D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 94 Citations 30,748 321 World Ranking 4866 National Ranking 159

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Enzyme

Cancer research, Epithelial–mesenchymal transition, Cancer, Pathology and Mesenchymal stem cell are his primary areas of study. His Cancer research research includes themes of Cell culture, CD44, Cell growth, Immunology and Signal transduction. His Immunology research is multidisciplinary, relying on both Organ development, Gene targets, Disease and Neoplasm Invasiveness.

Epithelial–mesenchymal transition is a primary field of his research addressed under Metastasis. His Cancer study combines topics in areas such as Phenotype, Cell and Epidermal growth factor. His research integrates issues of Muscle hypertrophy, Tibia, Matrix metalloproteinase and In vivo in his study of Pathology.

His most cited work include:

  • The epithelial–mesenchymal transition: new insights in signaling, development, and disease (1642 citations)
  • The epithelial–mesenchymal transition: new insights in signaling, development, and disease (1642 citations)
  • Epithelial--mesenchymal and mesenchymal--epithelial transitions in carcinoma progression. (876 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Cancer research, Pathology, Epithelial–mesenchymal transition, Metastasis and Cancer. His Cancer research study combines topics from a wide range of disciplines, such as Cell culture, Cell, Cancer cell, Immunology and Breast cancer. His Pathology study integrates concerns from other disciplines, such as Downregulation and upregulation and In vivo.

His study in Epithelial–mesenchymal transition is interdisciplinary in nature, drawing from both Epidermal growth factor, Mesenchymal stem cell, Cell biology, Vimentin and Tumor progression. The Metastasis study combines topics in areas such as Cancer stem cell, Carcinoma and Angiogenesis. Erik W. Thompson interconnects Phenotype and Endocrinology in the investigation of issues within Cancer.

He most often published in these fields:

  • Cancer research (53.80%)
  • Pathology (42.41%)
  • Epithelial–mesenchymal transition (41.30%)

What were the highlights of his more recent work (between 2014-2021)?

  • Cancer research (53.80%)
  • Epithelial–mesenchymal transition (41.30%)
  • Breast cancer (34.65%)

In recent papers he was focusing on the following fields of study:

His primary scientific interests are in Cancer research, Epithelial–mesenchymal transition, Breast cancer, Metastasis and Internal medicine. The various areas that he examines in his Cancer research study include Cancer, Tumor progression, Cell, Regulator and Mesenchymal stem cell. His Epithelial–mesenchymal transition research is multidisciplinary, incorporating elements of Epidermal growth factor, Gene expression profiling, Cell biology, Vimentin and MDA-MB-468.

His Breast cancer research incorporates themes from Carcinoma in situ, Signal transduction and Pathology. His study looks at the relationship between Metastasis and topics such as Cancer cell, which overlap with Cell adhesion and Reactive oxygen species. His work deals with themes such as Endocrinology and Oncology, which intersect with Internal medicine.

Between 2014 and 2021, his most popular works were:

  • Guidelines and definitions for research on epithelial–mesenchymal transition (174 citations)
  • Controversies around epithelial–mesenchymal plasticity in cancer metastasis (93 citations)
  • Controversies around epithelial–mesenchymal plasticity in cancer metastasis (93 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Enzyme

Erik W. Thompson spends much of his time researching Cancer research, Epithelial–mesenchymal transition, Metastasis, Breast cancer and Internal medicine. His biological study spans a wide range of topics, including Cancer, Tumor progression, Epidermal growth factor, Cell and Immunology. His Epithelial–mesenchymal transition research includes elements of Cadherin, Gene expression profiling, Cell biology, Regulator and Transforming growth factor beta.

His work carried out in the field of Metastasis brings together such families of science as Cancer cell, Phenotype, Mesenchymal stem cell and Stem cell. The concepts of his Breast cancer study are interwoven with issues in Clinical trial, Pathology, Intermittent hypoxia, Downregulation and upregulation and Signal transduction. His Internal medicine study incorporates themes from Endocrinology and Oncology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The epithelial–mesenchymal transition: new insights in signaling, development, and disease

Jonathan M. Lee;Shoukat Dedhar;Raghu Kalluri;Raghu Kalluri;Raghu Kalluri;Erik W. Thompson;Erik W. Thompson.
Journal of Cell Biology (2006)

2378 Citations

Epithelial--mesenchymal and mesenchymal--epithelial transitions in carcinoma progression.

Honor Hugo;M. Leigh Ackland;Tony Blick;Mitchell G. Lawrence.
Journal of Cellular Physiology (2007)

1266 Citations

Carcinoma Invasion and Metastasis: A Role for Epithelial-Mesenchymal Transition?

Erik W. Thompson;Donald F. Newgreen.
Cancer Research (2005)

831 Citations

The fallacy of epithelial mesenchymal transition in neoplasia.

David Tarin;Erik W Thompson;Donald F Newgreen.
Cancer Research (2005)

688 Citations

Association of increased basement membrane invasiveness with absence of estrogen receptor and expression of vimentin in human breast cancer cell lines.

Erik W. Thompson;Soonmyoung Paik;Nils Brünner;Connie L. Sommers.
Journal of Cellular Physiology (1992)

631 Citations

MATRIX METALLOPROTEINASE-13 DEFICIENT MICE ARE RESISTANT TO OSTEOARTHRITIC CARTILAGE EROSION BUT NOT CHONDROCYTE HYPERTROPHY OR OSTEOPHYTE DEVELOPMENT

C. B. Little;A. Barai;D. Burkhardt;S. M. Smith.
Arthritis & Rheumatism (2009)

533 Citations

Effects of inhibitors of plasminogen activator, serine proteinases, and collagenase IV on the invasion of basement membranes by metastatic cells.

Reuven Reich;Erik W. Thompson;Yukihide Iwamoto;George R. Martin.
Cancer Research (1988)

527 Citations

Transmembrane/cytoplasmic domain-mediated membrane type 1-matrix metalloprotease docking to invadopodia is required for cell invasion

Hirokazu Nakahara;Linda Howard;Erik W. Thompson;Hiroshi Sato.
Proceedings of the National Academy of Sciences of the United States of America (1997)

515 Citations

Mesenchymal-to-Epithelial Transition Facilitates Bladder Cancer Metastasis: Role of Fibroblast Growth Factor Receptor-2

Christine L. Chaffer;Janelle P. Brennan;Janelle P. Brennan;John L. Slavin;Tony Blick.
Cancer Research (2006)

500 Citations

Vimentin and epithelial-mesenchymal transition in human breast cancer--observations in vitro and in vivo

Maria I. Kokkinos;Razan Wafai;Meng Kang Wong;Donald F. Newgreen.
Cells Tissues Organs (2007)

455 Citations

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