1959 - Fellow of the American Association for the Advancement of Science (AAAS)
His scientific interests lie mostly in Cancer research, Molecular biology, MYB, Cell biology and Stem cell. His study looks at the relationship between Cancer research and fields such as Gene expression, as well as how they intersect with chemical problems. The various areas that Robert G. Ramsay examines in his Molecular biology study include Cell culture, Haematopoiesis, Cell cycle and Mutant.
His MYB research incorporates elements of Mutagenesis and Transcription. His research investigates the link between Cell biology and topics such as Adult stem cell that cross with problems in Intestinal epithelium and Cancer stem cell. His Stem cell study focuses on Progenitor cell in particular.
Robert G. Ramsay mainly investigates Cancer research, MYB, Colorectal cancer, Molecular biology and Cell biology. His biological study spans a wide range of topics, including Wnt signaling pathway, Intestinal mucosa, Carcinogenesis, Transcription factor and Adenomatous polyposis coli. His MYB research is multidisciplinary, incorporating perspectives in Transcription, Oncogene and Estrogen receptor.
His study in Molecular biology is interdisciplinary in nature, drawing from both Cell culture, Haematopoiesis, Transgene, Cell growth and Cell cycle. Robert G. Ramsay is interested in Stem cell, which is a field of Cell biology. Robert G. Ramsay studied Stem cell and Cellular differentiation that intersect with Gene expression.
Robert G. Ramsay focuses on Colorectal cancer, Cancer research, Internal medicine, Hyperthermic intraperitoneal chemotherapy and Oncology. His study on Colorectal cancer also encompasses disciplines like
Robert G. Ramsay combines subjects such as Inhibitor of apoptosis, Organoid, Gene signature, PI3K/AKT/mTOR pathway and Immunotherapy with his study of Cancer research. Robert G. Ramsay interconnects Gastroenterology and DNA in the investigation of issues within Internal medicine. His work carried out in the field of Oncology brings together such families of science as Meta-analysis, Cancer, Clinical trial and MEDLINE.
Robert G. Ramsay mainly focuses on Hyperthermic intraperitoneal chemotherapy, Internal medicine, Oncology, Colorectal cancer and Perioperative. As part of the same scientific family, Robert G. Ramsay usually focuses on Hyperthermic intraperitoneal chemotherapy, concentrating on Peritoneal Neoplasm and intersecting with Survival rate. His Oncology study combines topics in areas such as Targeted therapy and Immunotherapy.
His research in Colorectal cancer intersects with topics in Meta-analysis, Randomized controlled trial, Chemotherapy and MEDLINE. The Chemotherapy study which covers Treatment modality that intersects with Cancer research. His research integrates issues of Cell signaling and Transcription factor in his study of Cancer research.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
MYB function in normal and cancer cells.
Robert G. Ramsay;Thomas J. Gonda.
Nature Reviews Cancer (2008)
BET inhibitor resistance emerges from leukaemia stem cells
Chun Yew Fong;Omer Gilan;Omer Gilan;Enid Y N Lam;Alan Rubin.
RAD21 Mutations Cause a Human Cohesinopathy
Matthew A. Deardorff;Matthew A. Deardorff;Jonathan J. Wilde;Melanie Albrecht;Emma Dickinson.
American Journal of Human Genetics (2012)
Changes in gene expression associated with induced differentiation of erythroleukemia: protooncogenes, globin genes, and cell division.
Robert G. Ramsay;Kazuma Ikeda;Richard A. Rifkind;Paul A. Marks.
Proceedings of the National Academy of Sciences of the United States of America (1986)
Suppressor screen in Mpl-/- mice: c-Myb mutation causes supraphysiological production of platelets in the absence of thrombopoietin signaling
Marina R. Carpinelli;Douglas J. Hilton;Donald Metcalf;Jennifer L. Antonchuk.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Cohesin regulates tissue-specific expression by stabilizing highly occupied cis-regulatory modules
Andre J. Faure;Dominic Schmidt;Stephen Watt;Petra C. Schwalie.
Genome Research (2012)
c-Myb and Bcl-x overexpression predicts poor prognosis in colorectal cancer: clinical and experimental findings.
Annamaria Biroccio;Barbara Benassi;Igea D’Agnano;Carmen D’Angelo.
American Journal of Pathology (2001)
Two isoforms of murine hck, generated by utilization of alternative translational initiation codons, exhibit different patterns of subcellular localization.
P Lock;S Ralph;E Stanley;I Boulet.
Molecular and Cellular Biology (1991)
Transactivation and transformation by Myb are negatively regulated by a leucine-zipper structure
Chie Kanei-Ishii;E. M. Macmillan;T. Nomura;A. Sarai.
Proceedings of the National Academy of Sciences of the United States of America (1992)
Mechanism of and requirement for estrogen-regulated MYB expression in estrogen-receptor-positive breast cancer cells.
Yvette Drabsch;Honor Hugo;Rui Zhang;Dennis H. Dowhan.
Proceedings of the National Academy of Sciences of the United States of America (2007)
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