D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 72 Citations 17,358 149 World Ranking 2632 National Ranking 1389

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Mutation

His primary areas of study are Cell biology, Neuroscience, Myelin, Schwann cell and Connexin. His Cell biology research includes themes of Cell and Adherens junction. His work carried out in the field of Neuroscience brings together such families of science as Inherited neuropathies and Sciatic nerve.

His Myelin research also works with subjects such as

  • Cellular localization that connect with fields like Golgi apparatus and Regulation of gene expression,
  • Molecular biology most often made with reference to Mutant. Steven S. Scherer has researched Schwann cell in several fields, including Schwann cell differentiation, Focal adhesion, Phenotype, Transcription factor and Axon. The study incorporates disciplines such as X chromosome and Oligodendrocyte in addition to Connexin.

His most cited work include:

  • Connexin mutations in X-linked Charcot-Marie-Tooth disease (909 citations)
  • A Common Ankyrin-G-Based Mechanism Retains KCNQ and NaV Channels at Electrically Active Domains of the Axon (438 citations)
  • Connexin32 is a myelin-related protein in the PNS and CNS (363 citations)

What are the main themes of his work throughout his whole career to date?

Steven S. Scherer mostly deals with Cell biology, Neuroscience, Myelin, Schwann cell and Pathology. His Cell biology research incorporates themes from Wallerian degeneration, Mutant and Immunology. He interconnects Molecular genetics and NODAL in the investigation of issues within Neuroscience.

His work investigates the relationship between Myelin and topics such as Gene that intersect with problems in Disease. His research in Schwann cell intersects with topics in Gene expression, Neuroglia, Transcription factor, Axon and Sciatic nerve. His Pathology study combines topics in areas such as Peripheral neuropathy, Peripheral and Chronic inflammatory demyelinating polyneuropathy.

He most often published in these fields:

  • Cell biology (35.44%)
  • Neuroscience (29.11%)
  • Myelin (21.94%)

What were the highlights of his more recent work (between 2017-2021)?

  • Phenotype (13.92%)
  • Disease (7.59%)
  • Mutation (5.06%)

In recent papers he was focusing on the following fields of study:

His main research concerns Phenotype, Disease, Mutation, Internal medicine and Genetics. The Phenotype study combines topics in areas such as Autophagy and Spinocerebellar ataxia. His work in the fields of Disease, such as Tooth disease, overlaps with other areas such as Reimbursement.

His studies deal with areas such as Zinc finger, Myelopathy, Weakness and Bioinformatics as well as Mutation. His Internal medicine study integrates concerns from other disciplines, such as Longitudinal study and Endocrinology. His research investigates the link between Mutant and topics such as Neurofilament that cross with problems in Cell biology.

Between 2017 and 2021, his most popular works were:

  • Harmonizing Clinical Sequencing and Interpretation for the eMERGE III Network (44 citations)
  • Neurofascin antibodies in autoimmune, genetic, and idiopathic neuropathies. (43 citations)
  • Myopathy associated BAG3 mutations lead to protein aggregation by stalling Hsp70 networks (32 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Mutation

Steven S. Scherer focuses on Chronic inflammatory demyelinating polyneuropathy, Pathology, Gene, Immunology and Autoantibody. His Chronic inflammatory demyelinating polyneuropathy research integrates issues from Myelin, Sciatic nerve, Autoimmunity and Periostin. His Pathology research is multidisciplinary, incorporating elements of Nerve root, Nerve conduction velocity, Electrophysiology and Sensory system.

His work on Single-nucleotide polymorphism, Regulation of gene expression, Gene duplication and Charcot-Marie-Tooth Disease Type 1A as part of general Gene research is often related to Na+/K+-ATPase, thus linking different fields of science. His work in Immunology is not limited to one particular discipline; it also encompasses Idiopathic Neuropathy. Phenotype is a subfield of Genetics that he studies.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Connexin mutations in X-linked Charcot-Marie-Tooth disease

J Bergoffen;SS Scherer;S Wang;MO Scott.
Science (1993)

1293 Citations

Connexin32 is a myelin-related protein in the PNS and CNS

Steven S. Scherer;Suzanne M. Deschênes;Yi-Tian Xu;Judith B. Grinspan.
The Journal of Neuroscience (1995)

566 Citations

A Common Ankyrin-G-Based Mechanism Retains KCNQ and NaV Channels at Electrically Active Domains of the Axon

Zongming Pan;Tingching Kao;Zsolt Horvath;Julia Lemos.
The Journal of Neuroscience (2006)

525 Citations

Released form of CNTF receptor alpha component as a soluble mediator of CNTF responses

Samuel Davis;Thomas H. Aldrich;Nancy Y. Ip;Neil Stahl.
Science (1993)

504 Citations

KCNQ2 is a nodal K+ channel.

Jérôme J. Devaux;Kleopas A. Kleopa;Edward C. Cooper;Steven S. Scherer.
The Journal of Neuroscience (2004)

436 Citations

Regulation of ciliary neurotrophic factor expression in myelin-related Schwann cells in vivo

Beth Friedman;Steven S. Scherer;John S. Rudge;Maureen Helgren.
Neuron (1992)

433 Citations

Disease mechanisms in inherited neuropathies

Ueli Suter;Steven S. Scherer.
Nature Reviews Neuroscience (2003)

427 Citations

Investigations of caspr2, an autoantigen of encephalitis and neuromyotonia.

Eric Lancaster;Maartje G. M. Huijbers;Vered Bar;Anna Boronat.
Annals of Neurology (2011)

413 Citations

Connexin32‐null mice develop demyelinating peripheral neuropathy

Steven S. Scherer;Yi-Tian Xu;Eric Nelles;Kenneth Fischbeck.
Glia (1998)

395 Citations

The Axonal Membrane Protein Caspr, a Homologue of Neurexin IV, Is a Component of the Septate-like Paranodal Junctions That Assemble during Myelination

Steven Einheber;George Zanazzi;William Ching;Steven Scherer.
Journal of Cell Biology (1997)

386 Citations

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