The scientist’s investigation covers issues in Cytotoxic T cell, T cell, Immunology, Cell biology and Interleukin 21. IL-2 receptor, CD28 and Antigen-presenting cell are subfields of Cytotoxic T cell in which her conducts study. In her research, In vivo and CD137 is intimately related to Virus, which falls under the overarching field of CD28.
Her Antigen-presenting cell study deals with Natural killer T cell intersecting with CD40. The study of T cell is intertwined with the study of Molecular biology in a number of ways. Her ZAP70 research extends to Cell biology, which is thematically connected.
Tania H. Watts mostly deals with Immunology, Cytotoxic T cell, T cell, Cell biology and Immune system. Her Immunology study frequently draws connections between related disciplines such as Receptor. IL-2 receptor, Interleukin 21, ZAP70, Antigen-presenting cell and Natural killer T cell are among the areas of Cytotoxic T cell where Tania H. Watts concentrates her study.
Her Antigen-presenting cell study incorporates themes from Antigen presentation and CD40. Tania H. Watts has researched T cell in several fields, including Molecular biology and CD8, Antigen. Her biological study spans a wide range of topics, including Tumor necrosis factor alpha, Inflammation and Antibody.
Her primary areas of investigation include T cell, Immunology, Cytotoxic T cell, Effector and Memory T cell. Her work on CD137 as part of general T cell research is frequently linked to P110δ, bridging the gap between disciplines. She regularly links together related areas like Cancer in her Immunology studies.
As part of her studies on Cytotoxic T cell, Tania H. Watts often connects relevant subjects like Virology. In her work, CD80 and Inflammation is strongly intertwined with Immune system, which is a subfield of Effector. Her work in Antigen-presenting cell addresses subjects such as Antigen presentation, which are connected to disciplines such as Molecular biology.
Her primary areas of study are T cell, Cancer research, Inflammation, Acquired immune system and Memory T cell. Her work deals with themes such as Signal transduction and Immunophenotyping, which intersect with T cell. Her Inflammation study combines topics from a wide range of disciplines, such as Innate immune system, Immune system and Immunity.
Her Innate immune system research is classified as research in Immunology. Her Immunology research incorporates themes from Agonist and mTORC1. Her research integrates issues of TRAF1 and Kinase in her study of Cytotoxic T cell.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
TNF/TNFR FAMILY MEMBERS IN COSTIMULATION OF T CELL RESPONSES
Tania H Watts.
Annual Review of Immunology (2005)
Supported planar membranes in studies of cell-cell recognition in the immune system.
H.M. McConnell;T.H. Watts;R.M. Weis;A.A. Brian.
Biochimica et Biophysica Acta (1986)
The B7 family member B7-H3 preferentially down-regulates T helper type 1-mediated immune responses
Woong-Kyung Suh;Beata U Gajewska;Hitoshi Okada;Matthew A Gronski.
Nature Immunology (2003)
T cell co-stimulatory molecules other than CD28
Tania H Watts;Mark A DeBenedettet.
Current Opinion in Immunology (1999)
4-1BB Ligand Induces Cell Division, Sustains Survival, and Enhances Effector Function of CD4 and CD8 T Cells with Similar Efficacy
Jennifer L. Cannons;Peggy Lau;Birinder Ghumman;Mark A. DeBenedette.
Journal of Immunology (2001)
Dendritic cell-induced autoimmune heart failure requires cooperation between adaptive and innate immunity.
Urs Eriksson;Romeo Ricci;Lukas Hunziker;Michael O Kurrer.
Nature Medicine (2003)
Costimulation of CD28- T lymphocytes by 4-1BB ligand.
M A DeBenedette;A Shahinian;T W Mak;T H Watts.
Journal of Immunology (1997)
Analysis of 4-1BB Ligand (4-1BBL)-Deficient Mice and of Mice Lacking Both 4-1BBL and CD28 Reveals a Role for 4-1BBL in Skin Allograft Rejection and in the Cytotoxic T Cell Response to Influenza Virus
Mark A. DeBenedette;Tao Wen;Martin F. Bachmann;Pamela S. Ohashi.
Journal of Immunology (1999)
CD28-INDEPENDENT, TRAF2-DEPENDENT COSTIMULATION OF RESTING T CELLS BY 4-1BB LIGAND
Katina Saoulli;Soo Young Lee;Jennifer L. Cannons;Wen Chen Yeh.
Journal of Experimental Medicine (1998)
Temporal segregation of 4-1BB versus CD28-mediated costimulation: 4-1BB ligand influences T cell numbers late in the primary response and regulates the size of the T cell memory response following influenza infection
Edward M. Bertram;Peggy Lau;Tania H. Watts.
Journal of Immunology (2002)
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