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David G. Munoz

David G. Munoz

D-Index & Metrics

Medicine

D-Index
79
Citations
24206
World Ranking
17507
National Ranking
732

Overview

David G. Munoz is affiliated with the University of Toronto in Canada. Their research focuses primarily on medicine, producing extensive work across several subfields including neurology, genetics, molecular biology, psychiatry and mental health, and epidemiology.

The scientist has contributed notably to topics such as glioma diagnosis and treatment, dementia and cognitive impairment research, Alzheimer's disease research and treatments, meningioma and schwannoma management, cancer, hypoxia, and metabolism, schizophrenia research and treatment, and pituitary gland disorders and treatments.

Frequent collaborators in their research include the following coauthors:

  • Corinne E. Fischer
  • Tom A. Schweizer
  • Sunit Das
  • Julia Keith
  • Gelareh Zadeh

The main venues where David G. Munoz has published include:

  • Neuro-Oncology
  • Alzheimer's & Dementia
  • Neurology
  • Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques
  • American Journal of Geriatric Psychiatry

Recent publications by David G. Munoz highlight their research themes and include:

  • Magnetosomes Could Be Protective Shields Against Metal Stress in Magnetotactic Bacteria, 2020, Scientific Reports
  • In vivo distribution of α-synuclein in multiple tissues and biofluids in Parkinson disease, 2020, Neurology (coauthor Lana M. Chahine)
  • Necrosis and Brain Invasion Predict Radio-Resistance and Tumor Recurrence in Atypical Meningioma: A Retrospective Cohort Study, 2020, Neurosurgery (coauthor Monica Emili Garcia-Segura)
  • Resection of the medial wall of the cavernous sinus in functioning pituitary adenomas: Technical note and outcomes in a matched-cohort study, 2020, Clinical Neurology and Neurosurgery (coauthor Abdelsimar T. Omar)
  • Intravoxel incoherent motion (IVIM) modeling of diffusion MRI during chemoradiation predicts therapeutic response in IDH wildtype glioblastoma, 2021, Radiotherapy and Oncology (coauthor Pejman Maralani)

Best Publications

  • The new mutation, E46K, of alpha-synuclein causes Parkinson and Lewy body dementia.

    Juan J. Zarranz;Javier Alegre;Juan C. Gómez-Esteban;Elena Lezcano

  • Neuropathologic diagnostic and nosologic criteria for frontotemporal lobar degeneration: consensus of the Consortium for Frontotemporal Lobar Degeneration

    Nigel J. Cairns;Eileen H. Bigio;Ian R A Mackenzie;Manuela Neumann

  • The evolution and pathology of frontotemporal dementia.

    Andrew Kertesz;Paul McMonagle;Mervin Blair;Wilda Davidson

  • Common variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusions

    Vivianna M. Van Deerlin;Patrick M A Sleiman;Maria Martinez-Lage;Maria Martinez-Lage;Alice Chen-Plotkin

  • Aging-related tau astrogliopathy (ARTAG): harmonized evaluation strategy

    Gabor G. Kovacs;Isidro Ferrer;Lea T. Grinberg;Lea T. Grinberg;Irina Alafuzoff

  • Primary lateral sclerosis. Clinical features, neuropathology and diagnostic criteria.

    C. E. Pringle;A. J. Hudson;D. G. Munoz;J. A. Kiernan

  • Corticobasal degeneration and progressive supranuclear palsy share a common tau haplotype

    H. Houlden;M. Baker;H. R. Morris;N. MacDonald

  • Vascular Factors in Cognitive Impairment‐Where Are We Now?

    Vladimir Hachinski;David Munoz

  • The corticobasal degeneration syndrome overlaps progressive aphasia and frontotemporal dementia.

    Andrew Kertesz;P. Martinez-Lage;W. Davidson;D. G. Munoz

  • Nonsteroidal anti-inflammatory drug use and Alzheimer-type pathology in aging

    Ian R.A. Mackenzie;David G. Munoz

  • The pathology and nosology of primary progressive aphasia

    Andrew Kertesz;Lawrence Hudson;Ian R.A. Mackenzie;David G. Munoz

  • FET proteins TAF15 and EWS are selective markers that distinguish FTLD with FUS pathology from amyotrophic lateral sclerosis with FUS mutations.

    Manuela Neumann;Eva Bentmann;Dorothee Dormann;Ali Jawaid

  • FUS pathology defines the majority of tau- and TDP-43-negative frontotemporal lobar degeneration.

    Hazel Urwin;Keith A. Josephs;Jonathan D. Rohrer;Ian R. MacKenzie

  • FUS pathology in basophilic inclusion body disease

    David G. Munoz;Manuela Neumann;Hirofumi Kusaka;Osamu Yokota

  • Accumulation of phosphorylated neurofilaments in anterior horn motoneurons of amyotrophic lateral sclerosis patients.

    D. G. Munoz;C. Greene;D. P. Perl;D. J. Selkoe

  • Corticobasal degeneration shares a common genetic background with progressive supranuclear palsy.

    Emilio Di Maria;Massimo Tabaton;Tiziana Vigo;Giovanni Abbruzzese

  • Role of microglia in senile plaque formation

    Ian R.A. Mackenzie;Chunhai Hao;David G. Munoz

  • Pathologic Correlates of Increased Signals of the Centrum Ovale on Magnetic Resonance Imaging

    David G. Munoz;Shirish M. Hastak;Byrne Harper;Donald Lee

  • Causes of Alzheimer's disease

    David G. Munoz;Howard Feldman

  • Intention tremor, parkinsonism, and generalized brain atrophy in male carriers of fragile X. Authors' reply

    David G. Munoz;R. J. Hagerman;C. Greco;S. Jacquemont

Frequent Co-Authors

Corinne E. Fischer
Corinne E. Fischer St. Michael's Hospital
Tom A. Schweizer
Tom A. Schweizer St. Michael's Hospital
Andrew Kertesz
Andrew Kertesz University of Western Ontario
Ian R. Mackenzie
Ian R. Mackenzie University of British Columbia
Vladimir Hachinski
Vladimir Hachinski University of Western Ontario
Tarek K. Rajji
Tarek K. Rajji Centre for Addiction and Mental Health
Benoit H. Mulsant
Benoit H. Mulsant Centre for Addiction and Mental Health
Christopher R. Bowie
Christopher R. Bowie Queen's University
Manuela Neumann
Manuela Neumann University of Tübingen
Charles L. White
Charles L. White The University of Texas Southwestern Medical Center

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