What is he best known for?
The fields of study he is best known for:
- Gene
- Immune system
- Cytokine
The scientist’s investigation covers issues in Immunology, NOD mice, Nod, Insulitis and T cell.
David V. Serreze works mostly in the field of Immunology, limiting it down to concerns involving Cytotoxic T cell and, occasionally, Antigen.
His NOD mice research is multidisciplinary, incorporating elements of Cytokine, T lymphocyte, Bone marrow and Locus.
His work in Bone marrow tackles topics such as Antigen presentation which are related to areas like Cell biology.
His research integrates issues of Congenic, Internal medicine and MHC class I, Major histocompatibility complex in his study of Nod.
The concepts of his Antigen-presenting cell study are interwoven with issues in Molecular biology and Lymphocyte.
His most cited work include:
- B lymphocytes are essential for the initiation of T cell-mediated autoimmune diabetes: analysis of a new "speed congenic" stock of NOD.Ig mu null mice. (530 citations)
- Interleukin 4 reverses T cell proliferative unresponsiveness and prevents the onset of diabetes in nonobese diabetic mice. (490 citations)
- Single domain antibodies: promising experimental and therapeutic tools in infection and immunity. (462 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Immunology, NOD mice, Nod, Major histocompatibility complex and T cell.
His biological study spans a wide range of topics, including Diabetes mellitus, Type 1 diabetes and Cytotoxic T cell.
His study in NOD mice is interdisciplinary in nature, drawing from both Congenic and MHC class I, CD8, Antigen.
In Nod, he works on issues like Islet, which are connected to Transplantation.
The Major histocompatibility complex study combines topics in areas such as Haematopoiesis, Human leukocyte antigen and Antigen presentation.
His T cell study combines topics in areas such as Beta cell, Lymphocyte, Cell biology, Bone marrow and FOXP3.
He most often published in these fields:
- Immunology (73.78%)
- NOD mice (68.90%)
- Nod (57.93%)
What were the highlights of his more recent work (between 2013-2021)?
- NOD mice (68.90%)
- Immunology (73.78%)
- Nod (57.93%)
In recent papers he was focusing on the following fields of study:
David V. Serreze mainly focuses on NOD mice, Immunology, Nod, T cell and Cytotoxic T cell.
He combines subjects such as Phenotype, Major histocompatibility complex, Insulitis and Cell biology with his study of NOD mice.
His studies in Immunology integrate themes in fields like Type 1 diabetes and Pancreatic islets.
His research in Nod intersects with topics in Peripherin, Lacrimal gland and CD8, Antigen.
His T cell study integrates concerns from other disciplines, such as Myeloid and Regulatory B cells.
In his research, Metabolism and Antibiotics is intimately related to Immune system, which falls under the overarching field of Cytotoxic T cell.
Between 2013 and 2021, his most popular works were:
- Autoimmune manifestations in aged mice arise from early-life immune dysregulation. (30 citations)
- Mouse Invariant Monoclonal Antibody NKT14: A Novel Tool to Manipulate iNKT Cell Function In Vivo. (19 citations)
- Decreased pancreatic acinar cell number in type 1 diabetes. (18 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Immune system
- Cytokine
His main research concerns Immunology, NOD mice, Nod, T cell and Genetics.
His works in Antibody and Naive B cell are all subjects of inquiry into Immunology.
The various areas that he examines in his NOD mice study include Insulitis, Atacicept, Epitope, Cell biology and Peripherin.
His Nod research incorporates elements of Adoptive cell transfer and CD8, Antigen.
His study in the fields of Major histocompatibility complex, Negative selection, Immune system and Inbred strain under the domain of Genetics overlaps with other disciplines such as Genetic divergence.
David V. Serreze interconnects Cytotoxic T cell and Autoimmunity in the investigation of issues within Major histocompatibility complex.
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