H-Index & Metrics Best Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Immunology D-index 59 Citations 9,834 116 World Ranking 1832 National Ranking 154

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cytokine
  • Internal medicine

Arthritis, Immunology, Rheumatoid arthritis, Tumor necrosis factor alpha and Cytokine are his primary areas of study. Richard O. Williams interconnects Inflammation, T cell, Blockade and Pathology in the investigation of issues within Arthritis. His Type II collagen, Synovial membrane, Pathogenesis, Proinflammatory cytokine and Interleukin 17 study are his primary interests in Immunology.

His Type II collagen study combines topics in areas such as C57BL/6 and Autoimmune disease. Within one scientific family, he focuses on topics pertaining to Antibody under Rheumatoid arthritis, and may sometimes address concerns connected to Immunotherapy. His work deals with themes such as Interleukin, Analgesic and Monoclonal antibody, which intersect with Tumor necrosis factor alpha.

His most cited work include:

  • Anti-tumor necrosis factor ameliorates joint disease in murine collagen-induced arthritis. (934 citations)
  • Relationship between Th1/Th2 cytokine patterns and the arthritogenic response in collagen‐induced arthritis (293 citations)
  • Interleukin-10 inhibition of the progression of established collagen-induced arthritis. (243 citations)

What are the main themes of his work throughout his whole career to date?

Richard O. Williams focuses on Immunology, Arthritis, Rheumatoid arthritis, Tumor necrosis factor alpha and Inflammation. His research brings together the fields of Disease and Immunology. His studies in Arthritis integrate themes in fields like Proinflammatory cytokine, Monoclonal antibody, Pharmacology and Pathology.

His work in Rheumatoid arthritis addresses issues such as Psoriasis, which are connected to fields such as Ankylosing spondylitis. His Tumor necrosis factor alpha study integrates concerns from other disciplines, such as Alpha, Monoclonal, Blockade, Necrosis and Interleukin. His Cytokine research integrates issues from Pathogenesis and Synovial membrane.

He most often published in these fields:

  • Immunology (56.04%)
  • Arthritis (44.93%)
  • Rheumatoid arthritis (43.48%)

What were the highlights of his more recent work (between 2015-2021)?

  • Immunology (56.04%)
  • Arthritis (44.93%)
  • Inflammation (19.81%)

In recent papers he was focusing on the following fields of study:

Richard O. Williams mainly investigates Immunology, Arthritis, Inflammation, Rheumatoid arthritis and Immune system. His research combines Genetic association and Immunology. His Arthritis research includes themes of Regulatory T cell, Antibody, Biomarker and Dexamethasone.

His Inflammation research includes elements of Kynurenine pathway, Kynurenine, Cell, Cancer research and Pharmacology. The Rheumatoid arthritis study combines topics in areas such as Autoimmunity, Monoclonal antibody and Downregulation and upregulation. His Immune system research is multidisciplinary, incorporating perspectives in Proinflammatory cytokine, Autoimmune disease and Antigen.

Between 2015 and 2021, his most popular works were:

  • Effects of fluoxetine on functional outcomes after acute stroke (FOCUS): a pragmatic, double-blind, randomised, controlled trial (101 citations)
  • Antiplatelet therapy with aspirin, clopidogrel, and dipyridamole versus clopidogrel alone or aspirin and dipyridamole in patients with acute cerebral ischaemia (TARDIS): a randomised, open-label, phase 3 superiority trial (53 citations)
  • Abrogation of collagen-induced arthritis by a peptidyl arginine deiminase inhibitor is associated with modulation of T cell-mediated immune responses. (41 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Cancer

The scientist’s investigation covers issues in Immunology, T cell, Arthritis, Inflammation and Immune system. Richard O. Williams studies Tumor necrosis factor alpha which is a part of Immunology. His research integrates issues of Receptor and Rheumatoid arthritis in his study of T cell.

His work investigates the relationship between Arthritis and topics such as Biomarker that intersect with problems in Kynurenine pathway, Neurodegeneration, Huntington's disease, Regulatory T cell and Kynurenine. As a part of the same scientific study, Richard O. Williams usually deals with the Inflammation, concentrating on Autoimmune disease and frequently concerns with DNA methylation, Tumor necrosis factor receptor 2, Phenotype, FOXP3 and Proinflammatory cytokine. His Immune system study combines topics in areas such as In vitro, Antibody, Arginine and Protein-Arginine Deiminases.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Anti-tumor necrosis factor ameliorates joint disease in murine collagen-induced arthritis.

Richard O. Williams;Marc Feldmann;Ravinder N. Maini.
Proceedings of the National Academy of Sciences of the United States of America (1992)

1264 Citations

Relationship between Th1/Th2 cytokine patterns and the arthritogenic response in collagen‐induced arthritis

Claudia Mauri;Richard O. Williams;Marita Walmsley;Marc Feldmann.
European Journal of Immunology (1996)

411 Citations

Interleukin-10 inhibition of the progression of established collagen-induced arthritis.

Marita Walmsley;Peter D. Katsikis;Erika Abney;Sarah Parry.
Arthritis & Rheumatism (1996)

322 Citations

Synergy between anti-CD4 and anti-tumor necrosis factor in the amelioration of established collagen-induced arthritis.

Richard O. Williams;Lesley J. Mason;Marc Feldmann;Ravinder N. Maini.
Proceedings of the National Academy of Sciences of the United States of America (1994)

279 Citations

Monoclonal anti-TNF alpha antibody as a probe of pathogenesis and therapy of rheumatoid disease.

Ravinder N. Maini;Michael J. Elliott;Fionula M. Brennan;Richard O. Williams.
Immunological Reviews (1995)

259 Citations

Blockade of tumor necrosis factor in collagen-induced arthritis reveals a novel immunoregulatory pathway for Th1 and Th17 cells

Clare A. Notley;Julia J. Inglis;Saba Alzabin;Fiona E. McCann.
Journal of Experimental Medicine (2008)

254 Citations

RHAMM, a receptor for hyaluronan-mediated motility, compensates for CD44 in inflamed CD44-knockout mice: A different interpretation of redundancy

Shlomo Nedvetzki;Erez Gonen;Nathalie Assayag;Reuven Reich.
Proceedings of the National Academy of Sciences of the United States of America (2004)

231 Citations

Suppression of collagen-induced arthritis by continuous administration of IL-4.

A C Horsfall;D M Butler;L Marinova;P J Warden.
Journal of Immunology (1997)

217 Citations

Amelioration of collagen-induced arthritis and suppression of interferon-gamma, interleukin-12, and tumor necrosis factor alpha production by interferon-beta gene therapy.

Kostas A. Triantaphyllopoulos;Richard O. Williams;Hitakshi Tailor;Yuti Chernajovsky.
Arthritis & Rheumatism (1999)

200 Citations

Collagen-induced arthritis as a model of hyperalgesia: functional and cellular analysis of the analgesic actions of tumor necrosis factor blockade.

Julia J. Inglis;Clare A. Notley;David Essex;Alex W. Wilson.
Arthritis & Rheumatism (2007)

195 Citations

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