What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Cytokine
George Kollias mainly focuses on Immunology, Tumor necrosis factor alpha, Cytokine, Inflammation and Molecular biology.
George Kollias regularly ties together related areas like Cancer research in his Immunology studies.
His research in Tumor necrosis factor alpha intersects with topics in Stromal cell, Inflammatory bowel disease, Receptor, Cell biology and Multiple sclerosis.
His Cytokine study integrates concerns from other disciplines, such as Astrocyte and In vivo.
The various areas that George Kollias examines in his Inflammation study include Delayed hypersensitivity, Interferon and Antibody.
His studies in Molecular biology integrate themes in fields like Lipopolysaccharide, Transfection, Messenger RNA, Regulation of gene expression and p38 mitogen-activated protein kinases.
His most cited work include:
- The Transcriptional Landscape of the Mammalian Genome (2876 citations)
- Position-independent, high-level expression of the human β-globin gene in transgenic mice (1556 citations)
- Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis. (1395 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Tumor necrosis factor alpha, Immunology, Arthritis, Inflammation and Cancer research.
His biological study deals with issues like Genetically modified mouse, which deal with fields such as Molecular biology and Gene expression.
The concepts of his Immunology study are interwoven with issues in Transgene and Cell biology.
His biological study spans a wide range of topics, including Autoimmunity and Rheumatoid arthritis.
His research investigates the link between Inflammation and topics such as Osteoclast that cross with problems in Bone resorption.
His Cancer research study incorporates themes from Carcinogenesis, Fibroblast, Necrosis and RANKL.
He most often published in these fields:
- Tumor necrosis factor alpha (51.14%)
- Immunology (41.48%)
- Arthritis (26.99%)
What were the highlights of his more recent work (between 2017-2021)?
- Tumor necrosis factor alpha (51.14%)
- Cancer research (19.89%)
- Arthritis (26.99%)
In recent papers he was focusing on the following fields of study:
Tumor necrosis factor alpha, Cancer research, Arthritis, Inflammation and Mesenchymal stem cell are his primary areas of study.
Tumor necrosis factor alpha is a primary field of his research addressed under Internal medicine.
His studies deal with areas such as Experimental autoimmune encephalomyelitis, Fibroblast, Pathogenesis and RANKL as well as Cancer research.
His Arthritis research includes elements of CXCL1, Autoimmunity, Rheumatoid arthritis and Programmed cell death.
His Inflammation study is related to the wider topic of Immunology.
His Immunology study combines topics from a wide range of disciplines, such as Disease, Genetic predisposition and Degranulation.
Between 2017 and 2021, his most popular works were:
- A20 prevents inflammasome-dependent arthritis by inhibiting macrophage necroptosis through its ZnF7 ubiquitin-binding domain (49 citations)
- Inhibiting Interleukin 36 Receptor Signaling Reduces Fibrosis in Mice With Chronic Intestinal Inflammation (49 citations)
- Paracrine orchestration of intestinal tumorigenesis by a mesenchymal niche. (47 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Cytokine
George Kollias mostly deals with Cancer research, Mesenchymal stem cell, Tumor necrosis factor alpha, Arthritis and Cell biology.
George Kollias combines subjects such as Cancer cell, T cell, Adoptive cell transfer and Acquired immune system with his study of Cancer research.
His Mesenchymal stem cell research is multidisciplinary, incorporating perspectives in Carcinogenesis, Stromal cell and Tumor microenvironment.
His Tumor necrosis factor alpha study necessitates a more in-depth grasp of Internal medicine.
The study incorporates disciplines such as Inflammation and Programmed cell death, RIPK1, Necroptosis in addition to Arthritis.
His studies in Cell biology integrate themes in fields like Deubiquitinating enzyme, Ubiquitin binding and Inflammatory arthritis.
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