D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 94 Citations 30,686 306 World Ranking 137 National Ranking 5
Medicine D-index 76 Citations 20,317 190 World Ranking 11314 National Ranking 410

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Bacteria

His primary areas of investigation include Microbiology, Streptococcus pneumoniae, Virulence, Pneumolysin and Pneumococcal infections. His Microbiology research integrates issues from Gene, Escherichia coli, Virology and Immunology. His work is dedicated to discovering how Streptococcus pneumoniae, Serotype are connected with Genotype and other disciplines.

His research integrates issues of Mutation, Autolysin, Mutant, Mutagenesis and Molecular biology in his study of Virulence. His Pneumolysin research is multidisciplinary, incorporating perspectives in Virulence factor, Complement system, Antigen, TLR4 and Cytolysin. While the research belongs to areas of Pneumococcal infections, James C. Paton spends his time largely on the problem of Toxoid, intersecting his research to questions surrounding Adjuvant.

His most cited work include:

  • Pathogenesis and Diagnosis of Shiga Toxin-Producing Escherichia coli Infections (1255 citations)
  • Detection and characterization of Shiga toxigenic Escherichia coli by using multiplex PCR assays for stx1, stx2, eaeA, enterohemorrhagic E. coli hlyA, rfbO111, and rfbO157 (1029 citations)
  • The role of Streptococcus pneumoniae virulence factors in host respiratory colonization and disease. (840 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Microbiology, Streptococcus pneumoniae, Virulence, Pneumolysin and Escherichia coli. His Microbiology research is mostly focused on the topic Toxin. His Streptococcus pneumoniae research incorporates themes from Serotype, Immunology and Antigen.

His biological study spans a wide range of topics, including Pathogen, Pathogenesis, Mutation, Mutagenesis and Autolysin. James C. Paton has included themes like Virulence factor, Streptococcaceae and Toxoid in his Pneumolysin study. In his research on the topic of Escherichia coli, Nucleic acid sequence and Unfolded protein response is strongly related with Molecular biology.

He most often published in these fields:

  • Microbiology (57.33%)
  • Streptococcus pneumoniae (39.01%)
  • Virulence (23.92%)

What were the highlights of his more recent work (between 2013-2021)?

  • Microbiology (57.33%)
  • Streptococcus pneumoniae (39.01%)
  • Cell biology (15.30%)

In recent papers he was focusing on the following fields of study:

James C. Paton mostly deals with Microbiology, Streptococcus pneumoniae, Cell biology, Endoplasmic reticulum and Unfolded protein response. His Microbiology research includes themes of Shiga toxin, Escherichia coli and Mutant. His Streptococcus pneumoniae research is multidisciplinary, incorporating elements of Serotype, Virology and Gene, Virulence.

His Virulence study is concerned with Biochemistry in general. As a member of one scientific family, James C. Paton mostly works in the field of Cell biology, focusing on Cellular differentiation and, on occasion, Stem cell. His studies deal with areas such as Protein folding, Chaperone, Intracellular and Signal peptide as well as Endoplasmic reticulum.

Between 2013 and 2021, his most popular works were:

  • Opposing unfolded-protein-response signals converge on death receptor 5 to control apoptosis. (327 citations)
  • Streptococcus pneumoniae : transmission, colonization and invasion (207 citations)
  • A random six-phase switch regulates pneumococcal virulence via global epigenetic changes (141 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Bacteria

His main research concerns Microbiology, Streptococcus pneumoniae, Cell biology, Biochemistry and Endoplasmic reticulum. His study in Pneumolysin and Antibiotic resistance is carried out as part of his Microbiology studies. His research in Streptococcus pneumoniae intersects with topics in Pathogen, Immune system and Virulence.

His Virulence research is multidisciplinary, relying on both Molecular genetics and Mutant. James C. Paton combines subjects such as XBP1, Receptor, Messenger RNA and Protein kinase domain with his study of Cell biology. The various areas that James C. Paton examines in his Endoplasmic reticulum study include Heat shock protein, Chaperone, Pancreas, Proinsulin and Cellular compartment.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Pathogenesis and Diagnosis of Shiga Toxin-Producing Escherichia coli Infections

James C. Paton;Adrienne W. Paton.
Clinical Microbiology Reviews (1998)

1853 Citations

Detection and characterization of Shiga toxigenic Escherichia coli by using multiplex PCR assays for stx1, stx2, eaeA, enterohemorrhagic E. coli hlyA, rfbO111, and rfbO157

Adrienne W. Paton;James C. Paton.
Journal of Clinical Microbiology (1998)

1529 Citations

The role of Streptococcus pneumoniae virulence factors in host respiratory colonization and disease.

Aras Kadioglu;Jeffrey N. Weiser;James C. Paton;Peter W. Andrew.
Nature Reviews Microbiology (2008)

1228 Citations

Recognition of pneumolysin by Toll-like receptor 4 confers resistance to pneumococcal infection.

Richard Malley;Philipp Henneke;Sarah C. Morse;Michael J. Cieslewicz.
Proceedings of the National Academy of Sciences of the United States of America (2003)

809 Citations

Antibody Response to Pneumococcal Vaccination in Children Younger than Five Years of Age

R. M. Douglas;J. C. Paton;S. J. Duncan;D. J. Hansman.
The Journal of Infectious Diseases (1983)

517 Citations

The classical pathway is the dominant complement pathway required for innate immunity to Streptococcus pneumoniae infection in mice

Jeremy S. Brown;Tracy Hussell;Sarah M. Gilliland;David W. Holden.
Proceedings of the National Academy of Sciences of the United States of America (2002)

439 Citations

Opposing unfolded-protein-response signals converge on death receptor 5 to control apoptosis.

Min Lu;David A. Lawrence;Scot Marsters;Diego Acosta-Alvear.
Science (2014)

417 Citations

Reduced virulence of a defined pneumolysin-negative mutant of Streptococcus pneumoniae.

A M Berry;J Yother;D E Briles;D Hansman.
Infection and Immunity (1989)

410 Citations

Characterization of Saa, a Novel Autoagglutinating Adhesin Produced by Locus of Enterocyte Effacement-Negative Shiga-Toxigenic Escherichia coli Strains That Are Virulent for Humans

Adrienne W. Paton;Potjanee Srimanote;Matthew C. Woodrow;James C. Paton.
Infection and Immunity (2001)

404 Citations

Molecular microbiological investigation of an outbreak of hemolytic-uremic syndrome caused by dry fermented sausage contaminated with Shiga-like toxin-producing Escherichia coli.

A W Paton;R M Ratcliff;R M Doyle;J Seymour-Murray.
Journal of Clinical Microbiology (1996)

389 Citations

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