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Immunology

D-Index
69
Citations
16242
World Ranking
2513
National Ranking
216

Overview

Andrew P. Cope is affiliated with King's College London in the United Kingdom and has produced substantial research in the field of medicine, with a particular focus on rheumatology. Their scholarly activities concentrate primarily on autoimmune and inflammatory disorders, with notable work in rheumatoid arthritis and systemic lupus erythematosus research.

The main fields of study in which Andrew P. Cope has contributed include:

  • Medicine

The subfields related to this work are:

  • Rheumatology
  • Immunology
  • Hematology
  • Infectious Diseases
  • Pathology and Forensic Medicine

Topics frequently addressed in their research consist of:

  • Rheumatoid Arthritis Research and Therapies
  • Systemic Lupus Erythematosus Research
  • Autoimmune and Inflammatory Disorders Research
  • Lymphoma Diagnosis and Treatment
  • Spondyloarthritis Studies and Treatments
  • T-cell and B-cell Immunology
  • COVID-19 Clinical Research Studies

Among the recent publications by Andrew P. Cope are:

  • Abatacept in individuals at high risk of rheumatoid arthritis (APIPPRA): a randomised, double-blind, multicentre, parallel, placebo-controlled, phase 2b clinical trial, 2024, The Lancet

Andrew P. Cope has also coauthored works in collaboration with the following researchers:

  • James Galloway
  • Sam Norton
  • Maryam Adas
  • Katie Bechman
  • Mark Russell

The venues commonly publishing Andrew P. Cope's research include:

  • Annals of the Rheumatic Diseases
  • Lara D. Veeken
  • Arthritis & Rheumatology
  • The Lancet Rheumatology
  • bioRxiv (Cold Spring Harbor Laboratory)

Best Publications

  • Intracellular Complement Activation Sustains T Cell Homeostasis and Mediates Effector Differentiation

    M. Kathryn Liszewski;Martin Kolev;Gaelle Le Friec;Marilyn Leung

  • EULAR recommendations for terminology and research in individuals at risk of rheumatoid arthritis: report from the Study Group for Risk Factors for Rheumatoid Arthritis

    Danielle M. Gerlag;Karim Raza;Lisa G M van Baarsen;Elisabeth Brouwer

  • T helper 1 immunity requires complement-driven NLRP3 inflammasome activity in CD4+ T cells

    Giuseppina Arbore;Erin E. West;Rosanne Spolski;Avril A. B. Robertson

  • Associations between immune-suppressive and stimulating drugs and novel COVID-19 - A systematic review of current evidence

    Beth Russell;Charlotte Moss;Gincy George;Aida Santaolalla

  • Treg cells suppress osteoclast formation: a new link between the immune system and bone.

    Mario M. Zaiss;Roland Axmann;Jochen Zwerina;Karin Polzer

  • Increased levels of soluble tumor necrosis factor receptors in the sera and synovial fluid of patients with rheumatic diseases.

    A P Cope;D Aderka;M Doherty;H Engelmann

  • The central role of T cells in rheumatoid arthritis.

    Andrew P. Cope;Hendrik Schulze-Koops;Martin Aringer

  • Chronic Tumor Necrosis Factor Alters T Cell Responses by Attenuating T Cell Receptor Signaling

    Andrew P. Cope;Roland S. Liblau;Xiao-Dong Yang;Mauro Congia

  • Chronic exposure to tumor necrosis factor (TNF) in vitro impairs the activation of T cells through the T cell receptor/CD3 complex; reversal in vivo by anti-TNF antibodies in patients with rheumatoid arthritis.

    A. P. Cope;M. Londei;N. R. Chu;S. B. A. Cohen

  • The antiproliferative effect of mesenchymal stem cells is a fundamental property shared by all stromal cells.

    Simon Jones;Nicole Horwood;Andrew Cope;Francesco Dazzi

  • Complement regulator CD46 temporally regulates cytokine production by conventional and unconventional T cells.

    John Cardone;Gaelle Le Friec;Pierre Vantourout;Pierre Vantourout;Andrew Roberts;Andrew Roberts

  • The Th1 life cycle: molecular control of IFN-γ to IL-10 switching

    Andrew Cope;Gaelle Le Friec;John Cardone;Claudia Kemper

  • Why is PTPN22 a good candidate susceptibility gene for autoimmune disease

    Garth L. Burn;Lena Svensson;Cristina Sanchez-Blanco;Manoj Saini

  • A systematic review and meta-analysis of infection risk with small molecule JAK inhibitors in rheumatoid arthritis

    Katie Bechman;Sujith Subesinghe;Sam Norton;Fabiola Atzeni

  • The role of tumour necrosis factor (TNF-alpha) in experimental autoimmune uveoretinitis (EAU).

    A. D. Dick;John Vincent Forrester;Janet Mary Liversidge;A. P. Cope

  • Monocytes Induce STAT3 Activation in Human Mesenchymal Stem Cells to Promote Osteoblast Formation

    Vicky Nicolaidou;Mei Mei Wong;Andia N. Redpath;Adel Ersek

  • EULAR points to consider for the diagnosis and management of rheumatic immune-related adverse events due to cancer immunotherapy with checkpoint inhibitors.

    Marie Kostine;Axel Finckh;Clifton O. Bingham;Karen Visser

  • Innate-like T cells straddle innate and adaptive immunity by altering antigen-receptor responsiveness

    Melanie Wencker;Gleb Turchinovich;Rafael Di Marco Barros;Livija Deban

  • T cells in rheumatoid arthritis

    Andrew P Cope

  • Immunomodulatory properties of mesenchymal stem cells: a review based on an interdisciplinary meeting held at the Kennedy Institute of Rheumatology Division, London, UK, 31 October 2005

    Alan Tyndall;Ulrich A Walker;Andrew Cope;Francesco Dazzi

  • Correlation between serum levels of soluble tumor necrosis factor receptor and disease activity in systemic lupus erythematosus

    D Aderka;A Wysenbeek;H Engelmann;A P Cope

Frequent Co-Authors

David Scott
David Scott Ascension Health
Claudia Kemper
Claudia Kemper National Institutes of Health
Sam Norton
Sam Norton King's College London
Cathryn M. Lewis
Cathryn M. Lewis King's College London
Fionula M. Brennan
Fionula M. Brennan University of Oxford
Rose Zamoyska
Rose Zamoyska University of Edinburgh
Francesco Dazzi
Francesco Dazzi King's College London
Paul Emery
Paul Emery University of Leeds
Kimme L. Hyrich
Kimme L. Hyrich University of Manchester
Adrian Hayday
Adrian Hayday King's College London

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