His main research concerns Immunology, Microbiology, Cytokine, Toll-like receptor and Innate immune system. His Immunology study combines topics from a wide range of disciplines, such as Legionella pneumophila, Respiratory disease and Macrophage. His Respiratory disease study also includes
The Microbiology study combines topics in areas such as Francisella tularensis, Virulence, Secretion, Signal transduction and Myeloid Differentiation Factor 88. His Cytokine study integrates concerns from other disciplines, such as Inflammation and Lung. His Lung research focuses on Epithelium and how it relates to Lipopolysaccharide.
His scientific interests lie mostly in Immunology, Microbiology, Lung, Innate immune system and Immune system. The study incorporates disciplines such as Legionella pneumophila and Bronchoalveolar lavage in addition to Immunology. His research in Microbiology intersects with topics in Burkholderia pseudomallei, Francisella tularensis, Virulence, Burkholderia and Signal transduction.
His biological study deals with issues like Pneumonia, which deal with fields such as Respiratory disease, Intensive care medicine and Inhalation. His research integrates issues of Lipopolysaccharide, Immunity and Staphylococcus aureus in his study of Innate immune system. The various areas that Shawn J. Skerrett examines in his Immune system study include Respiratory infection and Antibiotics.
The scientist’s investigation covers issues in Microbiology, Alveolar macrophage, Antibiotics, Intracellular and Pharmacology. His Microbiology research is multidisciplinary, relying on both Pyrazinamide, Mycobacterium tuberculosis, Immune system, Macrophage and Lung disease. His work focuses on many connections between Pharmacology and other disciplines, such as Ciprofloxacin, that overlap with his field of interest in Sepsis, Minimum inhibitory concentration, Pharmacokinetics, Lung and Francisella tularensis.
Immunity is a subfield of Immunology that Shawn J. Skerrett investigates. Shawn J. Skerrett undertakes interdisciplinary study in the fields of Immunology and Cytometry through his works. Shawn J. Skerrett works mostly in the field of Tumor necrosis factor alpha, limiting it down to topics relating to Innate immune system and, in certain cases, Inflammation.
His primary scientific interests are in Intracellular, Pharmacology, Antibiotics, Alveolar macrophage and Intracellular parasite. His study on Intracellular is mostly dedicated to connecting different topics, such as Secretion. In his work, Lung, Francisella tularensis, Sepsis, In vivo and Minimum inhibitory concentration is strongly intertwined with Ciprofloxacin, which is a subfield of Pharmacology.
His studies link Francisella with Lung. His Alveolar macrophage study combines topics in areas such as ARDS, CD163, Immunology and Macrophage polarization. Shawn J. Skerrett interconnects Streptomycin, Hemolysis and Liposome in the investigation of issues within Intracellular parasite.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
A Common Dominant TLR5 Stop Codon Polymorphism Abolishes Flagellin Signaling and Is Associated with Susceptibility to Legionnaires' Disease
Thomas R. Hawn;Annelies Verbon;Annelies Verbon;Kamilla D. Lettinga;Lue Ping Zhao.
Journal of Experimental Medicine (2003)
Cytokine Balance in the Lungs of Patients with Acute Respiratory Distress Syndrome
William Y. Park;Richard B. Goodman;Kenneth P. Steinberg;John T. Ruzinski.
American Journal of Respiratory and Critical Care Medicine (2001)
Management of the critically ill patient with severe acute pancreatitis.
Avery B. Nathens;J. Randall Curtis;Richard J. Beale;Deborah J. Cook.
Critical Care Medicine (2004)
Burkholderia Type VI Secretion Systems Have Distinct Roles in Eukaryotic and Bacterial Cell Interactions
Sandra Schwarz;T. Eoin West;Frédéric Boyer;Wen Chi Chiang.
PLOS Pathogens (2010)
Clinical Features and Analysis of Risk Factors for Invasive Candidal Infection after Marrow Transplantation
James M. Goodrich;James M. Goodrich;Elizabeth Cecile Reed;Motomi Mori;Lloyd D. Fisher.
The Journal of Infectious Diseases (1991)
Cutting Edge: Myeloid Differentiation Factor 88 Is Essential for Pulmonary Host Defense against Pseudomonas aeruginosa but Not Staphylococcus aureus
Shawn J. Skerrett;H. Denny Liggitt;Adeline M. Hajjar;Christopher B. Wilson.
Journal of Immunology (2004)
Fas (CD95) induces proinflammatory cytokine responses by human monocytes and monocyte-derived macrophages
David R. Park;Anni R. Thomsen;Charles W. Frevert;Uyenvy Pham.
Journal of Immunology (2003)
Lack of In Vitro and In Vivo Recognition of Francisella tularensis Subspecies Lipopolysaccharide by Toll-Like Receptors
Adeline M. Hajjar;Megan D. Harvey;Scott A. Shaffer;David R. Goodlett.
Infection and Immunity (2006)
Respiratory epithelial cells regulate lung inflammation in response to inhaled endotoxin.
Shawn J. Skerrett;H. Denny Liggitt;Adeline M. Hajjar;Robert K. Ernst.
American Journal of Physiology-lung Cellular and Molecular Physiology (2004)
Type IV pili‐mediated secretion modulates Francisella virulence
Anthony J. Hager;Diana L. Bolton;Mark R. Pelletier;Mitchell J. Brittnacher.
Molecular Microbiology (2006)
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