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Biology and Biochemistry

D-Index
57
Citations
9627
World Ranking
14033
National Ranking
5920

Overview

John Kamholz is affiliated with the University of Iowa Hospitals and Clinics in the United States. Their research primarily spans the fields of medicine and neuroscience, with a focus on neurology. They have contributed significantly to subfields including molecular biology, cellular and molecular neuroscience, pathology and forensic medicine, as well as psychiatry and mental health.

Their work covers several main research topics such as transcranial magnetic stimulation studies, vestibular and auditory disorders, multiple sclerosis research, genetic neurodegenerative diseases, fibromyalgia and chronic fatigue syndrome research, neurological disorders and treatments, and stroke rehabilitation and recovery.

Kamholz has published in a variety of academic venues, notably including Brain Sciences, Neurology, Multiple Sclerosis Journal - Experimental Translational and Clinical, PLoS ONE, and Nature Medicine. These journals are among the most frequent outlets for their publications.

Recent papers authored or coauthored by Kamholz include:

  • Post-COVID-19 Fatigue: Potential Contributing Factors, 2020, Brain Sciences
  • Response Variability in Transcranial Direct Current Stimulation: Why Sex Matters, 2020, Frontiers in Psychiatry
  • Efficacy of Diet on Fatigue and Quality of Life in Multiple Sclerosis, 2022, Neurology
  • Impact of the Swank and Wahls elimination dietary interventions on fatigue and quality of life in relapsing-remitting multiple sclerosis: The WAVES randomized parallel-arm clinical trial, 2021, Multiple Sclerosis Journal - Experimental Translational and Clinical
  • Multiple sclerosis patients have an altered gut mycobiome and increased fungal to bacterial richness, 2022, PLoS ONE

Collaboration has been an important aspect of their research career. Frequent coauthors include Thorsten Rudroff, Craig D. Workman, Alexandra C. Fietsam, Linda Snetselaar, and Tyler J. Titcomb.

Best Publications

  • Neurological dysfunction and axonal degeneration in Charcot–Marie–Tooth disease type 1A

    Karen M. Krajewski;Richard A. Lewis;Darren R. Fuerst;Cheryl Turansky

  • Human cDNA clones for four species of G alpha s signal transduction protein

    P. Bray;A. Carter;C. Simons;V. Guo

  • Alternative splicing accounts for the four forms of myelin basic protein

    Francesca de Ferra;Helen Engh;Lynn Hudson;John Kamholz

  • Phenotypic clustering in MPZ mutations.

    Michael E. Shy;Agnes Jáni;Karen Krajewski;Marina Grandis

  • Patients lacking the major CNS myelin protein, proteolipid protein 1, develop length-dependent axonal degeneration in the absence of demyelination and inflammation.

    James Y. Garbern;Donald A. Yool;Gregory J. Moore;Ian B. Wilds

  • DM-GRASP, a novel immunoglobulin superfamily axonal surface protein that supports neurite extension.

    Frank R. Burns;Stephanie von Kannen;Leslie Guy;Jonathan A. Raper

  • Identification of three forms of human myelin basic protein by cDNA cloning.

    J Kamholz;F de Ferra;C Puckett;R Lazzarini

  • Transient central nervous system white matter abnormality in X‐linked Charcot‐Marie‐Tooth disease

    Henry L. Paulson;James Y. Garbern;Timothy F. Hoban;Karen M. Krajewski

  • Post-covid-19 fatigue: Potential contributing factors

    Thorsten Rudroff;Thorsten Rudroff;Alexandra C. Fietsam;Justin R. Deters;Andrew D. Bryant

  • Differential regulation of the 2′,3′-cyclic nucleotide 3′-phosphodiesterase gene during oligodendrocyte development

    Steven S. Scherer;Peter E. Braun;Judith Grinspan;Ellen Collarini

  • The molecular pathogenesis of Pelizaeus-Merzbacher disease.

    James Garbern;Franca Cambi;Michael Shy;John Kamholz

  • Axons regulate Schwann cell expression of the POU transcription factor SCIP

    S. S. Scherer;Da-Yuan Wang;R. Kuhn;G. Lemke

  • Proteolipid protein is necessary in peripheral as well as central myelin

    James Y. Garbern;Franca Cambi;Xue Ming Tang;Anders A.F. Sima

  • Beta 4 integrin expression in myelinating Schwann cells is polarized, developmentally regulated and axonally dependent

    M.L. Feltri;S.S. Scherer;R. Nemni;J. Kamholz

  • Heterozygous P0 knockout mice develop a peripheral neuropathy that resembles chronic inflammatory demyelinating polyneuropathy (CIDP).

    Michael E. Shy;Edgardo Arroyo;John Sladky;Daniela Menichella

  • Axons modulate the expression of transforming growth factor-betas in Schwann cells.

    Steven S. Scherer;John Kamholz;Sonia B. Jakowlew

  • Heterogeneous duplications in patients with Pelizaeus-Merzbacher disease suggest a mechanism of coupled homologous and nonhomologous recombination.

    Karen J. Woodward;Maria Cundall;Karen Sperle;Erik A. Sistermans

  • Molecular analysis of the 18q- syndrome--and correlation with phenotype

    A D Kline;M E White;R Wapner;K Rojas

  • Double-stranded RNA unwinding and modifying activity is detected ubiquitously in primary tissues and cell lines.

    R W Wagner;C Yoo;L Wrabetz;J Kamholz

  • Organization and expression of the human myelin basic protein gene.

    J. Kamholz;J. Toffenetti;R. A. Lazzarini

Frequent Co-Authors

Michael E. Shy
Michael E. Shy University of Iowa
Steven S. Scherer
Steven S. Scherer University of Pennsylvania
Lawrence Wrabetz
Lawrence Wrabetz University at Buffalo, State University of New York
David E Pleasure
David E Pleasure University of California, Davis
Maik Hüttemann
Maik Hüttemann Wayne State University
Lawrence I. Grossman
Lawrence I. Grossman Wayne State University
Elaine H. Zackai
Elaine H. Zackai Children's Hospital of Philadelphia
Robert A. Lazzarini
Robert A. Lazzarini Icahn School of Medicine at Mount Sinai
Laura L. Boles Ponto
Laura L. Boles Ponto University of Iowa Hospitals and Clinics
Thomas D. Bird
Thomas D. Bird University of Washington

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