What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include Scrapie, Virology, Molecular biology, Biochemistry and PrPSc Proteins.
The Scrapie study combines topics in areas such as Transgene, Proteinase K, Hamster and Gene isoform.
His Virology research includes themes of Pathogenesis, Bovine spongiform encephalopathy, Neurodegeneration, Disease and Genetically modified mouse.
His Molecular biology study combines topics from a wide range of disciplines, such as Complementary DNA, Messenger RNA, Gene, Blot and Epitope.
Biochemistry and Amyloid are commonly linked in his work.
His work carried out in the field of PrPSc Proteins brings together such families of science as Cell culture, Cell, Phospholipase C, Transmissible mink encephalopathy and Cell biology.
His most cited work include:
- Nobel Lecture: Prions (4075 citations)
- Novel proteinaceous infectious particles cause scrapie (4009 citations)
- Molecular biology of prion diseases (1717 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Scrapie, Virology, Biochemistry, Molecular biology and Gene isoform.
His work is dedicated to discovering how Scrapie, Infectivity are connected with Nucleic acid and other disciplines.
His Virology research includes elements of Genetically modified mouse, Gene, Transgene, Bovine spongiform encephalopathy and Disease.
His study connects Prion protein and Biochemistry.
His work carried out in the field of Molecular biology brings together such families of science as Amino acid, Recombinant DNA, Proteinase K and Epitope, Antibody.
His PrPSc Proteins study typically links adjacent topics like Cell culture.
He most often published in these fields:
- Scrapie (41.07%)
- Virology (34.96%)
- Biochemistry (25.97%)
What were the highlights of his more recent work (between 2006-2021)?
- Virology (34.96%)
- Biochemistry (25.97%)
- Genetically modified mouse (11.11%)
In recent papers he was focusing on the following fields of study:
His main research concerns Virology, Biochemistry, Genetically modified mouse, Molecular biology and Scrapie.
His biological study spans a wide range of topics, including Cell culture, PrPSc Proteins, Neurodegeneration, Chronic wasting disease and Gene isoform.
Stanley B. Prusiner has included themes like Infectivity, Prion protein and Amyloid in his Biochemistry study.
His Genetically modified mouse research is multidisciplinary, incorporating perspectives in Phenotype, Bovine spongiform encephalopathy, Neuroscience and Alpha-synuclein.
His Molecular biology study combines topics from a wide range of disciplines, such as Monoclonal antibody, In vivo and PRNP.
His Scrapie research is multidisciplinary, relying on both Pathogen, Microbiology, Proteinase K and Incubation.
Between 2006 and 2021, his most popular works were:
- Evidence for α-synuclein prions causing multiple system atrophy in humans with parkinsonism (377 citations)
- A Unifying Role for Prions in Neurodegenerative Diseases (362 citations)
- Biology and genetics of prions causing neurodegeneration. (294 citations)
In his most recent research, the most cited papers focused on:
Stanley B. Prusiner spends much of his time researching Virology, Biochemistry, Molecular biology, Amyloid and Genetically modified mouse.
His research integrates issues of Scrapie, Chronic wasting disease, Alpha-synuclein, Neurodegeneration and Gene isoform in his study of Virology.
The Scrapie study combines topics in areas such as Transport protein and Endosome.
In his work, Drug resistance is strongly intertwined with Cell culture, which is a subfield of Biochemistry.
His Molecular biology study incorporates themes from In vitro, Transgene, Prion infectivity, Neural crest and Bank vole.
His Amyloid study integrates concerns from other disciplines, such as Fungal protein, Saccharomyces cerevisiae and Recombinant DNA.
This overview was generated by a machine learning system which analysed the scientist’s
body of work. If you have any feedback, you can
contact us
here.
