What is she best known for?
The fields of study she is best known for:
Her scientific interests lie mostly in Immunology, Cytotoxic T cell, Molecular biology, Immune system and Interleukin 21.
Her Immunology research is multidisciplinary, incorporating elements of Carcinogenesis, Intestinal mucosa and Cell biology.
Hilde Cheroutre interconnects MHC class I and CD8 in the investigation of issues within Cell biology.
Her Cytotoxic T cell research is multidisciplinary, incorporating perspectives in T cell, Cancer and Major histocompatibility complex.
Her Immune system study integrates concerns from other disciplines, such as Proinflammatory cytokine, Cytokine and Evolutionary biology.
The various areas that Hilde Cheroutre examines in her Interleukin 21 study include CD28, FOXP3 and Antigen-presenting cell.
Her most cited work include:
- Reciprocal TH17 and regulatory T cell differentiation mediated by retinoic acid. (1617 citations)
- IL-6 and Stat3 Are Required for Survival of Intestinal Epithelial Cells and Development of Colitis-Associated Cancer (1523 citations)
- Adenoma-linked barrier defects and microbial products drive IL-23/IL-17-mediated tumour growth. (767 citations)
What are the main themes of her work throughout her whole career to date?
Her primary areas of study are Immunology, Cell biology, T cell, Antigen and Immune system.
Her research related to Intraepithelial lymphocyte, CD8, T-cell receptor, MHC class I and Effector might be considered part of Immunology.
Her Cell biology research is multidisciplinary, relying on both Cellular differentiation, Receptor, Cytotoxic T cell, Major histocompatibility complex and Retinoic acid.
Her work on Cytokine expands to the thematically related T cell.
Her research in Antigen intersects with topics in Molecular biology, Leukemia, Signal transduction and Inflammatory bowel disease.
Her Immune system research incorporates themes from Proinflammatory cytokine, Intestinal mucosa and Lymphocyte.
She most often published in these fields:
- Immunology (53.90%)
- Cell biology (34.75%)
- T cell (22.70%)
What were the highlights of her more recent work (between 2012-2020)?
- Cell biology (34.75%)
- Immunology (53.90%)
- Cytotoxic T cell (17.73%)
In recent papers she was focusing on the following fields of study:
Her primary scientific interests are in Cell biology, Immunology, Cytotoxic T cell, T cell and Immune system.
Her Cell biology study combines topics from a wide range of disciplines, such as Agonist, Innate lymphoid cell, Intraepithelial lymphocyte and T-cell receptor.
Her biological study deals with issues like Receptor, which deal with fields such as Signal transduction.
The T cell study combines topics in areas such as CD8 and Cellular differentiation.
The concepts of her Immune system study are interwoven with issues in Nuclear receptor, Antibody, Retinoic acid and Lymphocyte.
Her studies examine the connections between Antigen-presenting cell and genetics, as well as such issues in Interleukin 21, with regards to Natural killer T cell.
Between 2012 and 2020, her most popular works were:
- Transcriptional reprogramming of mature CD4+ helper T cells generates distinct MHC class II-restricted cytotoxic T lymphocytes (196 citations)
- T Cell-Derived Protein S Engages TAM Receptor Signaling in Dendritic Cells to Control the Magnitude of the Immune Response (111 citations)
- Critical Role of Glucose Metabolism in Rheumatoid Arthritis Fibroblast‐like Synoviocytes (95 citations)
In her most recent research, the most cited papers focused on:
Hilde Cheroutre focuses on Immunology, Cell biology, T cell, Major histocompatibility complex and Innate immune system.
Her research links Macrophage with Immunology.
Her study brings together the fields of Receptor and Cell biology.
Her Major histocompatibility complex research integrates issues from Cytotoxic T cell and T-cell receptor.
Her studies deal with areas such as Acquired immune system, Interleukin 23, Cytokine and Immunity as well as Innate immune system.
Her Immune system research incorporates elements of Nuclear receptor, Intestinal mucosa and Tretinoin.
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