His primary areas of study are Neuroscience, Cognition, Dementia, Psychiatry and Alzheimer's disease. As a member of one scientific family, Howard J. Aizenstein mostly works in the field of Neuroscience, focusing on Hyperintensity and, on occasion, Physical medicine and rehabilitation, Depression and Basal ganglia. His work in Cognition addresses issues such as Atrophy, which are connected to fields such as Brain damage and Stage.
His Dementia study incorporates themes from Cross-sectional study, Pediatrics, Lateral ventricles and Cardiology. In the field of Psychiatry, his study on Late life depression, Comorbidity and Psychomotor agitation overlaps with subjects such as Atypical antipsychotic. Howard J. Aizenstein interconnects Cognitive disorder, Neuropsychology and Cognitive decline in the investigation of issues within Alzheimer's disease.
Howard J. Aizenstein mostly deals with Internal medicine, Neuroscience, Neuroimaging, Dementia and Hyperintensity. Howard J. Aizenstein works mostly in the field of Internal medicine, limiting it down to topics relating to Depression and, in certain cases, Clinical psychology, as a part of the same area of interest. His study on Neuroscience is mostly dedicated to connecting different topics, such as Audiology.
Howard J. Aizenstein combines subjects such as Magnetic resonance imaging, Disease, Physical medicine and rehabilitation and Artificial intelligence with his study of Neuroimaging. His work deals with themes such as Alzheimer's disease and Pediatrics, which intersect with Dementia. His study on Hyperintensity also encompasses disciplines like
Internal medicine, Neuroimaging, Hyperintensity, Dementia and Depression are his primary areas of study. His work in Internal medicine covers topics such as Endocrinology which are related to areas like Cohort. His Neuroimaging course of study focuses on Physical medicine and rehabilitation and Fractional anisotropy.
The study incorporates disciplines such as White matter, Cognitive impairment and Cardiology in addition to Hyperintensity. His research integrates issues of Cardiorespiratory fitness and Physical therapy in his study of Depression. In his work, Artificial neural network is strongly intertwined with Cognition, which is a subfield of Late life depression.
His scientific interests lie mostly in Internal medicine, Neuroimaging, Clinical psychology, Depression and Late life depression. Howard J. Aizenstein regularly links together related areas like Endocrinology in his Internal medicine studies. His Neuroimaging study combines topics in areas such as Pathological, Disease and Age prediction.
His Depression study integrates concerns from other disciplines, such as Morning, Insomnia and Default mode network. In his research on the topic of Late life depression, Major depressive disorder is strongly related with Cognition. In his research, Hyperintensity is intimately related to Actigraphy, which falls under the overarching field of Dementia.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Frequent Amyloid Deposition Without Significant Cognitive Impairment Among the Elderly
Howard Jay Aizenstein;Robert D. Nebes;Judith A. Saxton;Julie C. Price.
JAMA Neurology (2008)
Fibrillar amyloid-β burden in cognitively normal people at 3 levels of genetic risk for Alzheimer's disease
Eric M. Reiman;Kewei Chen;Xiaofen Liu;Daniel Bandy.
Proceedings of the National Academy of Sciences of the United States of America (2009)
The vascular depression hypothesis: mechanisms linking vascular disease with depression.
Warren D. Taylor;Howard J. Aizenstein;George S. Alexopoulos.
Molecular Psychiatry (2013)
Pathways linking late-life depression to persistent cognitive impairment and dementia
Meryl A Butters;Jeffrey B. Young;Oscar Ramos Lopez;Howard J. Aizenstein.
Dialogues in clinical neuroscience (2008)
Thinning of the cerebral cortex visualized in HIV/AIDS reflects CD4+ T lymphocyte decline
Paul M. Thompson;Rebecca A. Dutton;Kiralee M. Hayashi;Arthur W. Toga.
Proceedings of the National Academy of Sciences of the United States of America (2005)
Amyloid Deposition Begins in the Striatum of Presenilin-1 Mutation Carriers from Two Unrelated Pedigrees
William E. Klunk;Julie C. Price;Chester A. Mathis;Nicholas D. Tsopelas.
The Journal of Neuroscience (2007)
Altered Reward Processing in Women Recovered From Anorexia Nervosa
Angela Wagner;Howard Aizenstein;Vijay K Venkatraman;Julie Fudge.
American Journal of Psychiatry (2007)
Amyloid imaging in mild cognitive impairment subtypes
David A. Wolk;David A. Wolk;Julie C. Price;Judy A. Saxton;Beth E. Snitz.
Annals of Neurology (2009)
Decreased Conflict- and Error-Related Activity in the Anterior Cingulate Cortex in Subjects With Schizophrenia
John G. Kerns;Jonathan D. Cohen;Angus W. MacDonald;Melissa K. Johnson.
American Journal of Psychiatry (2005)
Altered Insula Response to Taste Stimuli in Individuals Recovered from Restricting-Type Anorexia Nervosa
Angela Wagner;Angela Wagner;Howard Aizenstein;Laura Mazurkewicz;Julie Fudge.
Neuropsychopharmacology (2008)
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