His main research concerns Immunology, Complement system, Lectin pathway, Inflammation and Pharmacology. His research integrates issues of Reperfusion injury, Ischemia and Myocardial infarction in his study of Immunology. Gregory L. Stahl interconnects Oxidative stress, Internal medicine, Endocrinology, Lectin and Neuropathic pain in the investigation of issues within Complement system.
His biological study spans a wide range of topics, including Collectin and Classical complement pathway. His research in Lectin pathway intersects with topics in Immunoglobulin A, Mannan-binding lectin and Complement. His Inflammation research is multidisciplinary, incorporating perspectives in Periodontitis, Chemotaxis, Endogeny and Arthritis.
His primary areas of investigation include Complement system, Immunology, Mannan-binding lectin, Internal medicine and Molecular biology. His studies in Complement system integrate themes in fields like Lectin and Biochemistry. His Immunology research is multidisciplinary, relying on both Reperfusion injury and Pharmacology.
His research on Mannan-binding lectin also deals with topics like
Gregory L. Stahl spends much of his time researching Complement system, Immunology, Mannan-binding lectin, Alternative complement pathway and Lectin. The Complement system study combines topics in areas such as Inflammation, Molecular biology, Microbiology and Arthritis. His Immunology study incorporates themes from Reperfusion injury, Stroke and Cell biology.
His Mannan-binding lectin study combines topics in areas such as Ebola virus, Innate immune system, Internal medicine and C-type lectin. His study in Alternative complement pathway is interdisciplinary in nature, drawing from both Inflammatory arthritis, Complement factor B, Factor H and Classical complement pathway. The various areas that Gregory L. Stahl examines in his Lectin study include Coagulation and Pharmacology.
Complement system, Immunology, Mannan-binding lectin, Lectin and Alternative complement pathway are his primary areas of study. His studies deal with areas such as Proteases, Molecular biology, Internal medicine and Endocrinology as well as Complement system. The study incorporates disciplines such as Reperfusion injury, Ischemia and Stroke in addition to Immunology.
His study focuses on the intersection of Ischemia and fields such as Pharmacology with connections in the field of Troponin I, Troponin and Complement component 5. Gregory L. Stahl has included themes like Innate immune system, Microbiology and Ebola virus in his Mannan-binding lectin study. His Alternative complement pathway study integrates concerns from other disciplines, such as Tumor necrosis factor alpha, Complement factor B, Complement membrane attack complex and Arthritis.
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Predominant role for C5b-9 in renal ischemia/reperfusion injury
Wuding Zhou;Conrad A. Farrar;Katsushige Abe;Julian R. Pratt.
Journal of Clinical Investigation (2000)
Myocardial Infarction and Apoptosis After Myocardial Ischemia and Reperfusion Role of the Terminal Complement Components and Inhibition by Anti-C5 Therapy
Antti P. Vakeva;Azin Agah;Scott A. Rollins;Louis A. Matis.
Circulation (1998)
Human IgA Activates the Complement System via the Mannan-Binding Lectin Pathway
Roos A;Bouwman Lh;van Gijlswijk-Janssen Dj;Faber-Krol Mc.
Journal of Immunology (2001)
Pharmacology and Biological Efficacy of a Recombinant, Humanized, Single-Chain Antibody C5 Complement Inhibitor in Patients Undergoing Coronary Artery Bypass Graft Surgery With Cardiopulmonary Bypass
Jane C. K. Fitch;Scott Rollins;Louis Matis;Bernadette Alford.
Circulation (1999)
Glomerular activation of the lectin pathway of complement in IgA nephropathy is associated with more severe renal disease.
Anja Roos;Maria Pia Rastaldi;Novella Calvaresi;Beatrijs D. Oortwijn.
Journal of The American Society of Nephrology (2006)
Complement Activation after Oxidative Stress : Role of the Lectin Complement Pathway
Charles D. Collard;Antti Väkevä;Margaret A. Morrissey;Azin Agah.
American Journal of Pathology (2000)
Reduced Inflammation and Tissue Damage in Transgenic Rabbits Overexpressing 15-Lipoxygenase and Endogenous Anti-inflammatory Lipid Mediators
Charles N. Serhan;Ashish Jain;Sylvie Marleau;Clary Clish.
Journal of Immunology (2003)
Inhibition of Mannose-Binding Lectin Reduces Postischemic Myocardial Reperfusion Injury
James E. Jordan;Michael C. Montalto;Gregory L. Stahl.
Circulation (2001)
Mannose-Binding Lectin Is a Regulator of Inflammation That Accompanies Myocardial Ischemia and Reperfusion Injury
Mary C. Walsh;Todd Bourcier;Kazue Takahashi;Lei Shi.
Journal of Immunology (2005)
Neutrophil-derived 5′-Adenosine Monophosphate Promotes Endothelial Barrier Function via CD73-mediated Conversion to Adenosine and Endothelial A2B Receptor Activation
Paul F. Lennon;Cormac T. Taylor;Gregory L. Stahl;Sean P. Colgan.
Journal of Experimental Medicine (1998)
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