Harry LeVine

Overview

Best Publications

• Thioflavine T interaction with synthetic Alzheimer's disease beta-amyloid peptides: detection of amyloid aggregation in solution.

  Harry Levine

  3255
  Citations

• QUANTIFICATION OF BETA -SHEET AMYLOID FIBRIL STRUCTURES WITH THIOFLAVIN T

  Harry LeVine

  1990
  Citations

• Alzheimer's Disease and the Amyloid-β Peptide

  M. Paul Murphy;Harry LeVine Iii

  1788
  Citations

• A model for intracellular translocation of protein kinase C involving synergism between Ca2+ and phorbol esters.

  Marlene Wolf;Harry LeVine;W. Stratford May;Pedro Cuatrecasas

  618
  Citations

• Cognitive decline in Alzheimer's disease is associated with selective changes in calcineurin/NFAT signaling.

  Hafiz Mohmmad Abdul;Michelle A. Sama;Jennifer L. Furman;Diana M. Mathis

  329
  Citations

• Thioflavine T interaction with amyloid β-sheet structures

  Harry LeVine

  324
  Citations

• Protein kinase C phosphorylates topoisomerase II: topoisomerase activation and its possible role in phorbol ester-induced differentiation of HL-60 cells.

  Naji Sahyoun;Marlene Wolf;Jeffrey Besterman;Tao-Shih Hsieh

  300
  Citations

• Alzheimer's Therapeutics Targeting Amyloid Beta 1–42 Oligomers II: Sigma-2/PGRMC1 Receptors Mediate Abeta 42 Oligomer Binding and Synaptotoxicity

  Nicholas J. Izzo;Jinbin Xu;Chenbo Zeng;Molly J. Kirk;Molly J. Kirk

  282
  Citations

• Amiloride inhibits phorbol ester-stimulated Na+/H+ exchange and protein kinase C. An amiloride analog selectively inhibits Na+/H+ exchange.

  J M Besterman;W S May;H LeVine rd;E J Cragoe

  281
  Citations

• Alzheimer's Therapeutics Targeting Amyloid Beta 1–42 Oligomers I: Abeta 42 Oligomer Binding to Specific Neuronal Receptors Is Displaced by Drug Candidates That Improve Cognitive Deficits

  Nicholas J. Izzo;Agnes Staniszewski;Lillian To;Mauro Fa

  249
  Citations

• Phosphorylation and Activation of β-Adrenergic Receptor Kinase by Protein Kinase C

  Tsu Tshen Chuang;Harry LeVine;Antonio De Blasi

  229
  Citations

• Alzheimer’s β-peptide oligomer formation at physiologic concentrations

  Harry LeVine

  197
  Citations

• The role of microglial cells and astrocytes in fibrillar plaque evolution in transgenic APPSW mice

  Jerzy Wegiel;Kuo-Chiang Wang;Humi Imaki;Richard Rubenstein

  180
  Citations

• Stopped-flow kinetics reveal multiple phases of thioflavin T binding to Alzheimer beta (1-40) amyloid fibrils

  Harry Levine

  162
  Citations

• Exogenous seeding of cerebral β‐amyloid deposition in βAPP‐transgenic rats

  Rebecca F. Rosen;Jason J. Fritz;Jeromy Dooyema;Amarallys F. Cintron

  162
  Citations

• Pathological Tau Promotes Neuronal Damage by Impairing Ribosomal Function and Decreasing Protein Synthesis

  Shelby Meier;Michelle Bell;Danielle N. Lyons;Jennifer Rodriguez-Rivera

  160
  Citations

• Soluble multimeric Alzheimer β(1–40) pre-amyloid complexes in dilute solution

  Harry Levine

  148
  Citations

• Amyloid fibril polymorphism: a challenge for molecular imaging and therapy.

  M. Fändrich;S. Nyström;K. P. R. Nilsson;A. Böckmann

  139
  Citations

• The cerebral proteopathies: neurodegenerative disorders of protein conformation and assembly.

  Lary C. Walker;Harry LeVine

  136
  Citations

• Association between frontal cortex oxidative damage and beta-amyloid as a function of age in Down syndrome

  Giovanna Cenini;Amy L S Dowling;Tina L Beckett;Eugenio Barone;Eugenio Barone

  134
  Citations