Rudolf Martini spends much of his time researching Myelin, Schwann cell, Cell biology, Neuroscience and Axon. His Myelin study frequently intersects with other fields, such as Immunology. His Immunology study integrates concerns from other disciplines, such as Threshold of pain and Transient receptor potential channel.
His research in Schwann cell intersects with topics in Peripheral nervous system, Reinnervation, Genetically modified mouse and Compact myelin. His work carried out in the field of Cell biology brings together such families of science as Mutation, Embryonic stem cell and Mutant. His Neuroscience research is multidisciplinary, incorporating elements of Connexin 32, Gene and Peripheral nerve.
His primary scientific interests are in Myelin, Cell biology, Immunology, Schwann cell and Neuroscience. His work deals with themes such as Peripheral nervous system, Connexin 32, Axon and Pathology, which intersect with Myelin. His Pathology research includes elements of Peripheral, Regeneration and Anatomy.
Rudolf Martini combines subjects such as Myelin-associated glycoprotein, Downregulation and upregulation, Mutant and Proteolipid protein 1 with his study of Cell biology. His Immunology research is multidisciplinary, relying on both Molecular biology and Macrophage. His Schwann cell research is multidisciplinary, incorporating perspectives in Cell, Sciatic nerve, Neuroglia, Myelin maintenance and Immunoelectron microscopy.
Rudolf Martini mainly focuses on Neuroscience, Immunology, Myelin, Neuroinflammation and Neurodegeneration. As part of the same scientific family, Rudolf Martini usually focuses on Neuroscience, concentrating on Gene and intersecting with Late onset. His Myelin research incorporates themes from Schwann cell, Axon, Cell biology, Wallerian degeneration and Peripheral nervous system.
The various areas that Rudolf Martini examines in his Schwann cell study include Cytokine and Myelin protein zero. Rudolf Martini has researched Cell biology in several fields, including Schwann cell differentiation, Hair cell, Downregulation and upregulation, Axonal loss and Motor neuron. His research integrates issues of Neuronal ceroid lipofuscinosis, Palmitoyl protein thioesterase and Nervous system in his study of Neurodegeneration.
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Immunoelectron microscopic localization of neural cell adhesion molecules (L1, N-CAM, and MAG) and their shared carbohydrate epitope and myelin basic protein in developing sciatic nerve.
R Martini;M Schachner.
Journal of Cell Biology (1986)
Disruption of the mouse L1 gene leads to malformations of the nervous system
Miriam Dahme;Udo Bartsch;Rudolf Martini;Rudolf Martini;Brigitte Anliker.
Nature Genetics (1997)
Expression and functional roles of neural cell surface molecules and extracellular matrix components during development and regeneration of peripheral nerves
Rudolf Martini.
Journal of Neurocytology (1994)
Mouse P0 gene disruption leads to hypomyelination, abnormal expression of recognition molecules, and degeneration of myelin and axons
Karl Peter Giese;Rudolf Martini;Greg Lemke;Philippe Soriano.
Cell (1992)
Mice deficient for the myelin-associated glycoprotein show subtle abnormalities in myelin.
Dirk Montag;Karl Peter Giese;Udo Bartsch;Rudolf Martini.
Neuron (1994)
Immunoelectron microscopic localization of neural cell adhesion molecules (L1, N-CAM, and myelin-associated glycoprotein) in regenerating adult mouse sciatic nerve.
R Martini;M Schachner.
Journal of Cell Biology (1988)
CNTF is a major protective factor in demyelinating CNS disease: A neurotrophic cytokine as modulator in neuroinflammation
Ralf A. Linker;Mathias Mäurer;Stefanie Gaupp;Rudolf Martini.
Nature Medicine (2002)
Hypermyelination and demyelinating peripheral neuropathy in Pmp22 -deficient mice
Katrin Adlkofer;Rudolf Martini;Adriano Aguzzi;Jürgen Zielasek.
Nature Genetics (1995)
Structural Abnormalities and Deficient Maintenance of Peripheral Nerve Myelin in Mice Lacking the Gap Junction Protein Connexin 32
Patrizia Anzini;Dirk H.-H. Neuberg;Melitta Schachner;Eric Nelles.
The Journal of Neuroscience (1997)
Protein zero (P0)-deficient mice show myelin degeneration in peripheral nerves characteristic of inherited human neuropathies
R Martini;J Zielasek;K V Toyka;K P Giese.
Nature Genetics (1995)
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