1994 - Fellow of the American Association for the Advancement of Science (AAAS)
Claudio Basilico spends much of his time researching Fibroblast growth factor, Cell biology, Molecular biology, Cancer research and Signal transduction. His work deals with themes such as Cell surface receptor and Basic fibroblast growth factor, which intersect with Fibroblast growth factor. The concepts of his Cell biology study are interwoven with issues in DNA, Cell type and Immunology.
His Molecular biology research includes elements of Enhancer, 3T3 cells, Growth factor, Keratinocyte growth factor and Gene. The study incorporates disciplines such as SOX2 and Wnt signaling pathway in addition to Cancer research. His work carried out in the field of Signal transduction brings together such families of science as Chondrocyte, Cellular differentiation and Phosphorylation.
His primary scientific interests are in Molecular biology, Cell biology, Fibroblast growth factor, Cell culture and Mutant. Claudio Basilico combines subjects such as Transcription, Gene, DNA, DNA replication and Virus with his study of Molecular biology. His Cell biology study deals with 3T3 cells intersecting with Cell growth.
His study in Fibroblast growth factor is interdisciplinary in nature, drawing from both Signal transduction, Growth factor and Cellular differentiation. He focuses mostly in the field of Cell culture, narrowing it down to topics relating to Transformation and, in certain cases, Hamster. The various areas that he examines in his Mutant study include Mutation and Baby hamster kidney cell.
Claudio Basilico focuses on Cell biology, Cancer research, SOX2, Fibroblast growth factor and Wnt signaling pathway. His Cell biology study combines topics in areas such as Chondrocyte, Transcription factor, 3T3 cells and Coronal suture. His studies in Cancer research integrate themes in fields like Cyclin D, Adipogenesis, Cyclin A and Cyclin B.
His SOX2 research includes themes of Hippo signaling pathway, Cancer stem cell, Stem cell and Mesenchymal stem cell. His research on Stem cell frequently links to adjacent areas such as Molecular biology. His Fibroblast growth factor research is multidisciplinary, incorporating perspectives in Cell culture, Cellular differentiation, Phosphatase, Osteoblast and Signal transduction.
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The FGF family of growth factors and oncogenes.
Claudio Basilico;David Moscatelli.
Advances in Cancer Research (1992)
Developmental-specific activity of the FGF-4 enhancer requires the synergistic action of Sox2 and Oct-3.
Huabing Yuan;Nicoletta Corbi;Claudio Basilico;Lisa Dailey.
Genes & Development (1995)
Mechanisms underlying differential responses to FGF signaling.
Lisa Dailey;Davide Ambrosetti;Alka Mansukhani;Claudio Basilico.
Cytokine & Growth Factor Reviews (2005)
The anticoagulation factor protein S and its relative, Gas6, are ligands for the Tyro 3/Axl family of receptor tyrosine kinases
Trevor N Stitt;Greg Conn;Martin Goret;Martin Goret;Cary Lai.
Neuronal defects and delayed wound healing in mice lacking fibroblast growth factor 2.
Sagrario Ortega;Michael Ittmann;Stephen H. Tsang;Michelle Ehrlich.
Proceedings of the National Academy of Sciences of the United States of America (1998)
An oncogene isolated by transfection of Kaposi's sarcoma DNA encodes a growth factor that is a member of the FGF family
Pasquale Delli Bovi;Anna Maria Curatola;Francis G. Kern;Angela Greco.
Synergistic activation of the fibroblast growth factor 4 enhancer by Sox2 and Oct-3 depends on protein-protein interactions facilitated by a specific spatial arrangement of factor binding sites.
Davide-Carlo Ambrosetti;Claudio Basilico;Lisa Dailey.
Molecular and Cellular Biology (1997)
Targeted disruption of the FGF2 gene does not prevent choroidal neovascularization in a murine model
Takao Tobe;Sagrario Ortega;Jose D. Luna;Hiroaki Ozaki.
American Journal of Pathology (1998)
FGF signaling inhibits chondrocyte proliferation and regulates bone development through the STAT-1 pathway
Malika Sahni;Davide-Carlo Ambrosetti;Alka Mansukhani;Rachel Gertner.
Genes & Development (1999)
Compensation by Fibroblast Growth Factor 1 (FGF1) Does Not Account for the Mild Phenotypic Defects Observed in FGF2 Null Mice
David L. Miller;Sagrario Ortega;Omar Bashayan;Ross Basch.
Molecular and Cellular Biology (2000)
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