What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Internal medicine
His primary areas of investigation include Neuroscience, Alzheimer's disease, Cell biology, Disease and Tau protein.
His Neuroscience study incorporates themes from Substantia nigra, Transgene and Frontotemporal dementia.
His Frontotemporal dementia research includes elements of Axoplasmic transport, Genetically modified mouse and Trinucleotide repeat expansion.
Lars M. Ittner has researched Alzheimer's disease in several fields, including Endocrinology, Crosstalk and Degenerative disease.
His work carried out in the field of Cell biology brings together such families of science as Excitotoxicity and Postsynaptic potential.
His Disease research is multidisciplinary, incorporating perspectives in Neuroprotection and Molecular pathway.
His most cited work include:
- Dendritic Function of Tau Mediates Amyloid-β Toxicity in Alzheimer's Disease Mouse Models (1251 citations)
- Amyloid-β and tau — a toxic pas de deux in Alzheimer's disease (884 citations)
- Animal models of Alzheimer's disease and frontotemporal dementia (539 citations)
What are the main themes of his work throughout his whole career to date?
Lars M. Ittner mainly investigates Genetically modified mouse, Neuroscience, Cell biology, Transgene and Disease.
His Genetically modified mouse research integrates issues from Endocrinology, Tau protein, Pathology, Internal medicine and Tauopathy.
His Neuroscience research incorporates elements of Dementia, Frontotemporal dementia, Amyotrophic lateral sclerosis, Excitotoxicity and Alzheimer's disease.
His study in Cell biology is interdisciplinary in nature, drawing from both Gene expression and Immunology.
His biological study deals with issues like Molecular biology, which deal with fields such as Calcitonin gene-related peptide.
His research in Disease intersects with topics in Treatment strategy and Pathological.
He most often published in these fields:
- Genetically modified mouse (43.16%)
- Neuroscience (41.03%)
- Cell biology (36.75%)
What were the highlights of his more recent work (between 2016-2021)?
- Neuroscience (41.03%)
- Genetically modified mouse (43.16%)
- Tau protein (22.65%)
In recent papers he was focusing on the following fields of study:
His main research concerns Neuroscience, Genetically modified mouse, Tau protein, Disease and Cell biology.
His Neuroscience research includes themes of Alzheimer's disease, Phenotype, Hyperphosphorylation and Excitotoxicity.
Genetically modified mouse is the subject of his research, which falls under Transgene.
His Transgene research incorporates themes from Amyotrophic lateral sclerosis, Neurodegeneration and Pathology.
The various areas that Lars M. Ittner examines in his Disease study include Epothilone D and Pharmacology.
The Cell biology study combines topics in areas such as Tissue engineering, Cell culture and Gene expression.
Between 2016 and 2021, his most popular works were:
- The Link between Type 2 Diabetes and Neurodegeneration: Roles for Amyloid-β, Amylin, and Tau Proteins (84 citations)
- Dendritic Tau in Alzheimer's Disease. (83 citations)
- Dendritic Tau in Alzheimer's Disease. (83 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
Lars M. Ittner mainly focuses on Neuroscience, Disease, Tau protein, Alzheimer's disease and Frontotemporal dementia.
His studies in Neuroscience integrate themes in fields like Neuroinflammation, Amyotrophic lateral sclerosis, Neurodegeneration and MAPK/ERK pathway.
Disease is a subfield of Pathology that Lars M. Ittner studies.
His research integrates issues of Genetically modified mouse and Viral vector in his study of Pathology.
In his study, Amyloid is inextricably linked to Endocrinology, which falls within the broad field of Alzheimer's disease.
The study incorporates disciplines such as Neuropathology and Pathological in addition to Frontotemporal dementia.
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