University College London
Fellow of The Academy of Medical Sciences, United Kingdom
Pathology, Dementia, Alzheimer's disease, Atrophy and Disease are his primary areas of study. His Pathology research incorporates themes from Frontal lobe and Gene mutation. His Dementia research includes elements of Cerebral cortex, Genetics, Psychiatry and Age of onset.
His Alzheimer's disease study is concerned with the larger field of Internal medicine. His Atrophy study combines topics from a wide range of disciplines, such as Magnetic resonance imaging, Brain size, Central nervous system disease and Temporal lobe. The study incorporates disciplines such as Biomarker, Neuroimaging and Bioinformatics in addition to Disease.
His primary scientific interests are in Pathology, Dementia, Disease, Alzheimer's disease and Atrophy. His Pathology research is multidisciplinary, incorporating elements of Frontal lobe, Temporal lobe and Central nervous system disease. His studies in Dementia integrate themes in fields like Psychiatry, Neurology, Gerontology and Pediatrics.
He has included themes like Biomarker, Cognition, Neuroscience and Bioinformatics in his Disease study. Martin N. Rossor has researched Alzheimer's disease in several fields, including Genetics, Apolipoprotein E, Endocrinology and Degenerative disease. His Atrophy study combines topics in areas such as Magnetic resonance imaging and Nuclear medicine.
Martin N. Rossor focuses on Dementia, Disease, Pathology, Neuroscience and Frontotemporal dementia. His biological study spans a wide range of topics, including Alzheimer's disease, Psychiatry, Pediatrics and Gerontology. His Alzheimer's disease research incorporates elements of Asymptomatic and Age of onset.
His Disease research includes themes of Biomarker and Cognition. His study in Pathology is interdisciplinary in nature, drawing from both White matter and Grey matter. His Atrophy study incorporates themes from Precuneus and Magnetic resonance imaging.
Martin N. Rossor focuses on Alzheimer's disease, Dementia, Pathology, Disease and Frontotemporal dementia. His work carried out in the field of Alzheimer's disease brings together such families of science as Biomarker, Psychiatry, Cognition and Age of onset. His work in the fields of Dementia, such as Cognitive decline, intersects with other areas such as Public relations.
His research integrates issues of White matter, Grey matter and Hyperintensity in his study of Pathology. The Disease study combines topics in areas such as Cross-sectional study, Asymptomatic and Neuroimaging. His work deals with themes such as Differential diagnosis, Neuroradiology and Neuroscience, which intersect with Frontotemporal dementia.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The diagnosis of dementia due to Alzheimer’s disease: Recommendations from the National Institute on Aging-Alzheimer’s Association workgroups on diagnostic guidelines for Alzheimer's disease
Guy M. McKhann;Guy M. McKhann;David S. Knopman;Howard Chertkow;Bradley T. Hyman.
Alzheimers & Dementia (2011)
Current concepts in mild cognitive impairment.
Ronald C. Petersen;Rachelle Doody;Alexander Kurz;Richard C. Mohs.
JAMA Neurology (2001)
Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's disease.
Alison Goate;Marie-Christine Chartier-Harlin;Mike Mullan;Jeremy Brown.
Research criteria for the diagnosis of Alzheimer's disease: revising the NINCDS–ADRDA criteria
Bruno Dubois;Howard H. Feldman;Claudia Jacova;Steven T. DeKosky.
Lancet Neurology (2007)
Sensitivity of revised diagnostic criteria for the behavioural variant of frontotemporal dementia.
Katya Rascovsky;John R. Hodges;David Knopman;Mario F. Mendez.
Clinical and Biomarker Changes in Dominantly Inherited Alzheimer’s Disease
Randall J. Bateman;Chengjie Xiong;Tammie L.S. Benzinger;Anne M. Fagan.
The New England Journal of Medicine (2012)
Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease
Denise Harold;Richard Abraham;Paul Hollingworth;Rebecca Sims.
Nature Genetics (2009)
Erratum: Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease (Nature Genetics (2009) 41 (1088-1093))
D Harold;R Abraham;P Hollingworth;R Sims.
Nature Genetics (2013)
Revising the definition of Alzheimer's disease: a new lexicon.
Bruno Dubois;Howard H. Feldman;Howard H. Feldman;Howard H. Feldman;Claudia Jacova;Jeffrey L. Cummings.
Lancet Neurology (2010)
Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease.
Paul Hollingworth;Denise Harold;Rebecca Sims;Amy Gerrish.
Nature Genetics (2011)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: