His primary areas of study are Cell biology, Neuroscience, Myelin, Schwann cell and Connexin. His Cell biology research includes themes of Cell and Adherens junction. His work carried out in the field of Neuroscience brings together such families of science as Inherited neuropathies and Sciatic nerve.
His Myelin research also works with subjects such as
Steven S. Scherer mostly deals with Cell biology, Neuroscience, Myelin, Schwann cell and Pathology. His Cell biology research incorporates themes from Wallerian degeneration, Mutant and Immunology. He interconnects Molecular genetics and NODAL in the investigation of issues within Neuroscience.
His work investigates the relationship between Myelin and topics such as Gene that intersect with problems in Disease. His research in Schwann cell intersects with topics in Gene expression, Neuroglia, Transcription factor, Axon and Sciatic nerve. His Pathology study combines topics in areas such as Peripheral neuropathy, Peripheral and Chronic inflammatory demyelinating polyneuropathy.
His main research concerns Phenotype, Disease, Mutation, Internal medicine and Genetics. The Phenotype study combines topics in areas such as Autophagy and Spinocerebellar ataxia. His work in the fields of Disease, such as Tooth disease, overlaps with other areas such as Reimbursement.
His studies deal with areas such as Zinc finger, Myelopathy, Weakness and Bioinformatics as well as Mutation. His Internal medicine study integrates concerns from other disciplines, such as Longitudinal study and Endocrinology. His research investigates the link between Mutant and topics such as Neurofilament that cross with problems in Cell biology.
Steven S. Scherer focuses on Chronic inflammatory demyelinating polyneuropathy, Pathology, Gene, Immunology and Autoantibody. His Chronic inflammatory demyelinating polyneuropathy research integrates issues from Myelin, Sciatic nerve, Autoimmunity and Periostin. His Pathology research is multidisciplinary, incorporating elements of Nerve root, Nerve conduction velocity, Electrophysiology and Sensory system.
His work on Single-nucleotide polymorphism, Regulation of gene expression, Gene duplication and Charcot-Marie-Tooth Disease Type 1A as part of general Gene research is often related to Na+/K+-ATPase, thus linking different fields of science. His work in Immunology is not limited to one particular discipline; it also encompasses Idiopathic Neuropathy. Phenotype is a subfield of Genetics that he studies.
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Connexin mutations in X-linked Charcot-Marie-Tooth disease
J Bergoffen;SS Scherer;S Wang;MO Scott.
Connexin32 is a myelin-related protein in the PNS and CNS
Steven S. Scherer;Suzanne M. Deschênes;Yi-Tian Xu;Judith B. Grinspan.
The Journal of Neuroscience (1995)
A Common Ankyrin-G-Based Mechanism Retains KCNQ and NaV Channels at Electrically Active Domains of the Axon
Zongming Pan;Tingching Kao;Zsolt Horvath;Julia Lemos.
The Journal of Neuroscience (2006)
Released form of CNTF receptor alpha component as a soluble mediator of CNTF responses
Samuel Davis;Thomas H. Aldrich;Nancy Y. Ip;Neil Stahl.
KCNQ2 is a nodal K+ channel.
Jérôme J. Devaux;Kleopas A. Kleopa;Edward C. Cooper;Steven S. Scherer.
The Journal of Neuroscience (2004)
Regulation of ciliary neurotrophic factor expression in myelin-related Schwann cells in vivo
Beth Friedman;Steven S. Scherer;John S. Rudge;Maureen Helgren.
Disease mechanisms in inherited neuropathies
Ueli Suter;Steven S. Scherer.
Nature Reviews Neuroscience (2003)
Investigations of caspr2, an autoantigen of encephalitis and neuromyotonia.
Eric Lancaster;Maartje G. M. Huijbers;Vered Bar;Anna Boronat.
Annals of Neurology (2011)
Connexin32‐null mice develop demyelinating peripheral neuropathy
Steven S. Scherer;Yi-Tian Xu;Eric Nelles;Kenneth Fischbeck.
The Axonal Membrane Protein Caspr, a Homologue of Neurexin IV, Is a Component of the Septate-like Paranodal Junctions That Assemble during Myelination
Steven Einheber;George Zanazzi;William Ching;Steven Scherer.
Journal of Cell Biology (1997)
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