2015 - Member of the National Academy of Sciences
2012 - Fellow of the American Association for the Advancement of Science (AAAS)
Hao Wu focuses on Cell biology, Signal transduction, Molecular biology, Biochemistry and Protein structure. His research investigates the connection between Cell biology and topics such as Innate immune system that intersect with issues in Acquired immune system. The concepts of his Signal transduction study are interwoven with issues in TRAF4, Receptor, MATH domain and TRAF2.
The various areas that Hao Wu examines in his Molecular biology study include Cell culture, Regulation of gene expression, KDM1A and Supervillin. As part of one scientific family, Hao Wu deals mainly with the area of Biochemistry, narrowing it down to issues related to the Biophysics, and often Short linear motif. His biological study spans a wide range of topics, including Peptide sequence, Protein subunit, Plasma protein binding and CARD Signaling Adaptor Proteins.
Hao Wu mainly focuses on Cell biology, Biochemistry, Signal transduction, Biophysics and Caspase. His research integrates issues of Pyroptosis, Receptor, Inflammasome and Innate immune system in his study of Cell biology. His study in Inflammasome is interdisciplinary in nature, drawing from both Protein filament and Cytosol.
Hao Wu works in the field of Biochemistry, namely Protein structure. His research on Signal transduction frequently connects to adjacent areas such as Plasma protein binding. His Biophysics study frequently draws connections to other fields, such as Protein subunit.
His primary areas of study are Cell biology, Inflammasome, Biophysics, Pyroptosis and Cancer research. Hao Wu integrates many fields, such as Cell biology and Small nuclear RNA, in his works. His Inflammasome research is multidisciplinary, incorporating perspectives in Caspase, NLRP1 and Insulin resistance.
When carried out as part of a general Biophysics research project, his work on Cryo-electron microscopy is frequently linked to work in Mechanism, therefore connecting diverse disciplines of study. As part of the same scientific family, he usually focuses on Pyroptosis, concentrating on Immune system and intersecting with Effector. His work carried out in the field of Cancer research brings together such families of science as Apoptosis, Downregulation and upregulation, Ibrutinib, Cytotoxic T cell and Immunotherapy.
Hao Wu mainly investigates Cell biology, Inflammasome, Pyroptosis, Caspase and Biophysics. His studies in Cell biology integrate themes in fields like Inflammation, RNA splicing, Epitope, Innate immune system and Methyltransferase. His biological study deals with issues like Insulin resistance, which deal with fields such as Microbiome and Gut flora.
His Pyroptosis study deals with Immune system intersecting with Effector, Cancer cell and Flow cytometry. His research in Caspase intersects with topics in Transport protein, Pyrin domain, Cytokine and Pharmacology. His studies deal with areas such as Mutant, NAD+ kinase, Enzyme, Peptide and Intramolecular force as well as Biophysics.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
DNA methylation-related chromatin remodeling in activity-dependent BDNF gene regulation.
Keri Martinowich;Daisuke Hattori;Hao Wu;Shaun Fouse.
Science (2003)
ABAD directly links Abeta to mitochondrial toxicity in Alzheimer's disease.
Joyce W. Lustbader;Maurizio Cirilli;Chang Lin;Hong Wei Xu;Hong Wei Xu.
Science (2004)
Inflammasome-activated gasdermin D causes pyroptosis by forming membrane pores
Xing Liu;Zhibin Zhang;Zhibin Zhang;Jianbin Ruan;Jianbin Ruan;Youdong Pan.
Nature (2016)
Helical assembly in the MyD88–IRAK4–IRAK2 complex in TLR/IL-1R signalling
Su Chang Lin;Yu Chih Lo;Hao Wu.
Nature (2010)
Reversing DNA Methylation: Mechanisms, Genomics, and Biological Functions
Hao Wu;Yi Zhang.
Cell (2014)
The RIP1/RIP3 Necrosome Forms a Functional Amyloid Signaling Complex Required for Programmed Necrosis
Jixi Li;Thomas McQuade;Ansgar B. Siemer;Johanna Napetschnig.
Cell (2012)
Unified Polymerization Mechanism for the Assembly of ASC-Dependent Inflammasomes
Alvin Lu;Alvin Lu;Venkat Giri Magupalli;Venkat Giri Magupalli;Jianbin Ruan;Jianbin Ruan;Qian Yin;Qian Yin.
Cell (2014)
Molecular Basis of NF-κB Signaling
Johanna Napetschnig;Hao Wu.
Annual Review of Biophysics (2013)
Distinct molecular mechanism for initiating TRAF6 signalling.
Hong Ye;Joseph R. Arron;Joseph R. Arron;Betty Lamothe;Maurizio Cirilli.
Nature (2002)
All TRAFs are not created equal: common and distinct molecular mechanisms of TRAF-mediated signal transduction
Jee Y. Chung;Young Chul Park;Hong Ye;Hao Wu.
Journal of Cell Science (2002)
Boston Children's Hospital
Chinese Academy of Sciences
University of California, Los Angeles
University of Gothenburg
Harvard University
City University of Hong Kong
Cornell University
Columbia University
University of Utah
Chinese Academy of Sciences
Profile was last updated on December 6th, 2021.
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