2023 - Research.com Biology and Biochemistry in United Kingdom Leader Award
2005 - Fellow of the Royal Society, United Kingdom
Virology, Scrapie, Bovine spongiform encephalopathy, Genetics and PrPSc Proteins are his primary areas of study. His Virology research incorporates themes from Phenotype, Genetically modified mouse, Gene and Transgene. The Scrapie study combines topics in areas such as Spleen, Immunology, Hamster and Cell biology.
John Collinge has included themes like Creutzfeldt-Jakob Syndrome, Kuru, Genotype and Fatal familial insomnia in his Bovine spongiform encephalopathy study. His Genetics research focuses on subjects like Dementia, which are linked to Amyotrophic lateral sclerosis. His PrPSc Proteins research is multidisciplinary, relying on both Transmissible spongiform encephalopathy and In vitro.
His primary areas of investigation include Disease, Virology, Genetics, Scrapie and PRNP. His Disease research is multidisciplinary, incorporating elements of Transmission and Immunology. His work carried out in the field of Virology brings together such families of science as Creutzfeldt-Jakob Syndrome, Bovine spongiform encephalopathy, Prion protein, Fatal familial insomnia and PrPSc Proteins.
John Collinge has researched Bovine spongiform encephalopathy in several fields, including Transmissible spongiform encephalopathy, Genetically modified mouse and Kuru. His research is interdisciplinary, bridging the disciplines of Alzheimer's disease and Genetics. The concepts of his Scrapie study are interwoven with issues in In vitro, Incubation period and Antibody, Monoclonal antibody.
His primary areas of study are Disease, PRNP, Genetics, Internal medicine and Prion protein. His Disease study is related to the wider topic of Pathology. John Collinge usually deals with PRNP and limits it to topics linked to Kuru and Bovine spongiform encephalopathy.
Much of his study explores Genetics relationship to Family history. His studies in Prion protein integrate themes in fields like Biophysics and Virology. His Virology study integrates concerns from other disciplines, such as Genetically modified mouse, Variant cjd and Amyloid.
His primary scientific interests are in Disease, Neurodegeneration, Cell biology, Neurology and Creutzfeldt-Jakob Syndrome. His Disease research includes elements of Genome-wide association study and Bioinformatics. His Bovine spongiform encephalopathy study combines topics in areas such as Scrapie, Chronic wasting disease, Kuru, Cellular protein and Mutation.
His studies deal with areas such as Neurotoxicity, Neuroscience and Virology as well as Cellular protein. His Virology research integrates issues from Cell culture and Prion infectivity. His PRNP research is included under the broader classification of Genetics.
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Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease
Jean-Charles Lambert;Jean-Charles Lambert;Jean-Charles Lambert;Carla A Ibrahim-Verbaas;Denise Harold;Adam C Naj.
Nature Genetics (2013)
Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease
Denise Harold;Richard Abraham;Paul Hollingworth;Rebecca Sims.
Nature Genetics (2009)
Erratum: Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease (Nature Genetics (2009) 41 (1088-1093))
D Harold;R Abraham;P Hollingworth;R Sims.
Nature Genetics (2013)
TREM2 Variants in Alzheimer's Disease
Rita Guerreiro;Rita Guerreiro;Aleksandra Wojtas;Jose Bras;Minerva Carrasquillo.
The New England Journal of Medicine (2013)
Molecular analysis of prion strain variation and the aetiology of 'new variant' CJD
John Collinge;John Collinge;Katie C. L. Sidle;Julie Meads;James Ironside.
Nature (1996)
Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease.
Paul Hollingworth;Denise Harold;Rebecca Sims;Amy Gerrish.
Nature Genetics (2011)
The same prion strain causes vCJD and BSE
Andrew F. Hill;Melanie Desbruslais;Susan Joiner;Katie C. L. Sidle.
Nature (1997)
Prion Diseases of Humans and Animals: Their Causes and Molecular Basis
John Collinge.
Annual Review of Neuroscience (2001)
Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease.
Valentina Escott-Price;Céline Bellenguez;Li-San Wang;Seung-Hoan Choi.
PLOS ONE (2014)
A General Model of Prion Strains and Their Pathogenicity
John Collinge;Anthony R. Clarke.
Science (2007)
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