D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 47 Citations 8,619 172 World Ranking 11800 National Ranking 5078

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Immune system
  • Enzyme

Her primary areas of study are T cell, Molecular biology, Immunology, Virology and Epitope. Brigitte T. Huber has included themes like Interleukin 2, Cytotoxic T cell, Internal medicine and Endocrinology in her T cell study. Her work deals with themes such as Peptide sequence, Antibody, Protease and Dipeptidyl peptidase-4, which intersect with Molecular biology.

Her study in Lyme Arthritis, Borrelia burgdorferi and Immune system is done as part of Immunology. The various areas that she examines in her Immune system study include C57BL/6 and Antigen. Her Virology research is multidisciplinary, incorporating elements of Superantigen, Autoimmunity, Antigen presentation and MHC class II.

Her most cited work include:

  • Identification of LFA-1 as a Candidate Autoantigen in Treatment-Resistant Lyme Arthritis (433 citations)
  • Linkage of Mls genes to endogenous mammary tumour viruses of inbred mice. (248 citations)
  • Epstein-Barr Virus Transactivates the Human Endogenous Retrovirus HERV-K18 that Encodes a Superantigen (233 citations)

What are the main themes of her work throughout her whole career to date?

The scientist’s investigation covers issues in Immunology, Molecular biology, T cell, Virology and Antigen. Her research in Borrelia burgdorferi, Lyme Arthritis, Immune system, Arthritis and Autoimmunity are components of Immunology. Brigitte T. Huber focuses mostly in the field of Molecular biology, narrowing it down to topics relating to Gene and, in certain cases, Minor Lymphocyte Stimulatory Antigens and Endogeny.

Brigitte T. Huber interconnects Cytotoxic T cell, Cell, Endocrinology and Cell biology in the investigation of issues within T cell. Brigitte T. Huber combines subjects such as Superantigen, Major histocompatibility complex and Antigen presentation with her study of Virology. Her studies deal with areas such as Antibody, B cell and Bruton's tyrosine kinase as well as Antigen.

She most often published in these fields:

  • Immunology (46.20%)
  • Molecular biology (30.43%)
  • T cell (26.09%)

What were the highlights of her more recent work (between 2007-2017)?

  • Immunology (46.20%)
  • Virology (25.54%)
  • Cell biology (17.39%)

In recent papers she was focusing on the following fields of study:

Brigitte T. Huber mainly focuses on Immunology, Virology, Cell biology, Lyme disease and Borrelia burgdorferi. Her work often combines Immunology and TLR7 studies. Her Virology study integrates concerns from other disciplines, such as Chronic fatigue syndrome and Leukemia.

Brigitte T. Huber has researched Cell biology in several fields, including Autophagy, Cytotoxic T cell, T cell differentiation, T-cell receptor and Autoimmunity. Her Lyme disease research is multidisciplinary, incorporating perspectives in Lyme Arthritis, Antibody and Arthritis. Her Molecular biology research incorporates elements of CD28 and Biochemistry.

Between 2007 and 2017, her most popular works were:

  • Contamination of human DNA samples with mouse DNA can lead to false detection of XMRV-like sequences. (128 citations)
  • Complement Receptor 2 Is Expressed in Neural Progenitor Cells and Regulates Adult Hippocampal Neurogenesis (69 citations)
  • Human endogenous retrovirus-K18 Env as a risk factor in multiple sclerosis. (62 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Enzyme

Brigitte T. Huber focuses on Virology, Cell biology, Immunology, Virus and Superantigen. Her Virology research integrates issues from Borrelia burgdorferi and Inflammatory arthritis. Her Cell biology research is multidisciplinary, relying on both Resting state fMRI, Cell cycle progression, Protease inhibitor and Cell quiescence.

Her work on Inflammation and B cell as part of general Immunology research is frequently linked to TLR7, thereby connecting diverse disciplines of science. Her studies in Virus integrate themes in fields like Interferon and Immune system. As a member of one scientific family, she mostly works in the field of Superantigen, focusing on Tyrosine and, on occasion, Phosphorylation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Identification of LFA-1 as a Candidate Autoantigen in Treatment-Resistant Lyme Arthritis

Dawn M. Gross;Thomas Forsthuber;Magdalena Tary-Lehmann;Carey Etling.
Science (1998)

573 Citations

Linkage of Mls genes to endogenous mammary tumour viruses of inbred mice.

Wayne N. Frankel;Christine Rudy;John M. Coffin;Brigitte T. Huber.
Nature (1991)

380 Citations

Cholera Toxin Discriminates Between T Helper 1 and 2 Cells in T Cell Receptor-Mediated Activation: Role of cAMP in T Cell Proliferation

Eduardo Muñoz;Ana M. Zubiaga;Martha Merrow;Nicholas P. Sauter.
Journal of Experimental Medicine (1990)

296 Citations

Epstein-Barr Virus Transactivates the Human Endogenous Retrovirus HERV-K18 that Encodes a Superantigen

Natalie Sutkowski;Bernard Conrad;David A Thorley-Lawson;Brigitte T Huber.
Immunity (2001)

284 Citations

Cell-mediated immunity: delayed-type hypersensitivity and cytotoxic responses are mediated by different T-cell subclasses.

B Huber;O Devinsky;R K Gershon;H Cantor.
Journal of Experimental Medicine (1976)

281 Citations

Inhibition of dipeptidyl aminopeptidase IV (DP-IV) by Xaa-boroPro dipeptides and use of these inhibitors to examine the role of DP-IV in T-cell function.

G R Flentke;E Munoz;B T Huber;A G Plaut.
Proceedings of the National Academy of Sciences of the United States of America (1991)

234 Citations

Identification of a B-cell surface structure involved in antigen-dependent triggering: absence of this structure on B cells from CBA/N mutant mice.

B Huber;R K Gershon;H Cantor.
Journal of Experimental Medicine (1977)

227 Citations

IL-4 and IL-2 selectively rescue Th cell subsets from glucocorticoid-induced apoptosis.

A M Zubiaga;E Munoz;B T Huber.
Journal of Immunology (1992)

208 Citations

Human immunodeficiency virus 1 Tat binds to dipeptidyl aminopeptidase IV (CD26): a possible mechanism for Tat's immunosuppressive activity.

William G. Gutheil;Meena Subramanyam;George R. Flentke;David G. Sanford.
Proceedings of the National Academy of Sciences of the United States of America (1994)

176 Citations

Contamination of human DNA samples with mouse DNA can lead to false detection of XMRV-like sequences.

Brendan Oakes;Albert K Tai;Oya Cingöz;Madeleine H Henefield.
Retrovirology (2010)

176 Citations

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