2008 - Potamkin Prize for Research in Pick's, Alzheimer's, and Related Diseases, American Academy of Neurology
2008 - Sedgwick Memorial Medal, American Public Health Association
2004 - Metlife Foundation Award for Medical Research in Alzheimer's Disease
The scientist’s investigation covers issues in Alzheimer's disease, Pathology, Pittsburgh compound B, Amyloid and Dementia. His work deals with themes such as Apolipoprotein E, Alzheimer's disease biomarkers and Central nervous system disease, which intersect with Alzheimer's disease. The concepts of his Pathology study are interwoven with issues in Precuneus and Pediatrics.
His studies deal with areas such as Positron emission tomography, Nuclear medicine, Magnetic resonance imaging, Cognitive disorder and Brain mapping as well as Pittsburgh compound B. His Amyloid research is multidisciplinary, incorporating elements of In vitro, Biochemistry, Presenilin, Genetically modified mouse and In vivo. His Dementia research incorporates themes from Psychiatry, Clinical trial and Neuroscience.
William E. Klunk mostly deals with Pathology, Amyloid, Alzheimer's disease, Pittsburgh compound B and Internal medicine. The various areas that he examines in his Pathology study include Preclinical imaging and In vivo. His Amyloid study integrates concerns from other disciplines, such as Biopsy, Biochemistry, Amyloid beta and Amyloidosis.
His Alzheimer's disease study deals with Dementia intersecting with Biomarker. His research investigates the link between Pittsburgh compound B and topics such as Positron emission tomography that cross with problems in Magnetic resonance imaging. William E. Klunk combines subjects such as Endocrinology, Oncology, Cardiology, Cognition and Down syndrome with his study of Internal medicine.
William E. Klunk spends much of his time researching Down syndrome, Disease, Internal medicine, Amyloid and Pittsburgh compound B. His Down syndrome research is multidisciplinary, incorporating perspectives in White matter, Neurodegeneration, Neuroimaging, Dementia and Episodic memory. In Dementia, William E. Klunk works on issues like Neuropsychology, which are connected to Clinical Dementia Rating.
His Internal medicine study incorporates themes from Endocrinology and Oncology. William E. Klunk studied Amyloid and Neuroscience that intersect with Alzheimer's disease. His Pittsburgh compound B research focuses on subjects like Positron emission tomography, which are linked to Nuclear magnetic resonance.
His main research concerns Down syndrome, Disease, Pittsburgh compound B, Amyloid and Internal medicine. His Disease research is within the category of Pathology. His Pittsburgh compound B research includes elements of Positron emission tomography, Precuneus, Multivariate statistics and Genome-wide association study.
William E. Klunk interconnects Apolipoprotein E and Dementia in the investigation of issues within Amyloid. As a part of the same scientific family, William E. Klunk mostly works in the field of Internal medicine, focusing on Endocrinology and, on occasion, Pet imaging, Standardized uptake value, Repeated measures design and Genotype. The study incorporates disciplines such as Alzheimer's disease and Atrophy in addition to Neuropsychology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The diagnosis of dementia due to Alzheimer’s disease: Recommendations from the National Institute on Aging-Alzheimer’s Association workgroups on diagnostic guidelines for Alzheimer's disease
Guy M. McKhann;Guy M. McKhann;David S. Knopman;Howard Chertkow;Bradley T. Hyman.
Alzheimers & Dementia (2011)
Imaging brain amyloid in Alzheimer's disease with Pittsburgh Compound-B.
William E. Klunk;Henry Engler;Agneta Nordberg;Yanming Wang.
Annals of Neurology (2004)
Clinical and Biomarker Changes in Dominantly Inherited Alzheimer’s Disease
Randall J. Bateman;Chengjie Xiong;Tammie L.S. Benzinger;Anne M. Fagan.
The New England Journal of Medicine (2012)
Molecular, Structural, and Functional Characterization of Alzheimer's Disease: Evidence for a Relationship between Default Activity, Amyloid, and Memory
Randy L. Buckner;Abraham Z. Snyder;Benjamin J. Shannon;Gina LaRossa.
The Journal of Neuroscience (2005)
Two Phase 3 Trials of Bapineuzumab in Mild-to-Moderate Alzheimer's Disease
Stephen Salloway;Reisa Sperling;Nick C. Fox;Kaj Blennow.
The New England Journal of Medicine (2014)
Inverse relation between in vivo amyloid imaging load and cerebrospinal fluid Abeta42 in humans.
Anne M. Fagan;Mark A. Mintun;Robert H. Mach;Sang-Yoon Lee.
Annals of Neurology (2006)
Imaging beta-amyloid burden in aging and dementia.
Christopher C Rowe;Steven Ng;Uwe Ackermann;Sylvia Gong.
Neurology (2007)
[11C]PIB in a nondemented population: potential antecedent marker of Alzheimer disease.
M. A. Mintun;G. N. LaRossa;Y. I. Sheline;C. S. Dence.
Neurology (2006)
Frequent Amyloid Deposition Without Significant Cognitive Impairment Among the Elderly
Howard Jay Aizenstein;Robert D. Nebes;Judith A. Saxton;Julie C. Price.
JAMA Neurology (2008)
SYNTHESIS AND EVALUATION OF 11C-LABELED 6-SUBSTITUTED 2-ARYLBENZOTHIAZOLES AS AMYLOID IMAGING AGENTS
Chester A. Mathis;Yanming Wang;Daniel P. Holt;Guo Feng Huang.
Journal of Medicinal Chemistry (2003)
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