Clive Dickson spends much of his time researching Cell biology, Fibroblast growth factor, Internal medicine, Endocrinology and Molecular biology. His biological study spans a wide range of topics, including Fibroblast growth factor receptor 4, Fibroblast growth factor receptor 3 and Otic placode. Clive Dickson interconnects Limb bud, Zone of polarizing activity, Cellular differentiation and Limb development in the investigation of issues within Fibroblast growth factor.
His Internal medicine course of study focuses on Mesenchyme and Cell–cell interaction, Keratinocyte growth factor, Keratinocyte, Myogenesis and Gene expression. His Molecular biology research is multidisciplinary, relying on both Mouse mammary tumor virus, Chromosome, Gene and Oncogene. In Gene, Clive Dickson works on issues like Cancer research, which are connected to Cyclin D1, Cyclin-dependent kinase and Cyclin D.
His primary areas of study are Molecular biology, Cell biology, Fibroblast growth factor, Gene and Internal medicine. The various areas that Clive Dickson examines in his Molecular biology study include Open reading frame, Promoter, Provirus, Mouse mammary tumor virus and Transcription. His research integrates issues of Fibroblast growth factor receptor 4, Fibroblast growth factor receptor 3, Fibroblast growth factor receptor 2 and FGF10 in his study of Cell biology.
In his research on the topic of Fibroblast growth factor, 3T3 cells is strongly related with Fibroblast. The study incorporates disciplines such as Cancer research and DNA in addition to Gene. His Internal medicine research integrates issues from Wound healing, Endocrinology and Oncology.
Clive Dickson mainly investigates Cell biology, Internal medicine, Fibroblast growth factor, Endocrinology and FGF10. Cell biology connects with themes related to Epithelium in his study. He focuses mostly in the field of Internal medicine, narrowing it down to matters related to Wound healing and, in some cases, Dermal fibroblast, Downregulation and upregulation, Integrin and Transforming growth factor beta.
His Fibroblast growth factor research includes elements of Cell, Cancer research, Epidermis and Hair follicle. Clive Dickson combines subjects such as Cell signaling, Fibroblast Growth Factor Receptor 2b, Fibroblast growth factor receptor 2, FGF8 and Receptor with his study of Endocrinology. His FGF10 study incorporates themes from Ventricle, Morphogenesis, Mutant and Cellular differentiation.
Fibroblast growth factor, Cell biology, Internal medicine, Endocrinology and Receptor are his primary areas of study. His Fibroblast growth factor research incorporates elements of Mammary gland development, Morphogenesis and Hindbrain. Cell biology is frequently linked to Morpholino in his study.
Clive Dickson mostly deals with FGF10 in his studies of Internal medicine. His FGF10 research includes themes of Phenotype, Hypoplasia, Hair follicle, Wild type and Transplantation. His Endocrinology study integrates concerns from other disciplines, such as Epithelium, Mesenchyme, Mesenchymal stem cell, Sonic hedgehog and Mesoderm.
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An important role for the IIIb isoform of fibroblast growth factor receptor 2 (FGFR2) in mesenchymal-epithelial signalling during mouse organogenesis
L De Moerlooze;B Spencer-Dene;J M Revest;M Hajihosseini.
Amplification and Overexpression of Cyclin D1 in Breast Cancer Detected by Immunohistochemical Staining
Cheryl Gillett;Vera Fantl;Rosalind Smith;Charlotte Fisher.
Cancer Research (1994)
Fibroblast growth factor signaling in tumorigenesis
Richard Grose;Clive Dickson.
Cytokine & Growth Factor Reviews (2005)
D11S287, a putative oncogene on chromosome 11q13, is amplified and expressed in squamous cell and mammary carcinomas and linked to BCL-1.
Lammie Ga;Fantl;Smith R;Schuuring E.
Potential oncogene product related to growth factors.
Clive Dickson;Gordon Peters.
Disruption of Fgf10/Fgfr2b-coordinated epithelial-mesenchymal interactions causes cleft palate
Ritva Rice;Bradley Spencer-Dene;Elaine C. Connor;Amel Gritli-Linde.
Journal of Clinical Investigation (2004)
Tumorigenesis by mouse mammary tumor virus: Proviral activation of a cellular gene in the common integration region int-2
C. Dickson;R. Smith;S. Brookes;G. Peters.
FGF-7 (keratinocyte growth factor) expression during mouse development suggests roles in myogenesis, forebrain regionalisation and epithelial-mesenchymal interactions
Ivor J. Mason;Frances Fuller-Pace;Rosalind Smith;Clive Dickson.
Mechanisms of Development (1994)
Tumorigenesis by mouse mammary tumor virus: Evidence for a common region for provirus integration in mammary tumors
Gordon Peters;Sharon Brookes;Rosalind Smith;Clive Dickson.
Subcellular Fate of the int-2 Oncoprotein Is Determined by Choice of Initiation Codon
Piers Acland;Mark Dixon;Gordon Peters;Clive Dickson.
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