What is he best known for?
The fields of study he is best known for:
Greg Lemke mainly investigates Cell biology, C-Mer Tyrosine Kinase, GAS6, Molecular biology and Receptor.
His studies deal with areas such as Regulation of gene expression and Immunology as well as Cell biology.
His C-Mer Tyrosine Kinase study combines topics in areas such as Neurogenesis, Cancer research and AXL receptor tyrosine kinase.
The concepts of his Molecular biology study are interwoven with issues in Epidermal growth factor, Schwann cell, Myelin protein zero and Complementary DNA, Gene.
His Receptor research includes elements of Microglia, Dengue virus, RNA splicing, PI3K/AKT/mTOR pathway and Gene isoform.
As a member of one scientific family, Greg Lemke mostly works in the field of Receptor tyrosine kinase, focusing on ROR1 and, on occasion, Tyrosine kinase and Thrombin.
His most cited work include:
- Neuregulin 1 and Susceptibility to Schizophrenia (1442 citations)
- Aberrant neural and cardiac development in mice lacking the ErbB4 neuregulin receptor. (983 citations)
- TAM receptors are pleiotropic inhibitors of the innate immune response. (701 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Cell biology, Receptor, Receptor tyrosine kinase, Molecular biology and Schwann cell.
His Cell biology study frequently involves adjacent topics like Immunology.
His Receptor research focuses on Neuroscience and how it connects with Anatomy.
His work deals with themes such as ROR1, Cancer research and Immune system, which intersect with Receptor tyrosine kinase.
His research in Molecular biology intersects with topics in Myelin protein zero, Complementary DNA, Gene, Promoter and Proteolipid protein 1.
His Schwann cell study integrates concerns from other disciplines, such as Endocrinology, Myelin, Internal medicine, Axon and Growth factor.
He most often published in these fields:
- Cell biology (41.90%)
- Receptor (26.26%)
- Receptor tyrosine kinase (22.91%)
What were the highlights of his more recent work (between 2011-2021)?
- Receptor (26.26%)
- Receptor tyrosine kinase (22.91%)
- Cell biology (41.90%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are Receptor, Receptor tyrosine kinase, Cell biology, GAS6 and Phosphatidylserine.
His Receptor research integrates issues from Virus, Phospholipid and Apoptosis.
Greg Lemke is interested in C-Mer Tyrosine Kinase, which is a branch of Receptor tyrosine kinase.
He has researched C-Mer Tyrosine Kinase in several fields, including Retinal pigment epithelium, Retina, Retinal and Retinitis pigmentosa.
His Cell biology study incorporates themes from Immune system and Microglia.
The Signal transduction study combines topics in areas such as Proinflammatory cytokine, Langerhans cell differentiation and Cell growth.
Between 2011 and 2021, his most popular works were:
- The TIM and TAM families of phosphatidylserine receptors mediate dengue virus entry. (311 citations)
- Biology of the TAM receptors (265 citations)
- TAM receptors regulate multiple features of microglial physiology (246 citations)
In his most recent research, the most cited papers focused on:
His primary scientific interests are in Cell biology, Receptor tyrosine kinase, C-Mer Tyrosine Kinase, Signal transduction and Receptor.
Greg Lemke is involved in the study of Cell biology that focuses on Neurogenesis in particular.
Greg Lemke works mostly in the field of Receptor tyrosine kinase, limiting it down to topics relating to AXL receptor tyrosine kinase and, in certain cases, Langerhans cell, Cellular differentiation, Cell growth, Langerhans cell differentiation and Proinflammatory cytokine.
Greg Lemke studied C-Mer Tyrosine Kinase and Innate immune system that intersect with Immune tolerance.
His research on Signal transduction frequently links to adjacent areas such as Immune system.
His research in Receptor tackles topics such as Phosphatidylserine which are related to areas like Gene knockdown, Apoptotic cell clearance, Dengue virus, Virus and Ectopic expression.
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