What is he best known for?
The fields of study he is best known for:
Surender Kharbanda mainly focuses on Molecular biology, Cell biology, Apoptosis, Kinase and DNA damage.
Surender Kharbanda interconnects Protein kinase A, Proto-oncogene tyrosine-protein kinase Src, Protein kinase C and DNA fragmentation, Programmed cell death in the investigation of issues within Molecular biology.
His studies deal with areas such as Bcl-xL, Transfection and Chromosomal translocation as well as Cell biology.
His studies in Apoptosis integrate themes in fields like In vitro and Multiple myeloma.
His study in Kinase is interdisciplinary in nature, drawing from both Tyrosine kinase and Signal transduction.
His study explores the link between DNA damage and topics such as DNA repair that cross with problems in DNA supercoil.
His most cited work include:
- Proteolytic activation of protein kinase C delta by an ICE-like protease in apoptotic cells. (646 citations)
- p73 is regulated by tyrosine kinase c-Abl in the apoptotic response to DNA damage (540 citations)
- Apoptosis in heart failure: Release of cytochrome c from mitochondria and activation of caspase-3 in human cardiomyopathy (521 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Molecular biology, Cell biology, Cancer research, Apoptosis and Kinase.
The study incorporates disciplines such as Cell culture, Gene expression, Cellular differentiation, Protein kinase C and Myeloid leukemia in addition to Molecular biology.
His research combines Biochemistry and Cell biology.
Surender Kharbanda has researched Cancer research in several fields, including Tumor necrosis factor alpha, IκBα, MUC1, Signal transduction and Programmed cell death.
His work on Mitogen-activated protein kinase as part of general Kinase study is frequently connected to Myelin basic protein, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
His Tyrosine kinase research incorporates elements of Cell cycle, DNA damage and Proto-oncogene tyrosine-protein kinase Src.
He most often published in these fields:
- Molecular biology (47.92%)
- Cell biology (40.97%)
- Cancer research (29.86%)
What were the highlights of his more recent work (between 2004-2021)?
- MUC1 (9.72%)
- Cancer research (29.86%)
- Apoptosis (21.53%)
In recent papers he was focusing on the following fields of study:
Surender Kharbanda mainly investigates MUC1, Cancer research, Apoptosis, Cancer and Cell biology.
His MUC1 study incorporates themes from Molecular biology, Reactive oxygen species and Endocrinology.
Surender Kharbanda undertakes multidisciplinary studies into Molecular biology and Domain in his work.
His biological study spans a wide range of topics, including Cell culture, Downregulation and upregulation, IκBα, Cysteine and Programmed cell death.
The Apoptosis study combines topics in areas such as IC50 and In vitro.
Surender Kharbanda conducts interdisciplinary study in the fields of Cell biology and Death receptor ligand through his works.
Between 2004 and 2021, his most popular works were:
- Triterpenoid CDDO-Me Blocks the NF-κB Pathway by Direct Inhibition of IKKβ on Cys-179 (215 citations)
- MUC1 Oncoprotein Blocks Glycogen Synthase Kinase 3β–Mediated Phosphorylation and Degradation of β-Catenin (195 citations)
- MUC1 oncoprotein activates the IκB kinase β complex and constitutive NF-κB signalling (148 citations)
In his most recent research, the most cited papers focused on:
Surender Kharbanda mostly deals with Cancer research, MUC1, Phosphorylation, IκBα and Cell biology.
His Cancer research study frequently draws connections between adjacent fields such as Cyclin E.
His Phosphorylation study deals with the bigger picture of Biochemistry.
His IκBα study necessitates a more in-depth grasp of Signal transduction.
He has included themes like Transcription factor, Kinase, Activator and Programmed cell death in his Signal transduction study.
His Cell biology research includes elements of Necrosis, Internal medicine and Mucin.
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