Thomas R. Hawn mostly deals with Immunology, Innate immune system, Immune system, Toll-like receptor and Inflammation. Thomas R. Hawn has researched Immunology in several fields, including Mycobacterium marinum, Mycobacterium tuberculosis, Disease and Genotype. His work carried out in the field of Innate immune system brings together such families of science as Proinflammatory cytokine and Microbiology.
Thomas R. Hawn frequently studies issues relating to Flagellin and Immune system. His work on TIRAP as part of general Toll-like receptor study is frequently linked to Vascular resistance, therefore connecting diverse disciplines of science. Thomas R. Hawn combines subjects such as Virulence factor and Flagellum with his study of TIRAP.
His primary areas of investigation include Immunology, Immune system, Mycobacterium tuberculosis, Innate immune system and Tuberculosis. His studies in Immunology integrate themes in fields like Odds ratio and Disease. His Immune system research is multidisciplinary, relying on both Inflammation, Legionella pneumophila and Mycobacterium leprae.
His study in Innate immune system is interdisciplinary in nature, drawing from both Proinflammatory cytokine, Cytokine, Microbiology and TLR4. The concepts of his Microbiology study are interwoven with issues in Chemokine, Signal transduction and Flagellin. Thomas R. Hawn studies TIRAP, a branch of Toll-like receptor.
Thomas R. Hawn focuses on Mycobacterium tuberculosis, Tuberculosis, Immunology, Tuberculin and Internal medicine. The study incorporates disciplines such as Genetics, Microbiology and Virulence in addition to Mycobacterium tuberculosis. His Microbiology research includes themes of TLR5 and Bacteria.
His research in the fields of Tuberculosis vaccines and Mycobacterium bovis overlaps with other disciplines such as Library science and Political science. His work deals with themes such as Chemoprophylaxis, Cystic fibrosis and Disease, which intersect with Immunology. Thomas R. Hawn studied Internal medicine and Latent tuberculosis that intersect with Logistic regression, Interquartile range and Gastroenterology.
His primary areas of study are Tuberculosis, Mycobacterium tuberculosis, Intensive care medicine, Tuberculin and Repurposing. His Tuberculosis study combines topics in areas such as Antibody, Avidity and Antigen. Thomas R. Hawn has included themes like Chemoprophylaxis, Internal medicine, Immunology and Public health in his Mycobacterium tuberculosis study.
His Intensive care medicine research is multidisciplinary, incorporating perspectives in Vaccination against tuberculosis, Tuberculosis vaccines, Transmission, Tuberculosis Disease and Bacille Calmette Guerin. His biological study spans a wide range of topics, including Epidemiology, Interferon gamma release assay, Latent tuberculosis, Concordance and Asymptomatic. His Repurposing investigation overlaps with other disciplines such as Drug development, Host organism, Treatment duration, Immune defense and Tissue damage.
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The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5.
Fumitaka Hayashi;Kelly D. Smith;Adrian Ozinsky;Thomas R. Hawn;Thomas R. Hawn.
A Common Dominant TLR5 Stop Codon Polymorphism Abolishes Flagellin Signaling and Is Associated with Susceptibility to Legionnaires' Disease
Thomas R. Hawn;Annelies Verbon;Annelies Verbon;Kamilla D. Lettinga;Lue Ping Zhao.
Journal of Experimental Medicine (2003)
Toll-Like Receptor-4 Mediates Vascular Inflammation and Insulin Resistance in Diet-Induced Obesity
Francis Kim;Matilda Pham;Ian Luttrell;Douglas D. Bannerman.
Circulation Research (2007)
The influence of host and bacterial genotype on the development of disseminated disease with Mycobacterium tuberculosis
Maxine Caws;Maxine Caws;Guy Thwaites;Sarah Dunstan;Sarah Dunstan;Thomas R. Hawn.
PLOS Pathogens (2008)
Host Genotype-Specific Therapies Can Optimize the Inflammatory Response to Mycobacterial Infections
David M. Tobin;Francisco J. Roca;Sungwhan F. Oh;Ross McFarland.
The lta4h Locus Modulates Susceptibility to Mycobacterial Infection in Zebrafish and Humans
David M. Tobin;Jay C. Vary;John P. Ray;Gregory S. Walsh.
Toll-like receptor polymorphisms and susceptibility to human disease.
E. Ann Misch;Thomas R. Hawn.
Clinical Science (2008)
Cutting Edge: A Toll-Like Receptor 2 Polymorphism That Is Associated with Lepromatous Leprosy Is Unable to Mediate Mycobacterial Signaling
Pierre-Yves Bochud;Thomas R. Hawn;Thomas R. Hawn;Alan Aderem;Alan Aderem.
Journal of Immunology (2003)
Mycobacterium tuberculosis, macrophages, and the innate immune response: does common variation matter?
William R. Berrington;Thomas R. Hawn.
Immunological Reviews (2007)
A common human TLR1 polymorphism regulates the innate immune response to lipopeptides
Thomas R. Hawn;Thomas R. Hawn;E. Ann Misch;Sarah J. Dunstan;Guy E. Thwaites.
European Journal of Immunology (2007)
Profile was last updated on December 6th, 2021.
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