What is he best known for?
The fields of study he is best known for:
Kimitsuna Watanabe spends much of his time researching Transfer RNA, Biochemistry, Genetics, RNA and Gene.
His study in Transfer RNA is interdisciplinary in nature, drawing from both Mutation, Nucleic acid sequence and Mitochondrion.
His work carried out in the field of Mutation brings together such families of science as Molecular biology, GTPBP3, Uridine and TRNA Methyltransferases.
His studies in Mitochondrion integrate themes in fields like Consensus sequence, Post-transcriptional modification, Mitochondrial disease and Nucleotidyltransferase.
His study in Base pair, Nucleotide, Serine, Serine—tRNA ligase and Amino acid is carried out as part of his studies in Biochemistry.
His Mitochondrial DNA, Genome and D arm study in the realm of Gene connects with subjects such as Ascaris suum.
His most cited work include:
- Recent evidence for evolution of the genetic code. (465 citations)
- Taurine as a constituent of mitochondrial tRNAs: new insights into the functions of taurine and human mitochondrial diseases (269 citations)
- Codon-specific translational defect caused by a wobble modification deficiency in mutant tRNA from a human mitochondrial disease. (207 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Transfer RNA, Biochemistry, RNA, Genetics and Mitochondrion.
To a larger extent, Kimitsuna Watanabe studies Gene with the aim of understanding Transfer RNA.
Kimitsuna Watanabe combines subjects such as Crystallography, Guanosine, Nucleotide and Sequence alignment with his study of RNA.
His study brings together the fields of Serine and Genetics.
His research investigates the connection between Mitochondrion and topics such as Molecular biology that intersect with issues in D arm.
His research investigates the connection between Mutant and topics such as Mitochondrial disease that intersect with problems in Point mutation.
He most often published in these fields:
- Transfer RNA (64.14%)
- Biochemistry (62.15%)
- RNA (28.29%)
What were the highlights of his more recent work (between 2003-2017)?
- Transfer RNA (64.14%)
- Biochemistry (62.15%)
- Genetics (30.68%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Transfer RNA, Biochemistry, Genetics, Mitochondrion and Genetic code.
The Transfer RNA study combines topics in areas such as Translation, Mutant and Mutation.
Kimitsuna Watanabe has included themes like Molecular biology and Mitochondrial disease in his Mutation study.
His Genome, Mitochondrial translation, Mitochondrial DNA and TRNA modification study in the realm of Genetics interacts with subjects such as Ascaris suum.
His Mitochondrion research focuses on Uridine and how it connects with Nucleic acid, Halocynthia roretzi and Taurine.
Kimitsuna Watanabe has researched Genetic code in several fields, including Evolutionary biology and Statistical physics.
Between 2003 and 2017, his most popular works were:
- Codon-specific translational defect caused by a wobble modification deficiency in mutant tRNA from a human mitochondrial disease. (207 citations)
- Codon-specific translational defect caused by a wobble modification deficiency in mutant tRNA from a human mitochondrial disease. (207 citations)
- Mutation in TRMU related to transfer RNA modification modulates the phenotypic expression of the deafness-associated mitochondrial 12S ribosomal RNA mutations (185 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in Transfer RNA, Biochemistry, Genetics, Wobble base pair and Mitochondrion.
His study with Transfer RNA involves better knowledge in RNA.
His study in Translation and Genetic code falls under the purview of Genetics.
His Translation study deals with Ribosome intersecting with Mitochondrial translation and Protein biosynthesis.
His Mitochondrion study incorporates themes from Pyridoxal, Cysteine desulfurase, Biogenesis, Cytoplasm and Saccharomyces cerevisiae.
His study explores the link between Mutant and topics such as Mitochondrial disease that cross with problems in Post-transcriptional modification and Mitochondrial myopathy.
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