What is he best known for?
The fields of study he is best known for:
- Gene
- Cytokine
- Immune system
Marc Feldmann spends much of his time researching Immunology, Tumor necrosis factor alpha, Rheumatoid arthritis, Cytokine and Arthritis.
Immunology is a component of his Pathogenesis, Antigen, Antibody, Immune system and Monoclonal antibody studies.
His studies examine the connections between Tumor necrosis factor alpha and genetics, as well as such issues in Receptor, with regards to Extracellular.
Marc Feldmann usually deals with Rheumatoid arthritis and limits it to topics linked to Autoimmunity and Human leukocyte antigen.
His Cytokine research integrates issues from Proinflammatory cytokine, Inflammation, Synovial membrane and Autoimmune disease.
His studies deal with areas such as Alpha and Ex vivo as well as Arthritis.
His most cited work include:
- Infliximab and methotrexate in the treatment of rheumatoid arthritis. Anti-Tumor Necrosis Factor Trial in Rheumatoid Arthritis with Concomitant Therapy Study Group. (2688 citations)
- ROLE OF CYTOKINES IN RHEUMATOID ARTHRITIS (2251 citations)
- Infliximab (chimeric anti-tumour necrosis factor α monoclonal antibody) versus placebo in rheumatoid arthritis patients receiving concomitant methotrexate: a randomised phase III trial (2178 citations)
What are the main themes of his work throughout his whole career to date?
Marc Feldmann mainly investigates Immunology, Tumor necrosis factor alpha, Antigen, Cytokine and Rheumatoid arthritis.
His Immunology study frequently draws connections to adjacent fields such as Receptor.
His Tumor necrosis factor alpha study integrates concerns from other disciplines, such as Cancer research, Clinical trial, Blockade, Necrosis and Pharmacology.
His biological study spans a wide range of topics, including In vitro, T cell, Antigen-presenting cell, Molecular biology and Cell biology.
His work deals with themes such as Proinflammatory cytokine, Endocrinology and Synovial membrane, which intersect with Cytokine.
Autoimmune disease is closely connected to Autoimmunity in his research, which is encompassed under the umbrella topic of Rheumatoid arthritis.
He most often published in these fields:
- Immunology (57.85%)
- Tumor necrosis factor alpha (25.58%)
- Antigen (25.58%)
What were the highlights of his more recent work (between 2008-2021)?
- Immunology (57.85%)
- Tumor necrosis factor alpha (25.58%)
- Inflammation (10.03%)
In recent papers he was focusing on the following fields of study:
Marc Feldmann mostly deals with Immunology, Tumor necrosis factor alpha, Inflammation, Rheumatoid arthritis and Cytokine.
His Immunology study combines topics in areas such as Receptor and Internal medicine.
His studies in Internal medicine integrate themes in fields like Gastroenterology and Endocrinology.
His work carried out in the field of Tumor necrosis factor alpha brings together such families of science as Cancer research, Necrosis, Postoperative cognitive dysfunction, Neuroinflammation and Fibrosis.
His research on Rheumatoid arthritis also deals with topics like
- Etanercept most often made with reference to Infliximab,
- Fusion protein that connect with fields like Monoclonal antibody and Immunotherapy.
His Cytokine course of study focuses on Histone and Mitochondrial biogenesis.
Between 2008 and 2021, his most popular works were:
- IRF5 Promotes Inflammatory Macrophage Polarization and TH1-TH17 Responses (798 citations)
- Role of interleukin-1beta in postoperative cognitive dysfunction. (464 citations)
- Tumor necrosis factor-alpha triggers a cytokine cascade yielding postoperative cognitive decline. (440 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Immune system
- Cytokine
The scientist’s investigation covers issues in Immunology, Tumor necrosis factor alpha, Inflammation, Cytokine and Arthritis.
The study of Immunology is intertwined with the study of Receptor in a number of ways.
His Tumor necrosis factor alpha research includes themes of NFKB1, Neuroinflammation, Cellular infiltration and Postoperative cognitive dysfunction.
His Inflammation study incorporates themes from Signal transduction, Cell type and Pneumonia.
His study in Cytokine is interdisciplinary in nature, drawing from both Blockade, Antibody and FOXP3.
His Arthritis research incorporates themes from Porphyromonas gingivalis, IRF5 and Transcription factor.
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