Nikki J. Holbrook mainly investigates Cell biology, Molecular biology, Kinase, Programmed cell death and Apoptosis. Her study looks at the relationship between Cell biology and topics such as Oxidative stress, which overlap with Antioxidant. Her work is dedicated to discovering how Antioxidant, Oxidative phosphorylation are connected with Immunology and other disciplines.
Her Molecular biology research is multidisciplinary, incorporating perspectives in Cytoplasm, DNA damage, Interleukin 1 receptor, type II, Regulation of gene expression and ELAV-Like Protein 1. Her study looks at the relationship between Programmed cell death and fields such as Reactive oxygen species, as well as how they intersect with chemical problems. Her studies deal with areas such as Cell culture, Ectopic expression, Cancer research, Cell growth and Cell type as well as Apoptosis.
Her primary areas of study are Cell biology, Molecular biology, Kinase, Internal medicine and Endocrinology. Her Cell biology research integrates issues from Apoptosis, Programmed cell death and Gene expression. The study incorporates disciplines such as DNA damage and Transcription, Regulation of gene expression, Messenger RNA, Gene in addition to Molecular biology.
Her Kinase research incorporates elements of Cell growth and Phosphorylation. Her research in Oxidative stress and Angiotensin II are components of Endocrinology. Her study in Oxidative stress is interdisciplinary in nature, drawing from both Reactive oxygen species, Oxidative phosphorylation and Antioxidant.
Nikki J. Holbrook mainly focuses on Cell biology, Phosphorylation, Oxidative stress, Kinase and Signal transduction. Her research integrates issues of Apoptosis, Programmed cell death, c-jun and Molecular biology in her study of Cell biology. Her Oxidative stress research is multidisciplinary, relying on both Reactive oxygen species and Immunology.
The various areas that Nikki J. Holbrook examines in her Reactive oxygen species study include Oxidative phosphorylation and Antioxidant. Her work carried out in the field of Kinase brings together such families of science as Luciferase, Cytoplasm, Cell growth and Blot. Her research in Signal transduction intersects with topics in Cell and Cancer research.
Her primary scientific interests are in Cell biology, Programmed cell death, Reactive oxygen species, Oxidative stress and Antioxidant. Her work deals with themes such as Apoptosis and Cell culture, which intersect with Cell biology. Her Programmed cell death study incorporates themes from PI3K/AKT/mTOR pathway, Signal transduction and Growth factor.
Her Reactive oxygen species study integrates concerns from other disciplines, such as Metabolism, Molecular oxygen and Ageing. Her Antioxidant research is multidisciplinary, incorporating elements of Cell, Oxidative phosphorylation, Proto-Oncogene Proteins c-akt and Immunology. Her studies in Phosphorylation integrate themes in fields like Molecular biology and Epidermal growth factor receptor.
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Oxidants, oxidative stress and the biology of ageing.
Toren Finkel;Nikki J. Holbrook.
Cellular response to oxidative stress: signaling for suicide and survival.
Jennifer L. Martindale;Nikki J. Holbrook;Nikki J. Holbrook.
Journal of Cellular Physiology (2002)
Gadd153 Sensitizes Cells to Endoplasmic Reticulum Stress by Down-Regulating Bcl2 and Perturbing the Cellular Redox State
Karen D. McCullough;Jennifer L. Martindale;Lars-Oliver Klotz;Tak-Yee Aw.
Molecular and Cellular Biology (2001)
Activation of Mitogen-activated Protein Kinase by H2O2: ROLE IN CELL SURVIVAL FOLLOWING OXIDANT INJURY (∗)
Kathryn Z. Guyton;Yusen Liu;Myriam Gorospe;Qingbo Xu.
Journal of Biological Chemistry (1996)
Handbook of the Biology of Aging
Edward L. Schneider;John W. Rowe;Thomas E. Johnson;Nikki J. Holbrook.
The cellular response to oxidative stress: influences of mitogen-activated protein kinase signalling pathways on cell survival
Xiantao Wang;Jennifer L. Martindale;Yusen Liu;Nikki J. Holbrook.
Biochemical Journal (1998)
Mammalian genes coordinately regulated by growth arrest signals and DNA-damaging agents.
A J Fornace;D W Nebert;M C Hollander;J D Luethy.
Molecular and Cellular Biology (1989)
Requirement for ERK Activation in Cisplatin-induced Apoptosis *
Xiantao Wang;Jennifer L. Martindale;Nikki J. Holbrook.
Journal of Biological Chemistry (2000)
HuR regulates p21 mRNA stabilization by UV light.
Wengong Wang;Henry Furneaux;Huiming Cheng;M. Craig Caldwell.
Molecular and Cellular Biology (2000)
Complexes containing activating transcription factor (ATF)/cAMP-responsive-element-binding protein (CREB) interact with the CCAAT/enhancer-binding protein (C/EBP)-ATF composite site to regulate Gadd153 expression during the stress response.
Timothy W. Fawcett;Jennifer L. Martindale;Kathryn Z. Guyton;Tsonwin Hai.
Biochemical Journal (1999)
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