His primary areas of investigation include Molecular biology, DNA damage, Cell biology, Tyrosine kinase and Biochemistry. His Molecular biology study combines topics in areas such as Cell growth, Protein kinase A, Tyrosine phosphorylation, Receptor tyrosine kinase and Telomerase. His DNA damage research includes elements of Signal transduction, Kinase and DNA repair.
The various areas that he examines in his Signal transduction study include Tyrosine, Programmed cell death and Tumor Protein p73. His work carried out in the field of Cell biology brings together such families of science as Apoptosis and Bcl-xL. In his work, ABL Tyrosine Kinase and Downregulation and upregulation is strongly intertwined with Cell culture, which is a subfield of Tyrosine kinase.
His scientific interests lie mostly in Cell biology, Cancer research, Molecular biology, DNA damage and Kinase. His research integrates issues of Biochemistry and Mdm2 in his study of Cell biology. The Cancer research study combines topics in areas such as Cancer cell, Cancer, Cyclin-dependent kinase 2, ASK1 and Estrogen receptor.
His work deals with themes such as Immunoprecipitation, Carcinogenesis, Mutant, DNA-binding protein and Nuclear protein, which intersect with Molecular biology. In DNA damage, Zhi-Min Yuan works on issues like Apoptosis, which are connected to Protein kinase A. His Phosphorylation research includes themes of Tyrosine kinase and ABL.
Zhi-Min Yuan mainly focuses on Cell biology, Cancer research, Cancer cell, Signal transduction and MDMX. Zhi-Min Yuan is studying Kinase, which is a component of Cell biology. His Kinase study combines topics from a wide range of disciplines, such as Annexin, Reporter gene, FADD and Programmed cell death.
His studies deal with areas such as Carcinogenesis, Downregulation and upregulation, Immunology and Estrogen receptor, Estrogen receptor alpha as well as Cancer research. His work carried out in the field of Signal transduction brings together such families of science as Molecular biology and Apoptosis. His Gene isoform research incorporates themes from Oxidative stress, Cell survival, Mode of action and Bioinformatics.
His primary areas of study are Cell biology, Cancer research, Glycolysis, Cancer cell and Phosphorylation. His Cell biology research is multidisciplinary, incorporating elements of Regulation of gene expression and Transcription. The concepts of his Cancer research study are interwoven with issues in Protein subunit, Radiation therapy, Stem cell marker and Skin cancer.
In his study, which falls under the umbrella issue of Cancer cell, Molecular biology is strongly linked to Protein tyrosine phosphatase. He has researched Molecular biology in several fields, including Carcinogenesis and Biogenesis. His Phosphorylation research focuses on subjects like Signal transduction, which are linked to Kinase.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Autophagy (2012)
Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
p73 is regulated by tyrosine kinase c-Abl in the apoptotic response to DNA damage
Zhi-Min Yuan;Hisashi Shioya;Takatoshi Ishiko;Xiangao Sun.
Nature (1999)
Phosphorylation by aurora kinase A induces Mdm2-mediated destabilization and inhibition of p53
Hiroshi Katayama;Kaori Sasai;Hidehiko Kawai;Hidehiko Kawai;Zhi Min Yuan.
Nature Genetics (2004)
Translocation of SAPK/JNK to Mitochondria and Interaction with Bcl-xL in Response to DNA Damage
Surender Kharbanda;Satya Saxena;Kiyotsugu Yoshida;Pramod Pandey.
Journal of Biological Chemistry (2000)
Role for Bcl-xL as an inhibitor of cytosolic cytochrome C accumulation in DNA damage-induced apoptosis
Surender Kharbanda;Pramod Pandey;Lesley Schofield;Sara Israels.
Proceedings of the National Academy of Sciences of the United States of America (1997)
Mutual Dependence of MDM2 and MDMX in Their Functional Inactivation of p53
Jijie Gu;Hidehiko Kawai;Linghu Nie;Hiroyuki Kitao.
Journal of Biological Chemistry (2002)
Functional interaction between DNA-PK and c-Abl in response to DNA damage
Surender Kharbanda;Pramod Pandey;Shengfang Jin;Satoshi Inoue.
Nature (1997)
Role for c-Abl tyrosine kinase in growth arrest response to DNA damage
Zhi-Min Yuan;Yinyin Huang;Young Whang;Charles Sawyers.
Nature (1996)
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