His primary areas of investigation include Pharmacology, Biochemistry, Oxidative stress, Internal medicine and Alzheimer's disease. His Pharmacology research is multidisciplinary, relying on both Antidepressant, Receptor and Benzodiazepine receptor binding. In his study, Amyloid precursor protein secretase is strongly linked to Cell biology, which falls under the umbrella field of Biochemistry.
The study incorporates disciplines such as Ginkgo biloba, Neuroscience and Antioxidant in addition to Oxidative stress. His work in Internal medicine tackles topics such as Endocrinology which are related to areas like Membrane fluidity. His research integrates issues of Extracellular, Amyloid beta, Mitochondrion and Intracellular in his study of Alzheimer's disease.
Walter E. Müller spends much of his time researching Pharmacology, Internal medicine, Biochemistry, Endocrinology and Cell biology. His Pharmacology research incorporates themes from Antidepressant, Benzodiazepine and Serotonin. Biochemistry is represented through his Binding site, Mitochondrion and Oxidative stress research.
His study looks at the intersection of Mitochondrion and topics like Alzheimer's disease with Neuroscience and Dementia. His Oxidative stress research focuses on Programmed cell death and how it connects with Immunology. His study looks at the relationship between Cell biology and topics such as Amyloid precursor protein, which overlap with Apoptosis.
Walter E. Müller mainly focuses on Neuroscience, Pharmacology, Internal medicine, Cell biology and Mitochondrion. His Neuroscience study also includes fields such as
His Internal medicine research integrates issues from Endocrinology and Surgery. He works mostly in the field of Endocrinology, limiting it down to topics relating to Amyloid precursor protein and, in certain cases, Amyloid. His Mitochondrion study is concerned with the larger field of Biochemistry.
Walter E. Müller focuses on Cell biology, Disease, Oxidative stress, Internal medicine and Neuroscience. Walter E. Müller interconnects Respiratory chain and Pathogenesis in the investigation of issues within Oxidative stress. His work in Pathogenesis addresses issues such as Pharmacology, which are connected to fields such as Mitochondrial permeability transition pore.
His study in Endocrinology extends to Internal medicine with its themes. His Endocrinology research includes themes of Apolipoprotein E and Amyloid. While the research belongs to areas of Neuroscience, Walter E. Müller spends his time largely on the problem of Dementia, intersecting his research to questions surrounding Antioxidant.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Hyperforin as a possible antidepressant component of hypericum extracts
S.S. Chatterjee;S.K. Bhattacharya;M. Wonnemann;A. Singer.
Life Sciences (1998)
Hyperforin represents the neurotransmitter reuptake inhibiting constituent of hypericum extract.
W. E. Müller;A. Singer;M. Wonnemann;U. Hafner.
The location of drug binding sites in human serum albumin.
Klaus J. Fehske;Walter E. Müller;Uwe Wollert.
Biochemical Pharmacology (1981)
Abuse and dependence potential for the non-benzodiazepine hypnotics zolpidem and zopiclone: a review of case reports and epidemiological data.
G Hajak;WE Muller;HU Wittchen;D Pittrow.
Mitochondrial dysfunction: an early event in Alzheimer pathology accumulates with age in AD transgenic mice.
S. Hauptmann;I. Scherping;S. Dröse;U. Brandt.
Neurobiology of Aging (2009)
Effects of hypericum extract (LI 160) in biochemical models of antidepressant activity.
W E Müller;M Rolli;C Schäfer;U Hafner.
Hyperforin, a major antidepressant constituent of St. John's Wort, inhibits serotonin uptake by elevating free intracellular Na+1.
A Singer;M Wonnemann;W E Müller.
Journal of Pharmacology and Experimental Therapeutics (1999)
Proteomic and functional analyses reveal a mitochondrial dysfunction in P301L tau transgenic mice
Della C. David;Susanne Hauptmann;Isabel Scherping;Katrin Schuessel.
Journal of Biological Chemistry (2005)
Parkinson phenotype in aged PINK1-deficient mice is accompanied by progressive mitochondrial dysfunction in absence of neurodegeneration.
Suzana Gispert;Filomena Ricciardi;Alexander Kurz;Mekhman Azizov.
PLOS ONE (2009)
Mitochondrial dysfunction, apoptotic cell death, and Alzheimer's disease.
Anne Eckert;Uta Keil;Celio A. Marques;Astrid Bonert.
Biochemical Pharmacology (2003)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: