What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Gene
- Immune system
Steffen Thiel focuses on Mannan-binding lectin, Biochemistry, Complement system, Lectin pathway and Lectin.
His Mannan-binding lectin study integrates concerns from other disciplines, such as MASP2 and C3-convertase.
His work carried out in the field of Complement system brings together such families of science as Innate immune system and Pattern recognition receptor.
Steffen Thiel has included themes like Factor H and Complement component 2 in his Lectin pathway study.
His work in Lectin addresses issues such as Glycoprotein, which are connected to fields such as Peptide sequence, Function and Cell.
His biological study spans a wide range of topics, including Pseudomonas aeruginosa, Microbiology and Burn injury.
His most cited work include:
- A second serine protease associated with mannan-binding lectin that activates complement (779 citations)
- Interplay between promoter and structural gene variants control basal serum level of mannan-binding protein. (703 citations)
- COLLECTIONS AND FICOLINS: HUMORAL LECTINS OF THE INNATE IMMUNE DEFENSE (657 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Mannan-binding lectin, Complement system, Lectin pathway, Immunology and Biochemistry.
His research in Mannan-binding lectin intersects with topics in Internal medicine and Collectin.
His Internal medicine study combines topics from a wide range of disciplines, such as Gastroenterology and Diabetes mellitus, Endocrinology.
His Complement system research integrates issues from Proteases and Innate immune system.
His Lectin pathway research is multidisciplinary, relying on both C-type lectin, Complement component 2, Cell biology, MASP2 and Complement membrane attack complex.
As part of his studies on Biochemistry, he frequently links adjacent subjects like C3-convertase.
He most often published in these fields:
- Mannan-binding lectin (44.72%)
- Complement system (44.27%)
- Lectin pathway (35.06%)
What were the highlights of his more recent work (between 2015-2021)?
- Complement system (44.27%)
- Lectin pathway (35.06%)
- Immunology (33.93%)
In recent papers he was focusing on the following fields of study:
His main research concerns Complement system, Lectin pathway, Immunology, Mannan-binding lectin and Internal medicine.
His Complement system research incorporates themes from Innate immune system and Cell biology.
His Lectin pathway research incorporates elements of Serine protease, Lectin, Biochemistry, Collectin and Proteases.
His study on Inflammation is often connected to In patient as part of broader study in Immunology.
His Mannan-binding lectin research includes themes of MASP2 and Arthritis.
His Internal medicine research is multidisciplinary, incorporating elements of Gastroenterology and Diabetes mellitus, Type 1 diabetes, Endocrinology.
Between 2015 and 2021, his most popular works were:
- Lectin Complement Pathway Proteins in Healthy Individuals. (50 citations)
- Structure and activation of C1, the complex initiating the classical pathway of the complement cascade (42 citations)
- Functional and structural insight into properdin control of complement alternative pathway amplification (37 citations)
In his most recent research, the most cited papers focused on:
- Internal medicine
- Gene
- Immune system
Complement system, Lectin pathway, Internal medicine, Mannan-binding lectin and Immunology are his primary areas of study.
His study in Complement system is interdisciplinary in nature, drawing from both Proteases and Innate immune system.
His Lectin pathway research focuses on Serine protease and how it relates to Healthy individuals.
Mannan-binding lectin is the subject of his research, which falls under Lectin.
Steffen Thiel has researched Immunology in several fields, including Diabetes mellitus, Prospective cohort study, Genotyping and Kidney.
His research integrates issues of Biochemistry and Complement in his study of Alternative complement pathway.
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