D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 43 Citations 4,794 85 World Ranking 4255 National Ranking 181

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Neurotransmitter
  • Neuroscience

Jean-Marie Billard mostly deals with Neuroscience, Synaptic plasticity, Long-term potentiation, Inhibitory postsynaptic potential and NMDA receptor. He conducts interdisciplinary study in the fields of Neuroscience and Efferent through his research. Jean-Marie Billard interconnects Glutamate receptor, Glutamatergic and Memory consolidation in the investigation of issues within Synaptic plasticity.

His Long-term potentiation study combines topics from a wide range of disciplines, such as Hippocampal formation, Hippocampus, Neurotransmitter and Cognitive flexibility. His Inhibitory postsynaptic potential research includes elements of Inferior olivary nucleus, Stimulus, GABAA receptor, Membrane potential and CGP-35348. His work in NMDA receptor tackles topics such as Neurotransmission which are related to areas like Somatostatin receptor.

His most cited work include:

  • Contribution of the d-Serine-Dependent Pathway to the Cellular Mechanisms Underlying Cognitive Aging. (141 citations)
  • Deficits in memory and hippocampal long-term potentiation in mice with reduced calbindin D28K expression (138 citations)
  • Neuronal d-Serine and Glycine Release Via the Asc-1 Transporter Regulates NMDA Receptor-Dependent Synaptic Activity (135 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Neuroscience, Long-term potentiation, Synaptic plasticity, NMDA receptor and Hippocampus. His studies deal with areas such as Glutamatergic and Postsynaptic potential as well as Neuroscience. The concepts of his Long-term potentiation study are interwoven with issues in Neurotransmitter, Neurotransmission, Hippocampal formation and AMPA receptor, Long-term depression.

His work deals with themes such as Serine racemase, Duchenne muscular dystrophy and Memory consolidation, which intersect with Synaptic plasticity. His NMDA receptor research focuses on Endocrinology and how it connects with Calcium. The study incorporates disciplines such as Oxidative stress, Aging brain and Transgene in addition to Hippocampus.

He most often published in these fields:

  • Neuroscience (77.50%)
  • Long-term potentiation (57.50%)
  • Synaptic plasticity (45.00%)

What were the highlights of his more recent work (between 2015-2020)?

  • Long-term potentiation (57.50%)
  • Neuroscience (77.50%)
  • Hippocampal formation (37.50%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Long-term potentiation, Neuroscience, Hippocampal formation, NMDA receptor and Cell biology. His research integrates issues of Oxidative stress, Endocrinology, Hippocampus and Antioxidant in his study of Long-term potentiation. His work in Neuroscience covers topics such as Glutamate receptor which are related to areas like Neurotransmission, Ageing and Nervous system.

The Hippocampal formation study combines topics in areas such as Alzheimer's disease, Presenilin, Amyloid precursor protein and Glutamatergic. NMDA receptor is frequently linked to Transporter in his study. His Cell biology study combines topics in areas such as Synaptic plasticity and Mutation.

Between 2015 and 2020, his most popular works were:

  • Asc-1 Transporter Regulation of Synaptic Activity via the Tonic Release of d-Serine in the Forebrain (39 citations)
  • Deleting IGF-1 receptor from forebrain neurons confers neuroprotection during stroke and upregulates endocrine somatotropin. (25 citations)
  • Alzheimer’s disease-like APP processing in wild-type mice identifies synaptic defects as initial steps of disease progression (24 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Neurotransmitter
  • Internal medicine

Jean-Marie Billard spends much of his time researching Neuroprotection, Neuroscience, Long-term potentiation, Alzheimer's disease and Presenilin. His Neuroprotection study integrates concerns from other disciplines, such as Growth factor, Insulin-like growth factor, Neuroinflammation, Neurogenesis and Growth factor receptor. His research in the fields of Forebrain overlaps with other disciplines such as Somatotropic cell.

His Long-term potentiation research integrates issues from Extracellular, Cell biology and NMDA receptor, AMPA receptor. His study in Alzheimer's disease is interdisciplinary in nature, drawing from both Hippocampal formation and Glutamatergic.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Contribution of the d-Serine-Dependent Pathway to the Cellular Mechanisms Underlying Cognitive Aging.

Brigitte Potier;Fabrice R Turpin;Pierre-Marie Sinet;Emilie Rouaud.
Frontiers in Aging Neuroscience (2010)

312 Citations

Neuronal d-Serine and Glycine Release Via the Asc-1 Transporter Regulates NMDA Receptor-Dependent Synaptic Activity

Dina Rosenberg;Samar Artoul;Adi C. Segal;Goren Kolodney.
The Journal of Neuroscience (2013)

191 Citations

A critical role for the glial-derived neuromodulator D-serine in the age-related deficits of cellular mechanisms of learning and memory.

J. P. Mothet;E. Rouaud;P.-M. Sinet;B. Potier.
Aging Cell (2006)

179 Citations

Deficits in memory and hippocampal long-term potentiation in mice with reduced calbindin D28K expression

S Molinari;R Battini;S Ferrari;L Pozzi.
Proceedings of the National Academy of Sciences of the United States of America (1996)

166 Citations

Release of cerebellar inhibition by climbing fiber deafferentation.

C. Batini;J. M. Billard.
Experimental Brain Research (1985)

161 Citations

Impaired long-term spatial and recognition memory and enhanced CA1 hippocampal LTP in the dystrophin-deficient Dmdmdx mouse

Cyrille Vaillend;Jean-Marie Billard;Serge Laroche.
Neurobiology of Disease (2004)

158 Citations

Role of Mental Retardation-Associated Dystrophin-Gene Product Dp71 in Excitatory Synapse Organization, Synaptic Plasticity and Behavioral Functions

Fatma Daoud;Aurora Candelario-Martínez;Jean Marie Billard;Avi Avital;Avi Avital.
PLOS ONE (2009)

127 Citations

The red nucleus activity in rats deprived of the inferior olivary complex

J.M. Billard;C. Batini;H. Daniel.
Behavioural Brain Research (1988)

120 Citations

Parallel loss of hippocampal LTD and cognitive flexibility in a genetic model of hyperdopaminergia

Elise Morice;Jean-Marie Billard;Cécile Denis;Flavie Mathieu.
Neuropsychopharmacology (2007)

119 Citations

Involvement of sst2 somatostatin receptor in locomotor, exploratory activity and emotional reactivity in mice.

C. Viollet;Cyrille Vaillend;C. Videau;M.T. Bluet-Pajot.
European Journal of Neuroscience (2000)

118 Citations

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