What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Cancer
Jürgen Götz mainly investigates Alzheimer's disease, Genetically modified mouse, Tau protein, Neuroscience and Cell biology.
His research investigates the connection between Alzheimer's disease and topics such as Hyperphosphorylation that intersect with issues in Frontotemporal lobar degeneration and Dephosphorylation.
His Genetically modified mouse study introduces a deeper knowledge of Transgene.
The various areas that he examines in his Tau protein study include Molecular biology, Tauopathy and Gene isoform.
His research integrates issues of Substantia nigra, Disease, Pathology and Frontotemporal dementia in his study of Neuroscience.
His Cell biology study incorporates themes from Biochemistry, Neurodegeneration and Amyloid precursor protein.
His most cited work include:
- Formation of neurofibrillary tangles in P301L tau transgenic mice induced by Aβ42 fibrils (1262 citations)
- Dendritic Function of Tau Mediates Amyloid-β Toxicity in Alzheimer's Disease Mouse Models (1251 citations)
- Amyloid-β and tau — a toxic pas de deux in Alzheimer's disease (884 citations)
What are the main themes of his work throughout his whole career to date?
Jürgen Götz mostly deals with Cell biology, Tau protein, Neuroscience, Genetically modified mouse and Disease.
His study in Mutant extends to Cell biology with its themes.
To a larger extent, Jürgen Götz studies Alzheimer's disease with the aim of understanding Tau protein.
His studies deal with areas such as Neurodegeneration and Frontotemporal dementia as well as Neuroscience.
His Genetically modified mouse study contributes to a more complete understanding of Transgene.
Jürgen Götz usually deals with Mitochondrion and limits it to topics linked to Reactive oxygen species and Oxidative phosphorylation.
He most often published in these fields:
- Cell biology (34.62%)
- Tau protein (26.92%)
- Neuroscience (23.46%)
What were the highlights of his more recent work (between 2016-2021)?
- Cell biology (34.62%)
- Tauopathy (13.85%)
- Neuroscience (23.46%)
In recent papers he was focusing on the following fields of study:
Cell biology, Tauopathy, Neuroscience, Tau protein and Disease are his primary areas of study.
His Cell biology research is multidisciplinary, incorporating elements of Hippocampal formation, Autophagy and Monoclonal antibody.
The Tauopathy study combines topics in areas such as Frontotemporal lobar degeneration and Genetically modified mouse, Transgene.
His Neuroscience study integrates concerns from other disciplines, such as Personalized medicine, Pathogenesis and Protein folding.
Tau protein is the subject of his research, which falls under Alzheimer's disease.
Many of his research projects under Disease are closely connected to In patient with In patient, tying the diverse disciplines of science together.
Between 2016 and 2021, his most popular works were:
- Amyloid-β and tau complexity - towards improved biomarkers and targeted therapies. (153 citations)
- Tau-based therapies in neurodegeneration: opportunities and challenges. (109 citations)
- Rodent models for Alzheimer disease (91 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Cancer
His primary scientific interests are in Tauopathy, Cell biology, Tau protein, Alzheimer's disease and Neuroscience.
His Tauopathy research incorporates elements of Therapeutic ultrasound, Genetically modified mouse, Neuropathology and Frontotemporal lobar degeneration.
His work carried out in the field of Cell biology brings together such families of science as Autophagy and Transgene.
His Tau protein study combines topics in areas such as Organelle fusion, Microvesicles, Tetraspanin, Neuron and Endosome.
His research in Alzheimer's disease focuses on subjects like Signal transduction, which are connected to Hyperphosphorylation.
The concepts of his Neuroscience study are interwoven with issues in Protein processing, Post translational and Disease, Neurodegeneration.
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