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Biology and Biochemistry

D-Index
82
Citations
25069
World Ranking
3682
National Ranking
1833

Overview

Albee Messing is affiliated with the University of Wisconsin-Madison in the United States. Their research primarily focuses on the field of Biochemistry, Genetics and Molecular Biology, with a particular emphasis on Molecular Biology, Immunology, Cellular and Molecular Neuroscience, Neurology, and Cancer Research.

The main topics covered in Messing's work include:

  • RNA regulation and disease
  • RNA Research and Splicing
  • Interferon and immune responses
  • RNA and protein synthesis mechanisms
  • Cancer-related molecular mechanisms research
  • Neuroinflammation and Neurodegeneration Mechanisms
  • Neurogenesis and neuroplasticity mechanisms

Messing has contributed to several recent scientific papers, including:

  • "Reactive astrocyte nomenclature, definitions, and future directions," 2021, Nature Neuroscience
  • "Regulation of GFAP Expression," 2021, ASN NEURO
  • "GFAP at 50," 2020, ASN NEURO
  • "Antisense therapy in a rat model of Alexander disease reverses GFAP pathology, white matter deficits, and motor impairment," 2021, Science Translational Medicine
  • "Type II Alexander disease caused by splicing errors and aberrant overexpression of an uncharacterized GFAP isoform," 2020, Human Mutation

The most frequent coauthors collaborating with Messing include:

  • Tracy L. Hagemann
  • Amy Waldman
  • Michael Brenner
  • Ni-Hsuan Lin
  • Mel Β. Feany

Messing's work is published across multiple venues, with repeated contributions to:

  • ASN NEURO
  • Human Mutation
  • Neurology Genetics
  • bioRxiv (Cold Spring Harbor Laboratory)
  • Nature Neuroscience

Best Publications

  • Reactive astrocyte nomenclature, definitions, and future directions

    Carole Escartin;Elena Galea;András Lakatos;James P. O’Callaghan

  • Astrocytes: a central element in neurological diseases

    Milos Pekny;Milos Pekny;Milos Pekny;Marcela Pekna;Marcela Pekna;Marcela Pekna;Albee Messing;Christian Steinhäuser

  • Mutations in GFAP, encoding glial fibrillary acidic protein, are associated with Alexander disease.

    Michael Brenner;Anne B. Johnson;Odile Boespflug-Tanguy;Diana Rodriguez

  • hGFAP-cre transgenic mice for manipulation of glial and neuronal function in vivo.

    Lang Zhuo;Martin Theis;Ikuri Alvarez-Maya;Michael Brenner

  • Deletion of the KV1.1 Potassium Channel Causes Epilepsy in Mice

    Sharon L. Smart;Valeri Lopantsev;C.L. Zhang;Carol A. Robbins

  • Transgenic mice harboring SV40 t-antigen genes develop characteristic brain tumors

    Ralph L. Brinster;Howard Y. Chen;Albee Messing;Terry van Dyke

  • Deletion of brain dystroglycan recapitulates aspects of congenital muscular dystrophy

    Steven A. Moore;Fumiaki Saito;Jianguo Chen;Daniel E. Michele

  • Early inactivation of p53 tumor suppressor gene cooperating with NF1 loss induces malignant astrocytoma.

    Yuan Zhu;Yuan Zhu;Frantz Guignard;Dawen Zhao;Li Liu

  • GFAP promoter directs astrocyte-specific expression in transgenic mice

    M. Brenner;W. C. Kisseberth;Yuan Su;F. Besnard

  • Nrf2 activation in astrocytes protects against neurodegeneration in mouse models of familial amyotrophic lateral sclerosis.

    Marcelo R. Vargas;Delinda A. Johnson;Daniel W. Sirkis;Albee Messing

  • Targeted deletion in astrocyte intermediate filament (Gfap) alters neuronal physiology

    M A McCall;R G Gregg;R R Behringer;M Brenner

  • Live astrocytes visualized by green fluorescent protein in transgenic mice.

    Lang Zhuo;Biao Sun;Chuan-Li Zhang;Alan Fine

  • Cre/loxP-mediated inactivation of the murine Pten tumor suppressor gene.

    Ralf Lesche;Matthias Groszer;Matthias Groszer;Jing Gao;Ying Wang

  • GFAP promoter elements required for region‐specific and astrocyte‐specific expression

    Youngjin Lee;Albee Messing;Mu Su;Michael Brenner

  • Conditional disruption of β1 integrin in Schwann cells impedes interactions with axons

    M. Laura Feltri;Diana Graus Porta;Stefano C. Previtali;Alessandro Nodari

  • Receptor tyrosine kinase ErbB4 modulates neuroblast migration and placement in the adult forebrain.

    E. S. Anton;H. T. Ghashghaei;Janet L. Weber;Corey McCann

  • Neurological diseases as primary gliopathies: a reassessment of neurocentrism.

    Alexei Verkhratsky;Alexei Verkhratsky;Alexei Verkhratsky;Michael V. Sofroniew;Albee Messing;Nihal C. deLanerolle

  • Accelerated hippocampal spreading depression and enhanced locomotory activity in mice with astrocyte-directed inactivation of connexin43.

    Martin Theis;Regina Jauch;Lang Zhuo;Dina Speidel

  • Absence of the β subunit (cchb1) of the skeletal muscle dihydropyridine receptor alters expression of the α1 subunit and eliminates excitation-contraction coupling

    Ronald G. Gregg;Albee Messing;Caroline Strube;Maryline Beurg

  • SV40 enhancer and large-T antigen are instrumental in development of choroid plexus tumours in transgenic mice

    Richard D. Palmiter;Howard Y. Chen;Albee Messing;Ralph L. Brinster

Frequent Co-Authors

Michael Brenner
Michael Brenner University of Alabama at Birmingham
Lawrence Wrabetz
Lawrence Wrabetz University at Buffalo, State University of New York
Angelo Quattrini
Angelo Quattrini Vita-Salute San Raffaele University
James E. Goldman
James E. Goldman Columbia University
Ralph L. Brinster
Ralph L. Brinster University of Pennsylvania
Richard D. Palmiter
Richard D. Palmiter University of Washington
M. Laura Feltri
M. Laura Feltri University at Buffalo, State University of New York
Molly S. Shoichet
Molly S. Shoichet University of Toronto
Alexei Verkhratsky
Alexei Verkhratsky University of Manchester

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