D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Genetics and Molecular Biology D-index 69 Citations 17,329 154 World Ranking 1598 National Ranking 818

Research.com Recognitions

Awards & Achievements

1994 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Genetics

Claudio Basilico spends much of his time researching Fibroblast growth factor, Cell biology, Molecular biology, Cancer research and Signal transduction. His work deals with themes such as Cell surface receptor and Basic fibroblast growth factor, which intersect with Fibroblast growth factor. The concepts of his Cell biology study are interwoven with issues in DNA, Cell type and Immunology.

His Molecular biology research includes elements of Enhancer, 3T3 cells, Growth factor, Keratinocyte growth factor and Gene. The study incorporates disciplines such as SOX2 and Wnt signaling pathway in addition to Cancer research. His work carried out in the field of Signal transduction brings together such families of science as Chondrocyte, Cellular differentiation and Phosphorylation.

His most cited work include:

  • The FGF family of growth factors and oncogenes. (1088 citations)
  • Developmental-specific activity of the FGF-4 enhancer requires the synergistic action of Sox2 and Oct-3. (683 citations)
  • The anticoagulation factor protein S and its relative, Gas6, are ligands for the Tyro 3/Axl family of receptor tyrosine kinases (614 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Molecular biology, Cell biology, Fibroblast growth factor, Cell culture and Mutant. Claudio Basilico combines subjects such as Transcription, Gene, DNA, DNA replication and Virus with his study of Molecular biology. His Cell biology study deals with 3T3 cells intersecting with Cell growth.

His study in Fibroblast growth factor is interdisciplinary in nature, drawing from both Signal transduction, Growth factor and Cellular differentiation. He focuses mostly in the field of Cell culture, narrowing it down to topics relating to Transformation and, in certain cases, Hamster. The various areas that he examines in his Mutant study include Mutation and Baby hamster kidney cell.

He most often published in these fields:

  • Molecular biology (43.38%)
  • Cell biology (30.14%)
  • Fibroblast growth factor (24.20%)

What were the highlights of his more recent work (between 2006-2018)?

  • Cell biology (30.14%)
  • Cancer research (10.50%)
  • SOX2 (5.48%)

In recent papers he was focusing on the following fields of study:

Claudio Basilico focuses on Cell biology, Cancer research, SOX2, Fibroblast growth factor and Wnt signaling pathway. His Cell biology study combines topics in areas such as Chondrocyte, Transcription factor, 3T3 cells and Coronal suture. His studies in Cancer research integrate themes in fields like Cyclin D, Adipogenesis, Cyclin A and Cyclin B.

His SOX2 research includes themes of Hippo signaling pathway, Cancer stem cell, Stem cell and Mesenchymal stem cell. His research on Stem cell frequently links to adjacent areas such as Molecular biology. His Fibroblast growth factor research is multidisciplinary, incorporating perspectives in Cell culture, Cellular differentiation, Phosphatase, Osteoblast and Signal transduction.

Between 2006 and 2018, his most popular works were:

  • Sox2 maintains self renewal of tumor-initiating cells in osteosarcomas (168 citations)
  • Sox2 antagonizes the Hippo pathway to maintain stemness in cancer cells (140 citations)
  • SOX2 regulates YAP1 to maintain stemness and determine cell fate in the osteo-adipo lineage. (133 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The FGF family of growth factors and oncogenes.

Claudio Basilico;David Moscatelli.
Advances in Cancer Research (1992)

1724 Citations

Developmental-specific activity of the FGF-4 enhancer requires the synergistic action of Sox2 and Oct-3.

Huabing Yuan;Nicoletta Corbi;Claudio Basilico;Lisa Dailey.
Genes & Development (1995)

998 Citations

Mechanisms underlying differential responses to FGF signaling.

Lisa Dailey;Davide Ambrosetti;Alka Mansukhani;Claudio Basilico.
Cytokine & Growth Factor Reviews (2005)

884 Citations

The anticoagulation factor protein S and its relative, Gas6, are ligands for the Tyro 3/Axl family of receptor tyrosine kinases

Trevor N Stitt;Greg Conn;Martin Goret;Martin Goret;Cary Lai.
Cell (1995)

773 Citations

Neuronal defects and delayed wound healing in mice lacking fibroblast growth factor 2.

Sagrario Ortega;Michael Ittmann;Stephen H. Tsang;Michelle Ehrlich.
Proceedings of the National Academy of Sciences of the United States of America (1998)

700 Citations

An oncogene isolated by transfection of Kaposi's sarcoma DNA encodes a growth factor that is a member of the FGF family

Pasquale Delli Bovi;Anna Maria Curatola;Francis G. Kern;Angela Greco.
Cell (1987)

655 Citations

Synergistic activation of the fibroblast growth factor 4 enhancer by Sox2 and Oct-3 depends on protein-protein interactions facilitated by a specific spatial arrangement of factor binding sites.

Davide-Carlo Ambrosetti;Claudio Basilico;Lisa Dailey.
Molecular and Cellular Biology (1997)

518 Citations

Targeted disruption of the FGF2 gene does not prevent choroidal neovascularization in a murine model

Takao Tobe;Sagrario Ortega;Jose D. Luna;Hiroaki Ozaki.
American Journal of Pathology (1998)

478 Citations

FGF signaling inhibits chondrocyte proliferation and regulates bone development through the STAT-1 pathway

Malika Sahni;Davide-Carlo Ambrosetti;Alka Mansukhani;Rachel Gertner.
Genes & Development (1999)

474 Citations

Compensation by Fibroblast Growth Factor 1 (FGF1) Does Not Account for the Mild Phenotypic Defects Observed in FGF2 Null Mice

David L. Miller;Sagrario Ortega;Omar Bashayan;Ross Basch.
Molecular and Cellular Biology (2000)

441 Citations

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