Her primary areas of study are T cell, Molecular biology, Immunology, Virology and Epitope. Brigitte T. Huber has included themes like Interleukin 2, Cytotoxic T cell, Internal medicine and Endocrinology in her T cell study. Her work deals with themes such as Peptide sequence, Antibody, Protease and Dipeptidyl peptidase-4, which intersect with Molecular biology.
Her study in Lyme Arthritis, Borrelia burgdorferi and Immune system is done as part of Immunology. The various areas that she examines in her Immune system study include C57BL/6 and Antigen. Her Virology research is multidisciplinary, incorporating elements of Superantigen, Autoimmunity, Antigen presentation and MHC class II.
The scientist’s investigation covers issues in Immunology, Molecular biology, T cell, Virology and Antigen. Her research in Borrelia burgdorferi, Lyme Arthritis, Immune system, Arthritis and Autoimmunity are components of Immunology. Brigitte T. Huber focuses mostly in the field of Molecular biology, narrowing it down to topics relating to Gene and, in certain cases, Minor Lymphocyte Stimulatory Antigens and Endogeny.
Brigitte T. Huber interconnects Cytotoxic T cell, Cell, Endocrinology and Cell biology in the investigation of issues within T cell. Brigitte T. Huber combines subjects such as Superantigen, Major histocompatibility complex and Antigen presentation with her study of Virology. Her studies deal with areas such as Antibody, B cell and Bruton's tyrosine kinase as well as Antigen.
Brigitte T. Huber mainly focuses on Immunology, Virology, Cell biology, Lyme disease and Borrelia burgdorferi. Her work often combines Immunology and TLR7 studies. Her Virology study integrates concerns from other disciplines, such as Chronic fatigue syndrome and Leukemia.
Brigitte T. Huber has researched Cell biology in several fields, including Autophagy, Cytotoxic T cell, T cell differentiation, T-cell receptor and Autoimmunity. Her Lyme disease research is multidisciplinary, incorporating perspectives in Lyme Arthritis, Antibody and Arthritis. Her Molecular biology research incorporates elements of CD28 and Biochemistry.
Brigitte T. Huber focuses on Virology, Cell biology, Immunology, Virus and Superantigen. Her Virology research integrates issues from Borrelia burgdorferi and Inflammatory arthritis. Her Cell biology research is multidisciplinary, relying on both Resting state fMRI, Cell cycle progression, Protease inhibitor and Cell quiescence.
Her work on Inflammation and B cell as part of general Immunology research is frequently linked to TLR7, thereby connecting diverse disciplines of science. Her studies in Virus integrate themes in fields like Interferon and Immune system. As a member of one scientific family, she mostly works in the field of Superantigen, focusing on Tyrosine and, on occasion, Phosphorylation.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Identification of LFA-1 as a Candidate Autoantigen in Treatment-Resistant Lyme Arthritis
Dawn M. Gross;Thomas Forsthuber;Magdalena Tary-Lehmann;Carey Etling.
Linkage of Mls genes to endogenous mammary tumour viruses of inbred mice.
Wayne N. Frankel;Christine Rudy;John M. Coffin;Brigitte T. Huber.
Cholera Toxin Discriminates Between T Helper 1 and 2 Cells in T Cell Receptor-Mediated Activation: Role of cAMP in T Cell Proliferation
Eduardo Muñoz;Ana M. Zubiaga;Martha Merrow;Nicholas P. Sauter.
Journal of Experimental Medicine (1990)
Epstein-Barr Virus Transactivates the Human Endogenous Retrovirus HERV-K18 that Encodes a Superantigen
Natalie Sutkowski;Bernard Conrad;David A Thorley-Lawson;Brigitte T Huber.
Cell-mediated immunity: delayed-type hypersensitivity and cytotoxic responses are mediated by different T-cell subclasses.
B Huber;O Devinsky;R K Gershon;H Cantor.
Journal of Experimental Medicine (1976)
Inhibition of dipeptidyl aminopeptidase IV (DP-IV) by Xaa-boroPro dipeptides and use of these inhibitors to examine the role of DP-IV in T-cell function.
G R Flentke;E Munoz;B T Huber;A G Plaut.
Proceedings of the National Academy of Sciences of the United States of America (1991)
Identification of a B-cell surface structure involved in antigen-dependent triggering: absence of this structure on B cells from CBA/N mutant mice.
B Huber;R K Gershon;H Cantor.
Journal of Experimental Medicine (1977)
IL-4 and IL-2 selectively rescue Th cell subsets from glucocorticoid-induced apoptosis.
A M Zubiaga;E Munoz;B T Huber.
Journal of Immunology (1992)
Human immunodeficiency virus 1 Tat binds to dipeptidyl aminopeptidase IV (CD26): a possible mechanism for Tat's immunosuppressive activity.
William G. Gutheil;Meena Subramanyam;George R. Flentke;David G. Sanford.
Proceedings of the National Academy of Sciences of the United States of America (1994)
Contamination of human DNA samples with mouse DNA can lead to false detection of XMRV-like sequences.
Brendan Oakes;Albert K Tai;Oya Cingöz;Madeleine H Henefield.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: