What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Mutation
His main research concerns Acetylcholine receptor, Myasthenia gravis, Immunology, Congenital myasthenic syndrome and Neuromuscular junction.
His Acetylcholine receptor research is within the category of Internal medicine.
His research in Myasthenia gravis intersects with topics in Arthrogryposis multiplex congenita and Antigen.
His research integrates issues of Odds ratio, Receptor, Allele frequency and Age of onset in his study of Immunology.
David Beeson has researched Congenital myasthenic syndrome in several fields, including Surgery, Weakness and Neuromuscular transmission.
His Neuromuscular junction study combines topics from a wide range of disciplines, such as DPAGT1, Bioinformatics and Cell biology.
His most cited work include:
- Human mitochondrial DNA deletions associated with mutations in the gene encoding Twinkle, a phage T7 gene 4-like protein localized in mitochondria. (715 citations)
- N-methyl-d-aspartate antibody encephalitis: temporal progression of clinical and paraclinical observations in a predominantly non-paraneoplastic disorder of both sexes (699 citations)
- IgG1 antibodies to acetylcholine receptors in 'seronegative' myasthenia gravis. (333 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Acetylcholine receptor, Congenital myasthenic syndrome, Myasthenia gravis, Neuromuscular junction and Immunology.
David Beeson interconnects Molecular biology, Endocrinology and Protein subunit in the investigation of issues within Acetylcholine receptor.
His biological study spans a wide range of topics, including DPAGT1, Pediatrics and Neuromuscular transmission.
His work focuses on many connections between Neuromuscular transmission and other disciplines, such as Weakness, that overlap with his field of interest in Ptosis.
His work in Myasthenia gravis addresses subjects such as Antibody, which are connected to disciplines such as Virology.
The various areas that he examines in his Neuromuscular junction study include Agrin, Synapse, Muscle weakness and Cell biology.
He most often published in these fields:
- Acetylcholine receptor (47.29%)
- Congenital myasthenic syndrome (28.61%)
- Myasthenia gravis (26.20%)
What were the highlights of his more recent work (between 2014-2021)?
- Neuromuscular junction (26.81%)
- Acetylcholine receptor (47.29%)
- Neuromuscular transmission (21.99%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in Neuromuscular junction, Acetylcholine receptor, Neuromuscular transmission, Congenital myasthenic syndrome and Myasthenia gravis.
His Neuromuscular junction research is multidisciplinary, incorporating elements of Postsynaptic potential, Pyridostigmine, Muscle contraction, Cell biology and Synapse.
David Beeson combines subjects such as Endocrinology and Motor neuron with his study of Acetylcholine receptor.
His Neuromuscular transmission research is multidisciplinary, relying on both Genetics, Ptosis, Phenotype, Acetylcholine and Weakness.
The concepts of his Congenital myasthenic syndrome study are interwoven with issues in Missense mutation, Repetitive nerve stimulation, DPAGT1, Muscle weakness and Pediatrics.
His Myasthenia gravis research entails a greater understanding of Immunology.
Between 2014 and 2021, his most popular works were:
- Factors influencing success of clinical genome sequencing across a broad spectrum of disorders (227 citations)
- Antibodies to GABAA receptor α1 and γ2 subunits Clinical and serologic characterization (89 citations)
- Mutations in GMPPB cause congenital myasthenic syndrome and bridge myasthenic disorders with dystroglycanopathies. (75 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Mutation
- Internal medicine
His primary areas of study are Neuromuscular transmission, Neuromuscular junction, Genetics, Acetylcholine receptor and Myasthenia gravis.
His Neuromuscular transmission research is multidisciplinary, incorporating perspectives in Phenotype and Congenital myasthenic syndrome.
His Congenital myasthenic syndrome research includes themes of Frameshift mutation, Terminal bouton, Weakness and Pathology.
His Neuromuscular junction research includes elements of Laminin, Glycosylation and Pyridostigmine.
His work deals with themes such as Myotube differentiation and Cell biology, which intersect with Acetylcholine receptor.
His Myasthenia gravis study incorporates themes from Autoantibody and Severity of illness.
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