
Molecular and Cellular Biology (2001)

2184 Citations

The cellular response to oxidative stress: influences of mitogen-activated protein kinase signalling pathways on cell survival

Xiantao Wang;Jennifer L. Martindale;Yusen Liu;Nikki J. Holbrook.
Biochemical Journal (1998)

1081 Citations

LincRNA-p21 Suppresses Target mRNA Translation

Je Hyun Yoon;Kotb Abdelmohsen;Subramanya Srikantan;Xiaoling Yang.
Molecular Cell (2012)

946 Citations

Requirement for ERK Activation in Cisplatin-induced Apoptosis *

Xiantao Wang;Jennifer L. Martindale;Nikki J. Holbrook.
Journal of Biological Chemistry (2000)

895 Citations

HuR recruits let-7/RISC to repress c-Myc expression

Hyeon Ho Kim;Yuki Kuwano;Subramanya Srikantan;Eun Kyung Lee.
Genes & Development (2009)

625 Citations

Complexes containing activating transcription factor (ATF)/cAMP-responsive-element-binding protein (CREB) interact with the CCAAT/enhancer-binding protein (C/EBP)-ATF composite site to regulate Gadd153 expression during the stress response.

Timothy W. Fawcett;Jennifer L. Martindale;Kathryn Z. Guyton;Tsonwin Hai.
Biochemical Journal (1999)

600 Citations

Identification of HuR target circular RNAs uncovers suppression of PABPN1 translation by CircPABPN1.

Kotb Abdelmohsen;Amaresh C. Panda;Rachel Munk;Ioannis Grammatikakis.
RNA Biology (2017)

572 Citations

Concurrent versus individual binding of HuR and AUF1 to common labile target mRNAs

Ashish Lal;Krystyna Mazan-Mamczarz;Tomoko Kawai;Xiaoling Yang.
The EMBO Journal (2004)

563 Citations

RNA-binding protein HuR enhances p53 translation in response to ultraviolet light irradiation

Krystyna Mazan-Mamczarz;Stefanie Galbán;Isabel López de Silanes;Jennifer L. Martindale.
Proceedings of the National Academy of Sciences of the United States of America (2003)

524 Citations

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