D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 54 Citations 19,579 83 World Ranking 2399 National Ranking 1157

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Internal medicine

Thomas R. Hawn mostly deals with Immunology, Innate immune system, Immune system, Toll-like receptor and Inflammation. Thomas R. Hawn has researched Immunology in several fields, including Mycobacterium marinum, Mycobacterium tuberculosis, Disease and Genotype. His work carried out in the field of Innate immune system brings together such families of science as Proinflammatory cytokine and Microbiology.

Thomas R. Hawn frequently studies issues relating to Flagellin and Immune system. His work on TIRAP as part of general Toll-like receptor study is frequently linked to Vascular resistance, therefore connecting diverse disciplines of science. Thomas R. Hawn combines subjects such as Virulence factor and Flagellum with his study of TIRAP.

His most cited work include:

  • The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5. (2988 citations)
  • The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5. (2988 citations)
  • A Common Dominant TLR5 Stop Codon Polymorphism Abolishes Flagellin Signaling and Is Associated with Susceptibility to Legionnaires' Disease (528 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Immunology, Immune system, Mycobacterium tuberculosis, Innate immune system and Tuberculosis. His studies in Immunology integrate themes in fields like Odds ratio and Disease. His Immune system research is multidisciplinary, relying on both Inflammation, Legionella pneumophila and Mycobacterium leprae.

His study in Innate immune system is interdisciplinary in nature, drawing from both Proinflammatory cytokine, Cytokine, Microbiology and TLR4. The concepts of his Microbiology study are interwoven with issues in Chemokine, Signal transduction and Flagellin. Thomas R. Hawn studies TIRAP, a branch of Toll-like receptor.

He most often published in these fields:

  • Immunology (82.76%)
  • Immune system (42.24%)
  • Mycobacterium tuberculosis (31.90%)

What were the highlights of his more recent work (between 2018-2021)?

  • Mycobacterium tuberculosis (31.90%)
  • Tuberculosis (29.31%)
  • Immunology (82.76%)

In recent papers he was focusing on the following fields of study:

Thomas R. Hawn focuses on Mycobacterium tuberculosis, Tuberculosis, Immunology, Tuberculin and Internal medicine. The study incorporates disciplines such as Genetics, Microbiology and Virulence in addition to Mycobacterium tuberculosis. His Microbiology research includes themes of TLR5 and Bacteria.

His research in the fields of Tuberculosis vaccines and Mycobacterium bovis overlaps with other disciplines such as Library science and Political science. His work deals with themes such as Chemoprophylaxis, Cystic fibrosis and Disease, which intersect with Immunology. Thomas R. Hawn studied Internal medicine and Latent tuberculosis that intersect with Logistic regression, Interquartile range and Gastroenterology.

Between 2018 and 2021, his most popular works were:

  • Bacteriophage trigger antiviral immunity and prevent clearance of bacterial infection (115 citations)
  • IFN-γ-independent immune markers of Mycobacterium tuberculosis exposure. (78 citations)
  • Remembering the Host in Tuberculosis Drug Development. (17 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Internal medicine

His primary areas of study are Tuberculosis, Mycobacterium tuberculosis, Intensive care medicine, Tuberculin and Repurposing. His Tuberculosis study combines topics in areas such as Antibody, Avidity and Antigen. Thomas R. Hawn has included themes like Chemoprophylaxis, Internal medicine, Immunology and Public health in his Mycobacterium tuberculosis study.

His Intensive care medicine research is multidisciplinary, incorporating perspectives in Vaccination against tuberculosis, Tuberculosis vaccines, Transmission, Tuberculosis Disease and Bacille Calmette Guerin. His biological study spans a wide range of topics, including Epidemiology, Interferon gamma release assay, Latent tuberculosis, Concordance and Asymptomatic. His Repurposing investigation overlaps with other disciplines such as Drug development, Host organism, Treatment duration, Immune defense and Tissue damage.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5.

Fumitaka Hayashi;Kelly D. Smith;Adrian Ozinsky;Thomas R. Hawn;Thomas R. Hawn.
Nature (2001)

4114 Citations

A Common Dominant TLR5 Stop Codon Polymorphism Abolishes Flagellin Signaling and Is Associated with Susceptibility to Legionnaires' Disease

Thomas R. Hawn;Annelies Verbon;Annelies Verbon;Kamilla D. Lettinga;Lue Ping Zhao.
Journal of Experimental Medicine (2003)

717 Citations

Toll-Like Receptor-4 Mediates Vascular Inflammation and Insulin Resistance in Diet-Induced Obesity

Francis Kim;Matilda Pham;Ian Luttrell;Douglas D. Bannerman.
Circulation Research (2007)

625 Citations

The influence of host and bacterial genotype on the development of disseminated disease with Mycobacterium tuberculosis

Maxine Caws;Maxine Caws;Guy Thwaites;Sarah Dunstan;Sarah Dunstan;Thomas R. Hawn.
PLOS Pathogens (2008)

579 Citations

Host Genotype-Specific Therapies Can Optimize the Inflammatory Response to Mycobacterial Infections

David M. Tobin;Francisco J. Roca;Sungwhan F. Oh;Ross McFarland.
Cell (2012)

536 Citations

The lta4h Locus Modulates Susceptibility to Mycobacterial Infection in Zebrafish and Humans

David M. Tobin;Jay C. Vary;John P. Ray;Gregory S. Walsh.
Cell (2010)

520 Citations

Toll-like receptor polymorphisms and susceptibility to human disease.

E. Ann Misch;Thomas R. Hawn.
Clinical Science (2008)

333 Citations

Cutting Edge: A Toll-Like Receptor 2 Polymorphism That Is Associated with Lepromatous Leprosy Is Unable to Mediate Mycobacterial Signaling

Pierre-Yves Bochud;Thomas R. Hawn;Thomas R. Hawn;Alan Aderem;Alan Aderem.
Journal of Immunology (2003)

320 Citations

Mycobacterium tuberculosis, macrophages, and the innate immune response: does common variation matter?

William R. Berrington;Thomas R. Hawn.
Immunological Reviews (2007)

248 Citations

A common human TLR1 polymorphism regulates the innate immune response to lipopeptides

Thomas R. Hawn;Thomas R. Hawn;E. Ann Misch;Sarah J. Dunstan;Guy E. Thwaites.
European Journal of Immunology (2007)

221 Citations

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Profile was last updated on December 6th, 2021.
Research.com Ranking is based on data retrieved from the Microsoft Academic Graph (MAG).
The ranking d-index is inferred from publications deemed to belong to the considered discipline.

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