1967 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary scientific interests are in Internal medicine, Endocrinology, Insulin, Islet and Diabetes mellitus. The Internal medicine study combines topics in areas such as Gene expression and Streptozotocin. All of his Endocrinology and Glucagon, Beta cell, Pancreas, Somatostatin and Type 2 diabetes investigations are sub-components of the entire Endocrinology study.
His research integrates issues of Receptor, In vitro and Arginine in his study of Insulin. His work deals with themes such as Glucose tolerance test, Pancreatic hormone, Transplantation and Endocrine system, which intersect with Islet. His studies in Diabetes mellitus integrate themes in fields like Inflammation, Exocytosis, Glucose transporter and Surgery.
Gordon C. Weir spends much of his time researching Internal medicine, Endocrinology, Islet, Insulin and Diabetes mellitus. His Internal medicine research focuses on Pancreas, Beta cell, Insulin resistance, Insulin oscillation and Glucagon secretion. His Glucagon, Somatostatin, Streptozotocin, Pancreatic islets and Secretion study are his primary interests in Endocrinology.
His work carried out in the field of Islet brings together such families of science as Cell culture, Immunology, Immune system, Cell biology and Transplantation. His Transplantation research is multidisciplinary, incorporating perspectives in Immunosuppression and Ratón. His Insulin research is multidisciplinary, incorporating elements of Hormone and Arginine.
The scientist’s investigation covers issues in Internal medicine, Endocrinology, Islet, Insulin and Diabetes mellitus. His Internal medicine study incorporates themes from Type 1 diabetes and Type 2 diabetes. His Endocrinology study frequently draws connections to adjacent fields such as Cell biology.
His research in Islet intersects with topics in Oxygen supply, Peritoneal cavity, Transplantation and Pathology. The various areas that Gordon C. Weir examines in his Insulin study include Receptor, Unfolded protein response, Genetically modified mouse and Type 2 Diabetes Mellitus. In Diabetes mellitus, Gordon C. Weir works on issues like Cell, which are connected to Bioinformatics.
Gordon C. Weir focuses on Internal medicine, Endocrinology, Diabetes mellitus, Islet and Transplantation. His Internal medicine research incorporates elements of Type 2 diabetes and Oncology. Gordon C. Weir works on Endocrinology which deals in particular with Insulin.
His study in Diabetes mellitus is interdisciplinary in nature, drawing from both Cell, Pancreas and Intensive care medicine. His studies deal with areas such as Pharmacology, Peritoneal cavity, Immune system and Cell biology as well as Islet. His Transplantation study also includes
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Induced pluripotent stem cells generated without viral integration
Matthias Stadtfeld;Masaki Nagaya;Masaki Nagaya;Jochen Utikal;Gordon Weir;Gordon Weir.
In vitro cultivation of human islets from expanded ductal tissue
Susan Bonner-Weir;Monica Taneja;Gordon C. Weir;Krystyna Tatarkiewicz.
Proceedings of the National Academy of Sciences of the United States of America (2000)
Five Stages of Evolving Beta-Cell Dysfunction During Progression to Diabetes
Gordon C. Weir;Susan Bonner-Weir.
Insulinotropin: glucagon-like peptide I (7-37) co-encoded in the glucagon gene is a potent stimulator of insulin release in the perfused rat pancreas.
S Mojsov;G C Weir;J F Habener.
Journal of Clinical Investigation (1987)
Pancreatic islet cell toxicity of amylin associated with type-2 diabetes mellitus
Alfredo Lorenzo;Bronwyn Razzaboni;Gordon C. Weir;Bruce A. Yankner.
Chronic Hyperglycemia Triggers Loss of Pancreatic β Cell Differentiation in an Animal Model of Diabetes
Jean-Christophe Jonas;Arun Sharma;Wendy Hasenkamp;Hasan Ilkova.
Journal of Biological Chemistry (1999)
Compensatory growth of pancreatic beta-cells in adult rats after short-term glucose infusion.
Susan Bonner-Weir;Deanna Deery;John L Leahy;Gordon C Weir.
Continuous, clonal, insulin- and somatostatin-secreting cell lines established from a transplantable rat islet cell tumor.
Adi F. Gazdar;William L. Chick;Herbert K. Oie;Harvie L. Sims.
Proceedings of the National Academy of Sciences of the United States of America (1980)
Partial pancreatectomy in the rat and subsequent defect in glucose-induced insulin release.
S Bonner-Weir;D F Trent;G C Weir.
Journal of Clinical Investigation (1983)
New sources of pancreatic beta-cells.
Susan Bonner-Weir;Gordon C Weir.
Nature Biotechnology (2005)
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