D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 53 Citations 11,166 175 World Ranking 3028 National Ranking 202

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Mutation
  • DNA

Immunology, Molecular biology, Severe combined immunodeficiency, DNA repair and Cell biology are his primary areas of study. His study in Immunology is interdisciplinary in nature, drawing from both Mutation, Hemophagocytic lymphohistiocytosis, Hematopoietic stem cell transplantation and Anemia. His Molecular biology research incorporates elements of LIG4 syndrome, DNA-PKcs, Ku80, Endonuclease and LIG4.

His Severe combined immunodeficiency research is multidisciplinary, incorporating perspectives in DCLRE1C, Recombination-activating gene, Omenn syndrome, CD8 and Graft-versus-host disease. His studies in DCLRE1C integrate themes in fields like Non-homologous end joining and Gene rearrangement. His Cell biology study integrates concerns from other disciplines, such as Immune system, Chemotaxis and DNA, DNA polymerase.

His most cited work include:

  • Hairpin Opening and Overhang Processing by an Artemis/DNA-Dependent Protein Kinase Complex in Nonhomologous End Joining and V(D)J Recombination (933 citations)
  • Mechanism and regulation of human non-homologous DNA end-joining (838 citations)
  • Regulated Expression of Nuclear Receptor RORγt Confers Distinct Functional Fates to NK Cell Receptor-Expressing RORγt+ Innate Lymphocytes (483 citations)

What are the main themes of his work throughout his whole career to date?

Klaus Schwarz mainly focuses on Immunology, Molecular biology, Immunodeficiency, Genetics and Severe combined immunodeficiency. His Immunology research integrates issues from Mutation, Hematopoietic stem cell transplantation, Transplantation and Autoimmune lymphoproliferative syndrome. His Molecular biology study incorporates themes from DNA repair protein XRCC4, Non-homologous end joining, VJ recombination, DNA and Antigen.

As part of the same scientific family, Klaus Schwarz usually focuses on Immunodeficiency, concentrating on Pathology and intersecting with Antibody. The concepts of his Severe combined immunodeficiency study are interwoven with issues in Gene rearrangement, Virology, T cell, Omenn syndrome and Disease. His Omenn syndrome study combines topics in areas such as DCLRE1C and RAG2.

He most often published in these fields:

  • Immunology (60.22%)
  • Molecular biology (22.58%)
  • Immunodeficiency (23.30%)

What were the highlights of his more recent work (between 2018-2021)?

  • Immunology (60.22%)
  • Immune system (10.39%)
  • Cell biology (18.28%)

In recent papers he was focusing on the following fields of study:

Klaus Schwarz mainly investigates Immunology, Immune system, Cell biology, Hematopoietic stem cell transplantation and Immunodeficiency. His work deals with themes such as Treatment outcome and Graft survival, which intersect with Immunology. His study on Immune system also encompasses disciplines like

  • Enteropathy, which have a strong connection to Immunity and Cytotoxic T cell,
  • Cytokine which intersects with area such as Autoimmune lymphoproliferative syndrome, CD8, T cell differentiation and CD38.

His studies examine the connections between Cell biology and genetics, as well as such issues in Receptor-mediated endocytosis, with regards to Function, Antigen and Clathrin. His study focuses on the intersection of Hematopoietic stem cell transplantation and fields such as Severe combined immunodeficiency with connections in the field of Somatic cell, Progenitor cell, Reversion and Robustness. His biological study spans a wide range of topics, including Missense mutation, Sepsis and Splice site mutation.

Between 2018 and 2021, his most popular works were:

  • Human RIPK1 deficiency causes combined immunodeficiency and inflammatory bowel diseases (49 citations)
  • Human RIPK1 deficiency causes combined immunodeficiency and inflammatory bowel diseases (49 citations)
  • A combined immunodeficiency with severe infections, inflammation, and allergy caused by ARPC1B deficiency. (30 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Mutation
  • DNA

Klaus Schwarz focuses on Immunology, Inflammation, Immunodeficiency, Immune system and Antibody. Klaus Schwarz regularly links together related areas like Mutation in his Immunology studies. His Immunodeficiency research includes elements of Inflammasome, Primary immunodeficiency, Programmed cell death and Pathogenesis.

The Immune system study combines topics in areas such as HLA-DR and Enteropathy. His work in Antibody tackles topics such as Subcutaneous Panniculitis-Like T-Cell Lymphoma which are related to areas like Hematopoietic stem cell transplantation. His Hematopoietic stem cell transplantation research incorporates elements of Lymphoma, Prevalence, Age of onset, Adipose tissue and Pediatrics.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Hairpin Opening and Overhang Processing by an Artemis/DNA-Dependent Protein Kinase Complex in Nonhomologous End Joining and V(D)J Recombination

Yunmei Ma;Ulrich Pannicke;Klaus Schwarz;Michael R. Lieber.
Cell (2002)

1547 Citations

Mechanism and regulation of human non-homologous DNA end-joining

Michael R. Lieber;Yunmei Ma;Ulrich Pannicke;Klaus Schwarz.
Nature Reviews Molecular Cell Biology (2003)

1349 Citations

RAG Mutations in Human B Cell-Negative SCID

Klaus Schwarz;George H. Gauss;Leopold Ludwig;Ulrich Pannicke.
Science (1996)

701 Citations

Regulated Expression of Nuclear Receptor RORγt Confers Distinct Functional Fates to NK Cell Receptor-Expressing RORγt+ Innate Lymphocytes

Cedric Vonarbourg;Arthur Mortha;Viet L. Bui;Viet L. Bui;Pedro P. Hernandez;Pedro P. Hernandez.
Immunity (2010)

658 Citations

Partial V(D)J Recombination Activity Leads to Omenn Syndrome

Anna Villa;Sandro Santagata;Fabio Bozzi;Silvia Giliani.
Cell (1998)

475 Citations

A Biochemically Defined System for Mammalian Nonhomologous DNA End Joining

Yunmei Ma;Haihui Lu;Brigette Tippin;Myron F. Goodman.
Molecular Cell (2004)

429 Citations

DNA Ligase IV Is Essential for V(D)J Recombination and DNA Double-Strand Break Repair in Human Precursor Lymphocytes

Ulf Grawunder;David Zimmer;Sebastian Fugmann;Klaus Schwarz.
Molecular Cell (1998)

415 Citations

V(D)J recombination defects in lymphocytes due to RAG mutations: severe immunodeficiency with a spectrum of clinical presentations.

Anna Villa;Cristina Sobacchi;Luigi D. Notarangelo;Fabio Bozzi.
Blood (2001)

377 Citations

A bound form of silicon in glycosaminoglycans and polyuronides.

Klaus Schwarz.
Proceedings of the National Academy of Sciences of the United States of America (1973)

375 Citations

Autoimmunity in Wiskott-Aldrich syndrome: risk factors, clinical features, and outcome in a single-center cohort of 55 patients.

Sophie Dupuis-Girod;Jacques Medioni;Elie Haddad;Pierre Quartier.
Pediatrics (2003)

372 Citations

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