What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Internal medicine
His main research concerns Biochemistry, Gene isoform, Molecular biology, Cell biology and Scrapie.
His Biochemistry research is multidisciplinary, relying on both Neurotoxicity and In vivo.
His Gene isoform research incorporates elements of Cell culture, Cleavage, Pathogenesis and Glycoprotein.
The various areas that David A. Harris examines in his Molecular biology study include PrPSc Proteins, Genetically modified mouse and Mutant.
His Cell biology research incorporates themes from Cell and Endocytosis.
His biological study spans a wide range of topics, including Protein structure and Transfection.
His most cited work include:
- SULFATED GLYCANS STIMULATE ENDOCYTOSIS OF THE CELLULAR ISOFORM OF THE PRION PROTEIN, PRPC, IN CULTURED CELLS (659 citations)
- Copper Stimulates Endocytosis of the Prion Protein (524 citations)
- The Cellular Prion Protein (PrPC): Its Physiological Function and Role in Disease (304 citations)
What are the main themes of his work throughout his whole career to date?
David A. Harris spends much of his time researching Cell biology, Molecular biology, Mutant, Biochemistry and Gene isoform.
His Cell biology research focuses on Cell culture and how it connects with Amino acid.
He combines subjects such as Phenotype, PrPSc Proteins, Neurodegeneration, Transfection and Genetically modified mouse with his study of Molecular biology.
His work deals with themes such as Signal peptide, Endoplasmic reticulum, Chinese hamster ovary cell and Virology, which intersect with Mutant.
His study looks at the intersection of Endoplasmic reticulum and topics like Transmembrane protein with Transmembrane domain.
His Gene isoform research includes themes of Scrapie, Pathogenesis and Glycoprotein.
He most often published in these fields:
- Cell biology (28.04%)
- Molecular biology (23.81%)
- Mutant (23.81%)
What were the highlights of his more recent work (between 2016-2021)?
- Sleeve gastrectomy (5.29%)
- Internal medicine (10.58%)
- Cell biology (28.04%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Sleeve gastrectomy, Internal medicine, Cell biology, Surgery and Prion protein.
He studies Cell biology, focusing on Effector in particular.
His Prion protein study combines topics from a wide range of disciplines, such as Genetics, Gene and Toxicity.
The Neuroprotection study which covers Glycoprotein that intersects with Neurotoxicity.
His Antibody research integrates issues from Amyloid β, Amyloid, Aβ oligomers, Neurite and Biochemistry.
His study focuses on the intersection of Docking and fields such as Epitope with connections in the field of Gene isoform.
Between 2016 and 2021, his most popular works were:
- PrP is a central player in toxicity mediated by soluble aggregates of neurodegeneration-causing proteins (32 citations)
- The N-terminus of the prion protein is a toxic effector regulated by the C-terminus (32 citations)
- Prions activate a p38 MAPK synaptotoxic signaling pathway. (25 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
David A. Harris mainly focuses on Type 2 diabetes, Surgery, Receptor, Sleeve gastrectomy and Neuroscience.
His Surgery research includes elements of Agonist, Diabetes mellitus and G protein-coupled bile acid receptor, Bile acid.
His Receptor study deals with Biophysics intersecting with Mutation, Protein domain and Ion channel.
His Sleeve gastrectomy study is associated with Internal medicine.
David A. Harris has researched Neuroscience in several fields, including Neurotoxicity, Molecular pathology and Signal transduction, Calcium signaling.
His work deals with themes such as Dendritic spine, Actin cytoskeleton, Kinase and Neurotransmission, which intersect with Neurotoxicity.
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