D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 53 Citations 12,705 135 World Ranking 8421 National Ranking 637

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Genetics

Rudolf Martini spends much of his time researching Myelin, Schwann cell, Cell biology, Neuroscience and Axon. His Myelin study frequently intersects with other fields, such as Immunology. His Immunology study integrates concerns from other disciplines, such as Threshold of pain and Transient receptor potential channel.

His research in Schwann cell intersects with topics in Peripheral nervous system, Reinnervation, Genetically modified mouse and Compact myelin. His work carried out in the field of Cell biology brings together such families of science as Mutation, Embryonic stem cell and Mutant. His Neuroscience research is multidisciplinary, incorporating elements of Connexin 32, Gene and Peripheral nerve.

His most cited work include:

  • Immunoelectron microscopic localization of neural cell adhesion molecules (L1, N-CAM, and MAG) and their shared carbohydrate epitope and myelin basic protein in developing sciatic nerve. (446 citations)
  • Mouse P0 gene disruption leads to hypomyelination, abnormal expression of recognition molecules, and degeneration of myelin and axons (438 citations)
  • Disruption of the mouse L1 gene leads to malformations of the nervous system (433 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Myelin, Cell biology, Immunology, Schwann cell and Neuroscience. His work deals with themes such as Peripheral nervous system, Connexin 32, Axon and Pathology, which intersect with Myelin. His Pathology research includes elements of Peripheral, Regeneration and Anatomy.

Rudolf Martini combines subjects such as Myelin-associated glycoprotein, Downregulation and upregulation, Mutant and Proteolipid protein 1 with his study of Cell biology. His Immunology research is multidisciplinary, relying on both Molecular biology and Macrophage. His Schwann cell research is multidisciplinary, incorporating perspectives in Cell, Sciatic nerve, Neuroglia, Myelin maintenance and Immunoelectron microscopy.

He most often published in these fields:

  • Myelin (68.10%)
  • Cell biology (52.15%)
  • Immunology (41.10%)

What were the highlights of his more recent work (between 2012-2021)?

  • Neuroscience (33.74%)
  • Immunology (41.10%)
  • Myelin (68.10%)

In recent papers he was focusing on the following fields of study:

Rudolf Martini mainly focuses on Neuroscience, Immunology, Myelin, Neuroinflammation and Neurodegeneration. As part of the same scientific family, Rudolf Martini usually focuses on Neuroscience, concentrating on Gene and intersecting with Late onset. His Myelin research incorporates themes from Schwann cell, Axon, Cell biology, Wallerian degeneration and Peripheral nervous system.

The various areas that Rudolf Martini examines in his Schwann cell study include Cytokine and Myelin protein zero. Rudolf Martini has researched Cell biology in several fields, including Schwann cell differentiation, Hair cell, Downregulation and upregulation, Axonal loss and Motor neuron. His research integrates issues of Neuronal ceroid lipofuscinosis, Palmitoyl protein thioesterase and Nervous system in his study of Neurodegeneration.

Between 2012 and 2021, his most popular works were:

  • Neuroinflammation in the peripheral nerve: Cause, modulator, or bystander in peripheral neuropathies? (49 citations)
  • Long-lasting significant functional improvement in chronic severe spinal cord injury following scar resection and polyethylene glycol implantation. (49 citations)
  • Myelin and macrophages in the PNS: An intimate relationship in trauma and disease. (48 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Immunoelectron microscopic localization of neural cell adhesion molecules (L1, N-CAM, and MAG) and their shared carbohydrate epitope and myelin basic protein in developing sciatic nerve.

R Martini;M Schachner.
Journal of Cell Biology (1986)

687 Citations

Disruption of the mouse L1 gene leads to malformations of the nervous system

Miriam Dahme;Udo Bartsch;Rudolf Martini;Rudolf Martini;Brigitte Anliker.
Nature Genetics (1997)

658 Citations

Expression and functional roles of neural cell surface molecules and extracellular matrix components during development and regeneration of peripheral nerves

Rudolf Martini.
Journal of Neurocytology (1994)

601 Citations

Mouse P0 gene disruption leads to hypomyelination, abnormal expression of recognition molecules, and degeneration of myelin and axons

Karl Peter Giese;Rudolf Martini;Greg Lemke;Philippe Soriano.
Cell (1992)

537 Citations

Mice deficient for the myelin-associated glycoprotein show subtle abnormalities in myelin.

Dirk Montag;Karl Peter Giese;Udo Bartsch;Rudolf Martini.
Neuron (1994)

511 Citations

Immunoelectron microscopic localization of neural cell adhesion molecules (L1, N-CAM, and myelin-associated glycoprotein) in regenerating adult mouse sciatic nerve.

R Martini;M Schachner.
Journal of Cell Biology (1988)

505 Citations

CNTF is a major protective factor in demyelinating CNS disease: A neurotrophic cytokine as modulator in neuroinflammation

Ralf A. Linker;Mathias Mäurer;Stefanie Gaupp;Rudolf Martini.
Nature Medicine (2002)

438 Citations

Hypermyelination and demyelinating peripheral neuropathy in Pmp22 -deficient mice

Katrin Adlkofer;Rudolf Martini;Adriano Aguzzi;Jürgen Zielasek.
Nature Genetics (1995)

402 Citations

Structural Abnormalities and Deficient Maintenance of Peripheral Nerve Myelin in Mice Lacking the Gap Junction Protein Connexin 32

Patrizia Anzini;Dirk H.-H. Neuberg;Melitta Schachner;Eric Nelles.
The Journal of Neuroscience (1997)

335 Citations

Protein zero (P0)-deficient mice show myelin degeneration in peripheral nerves characteristic of inherited human neuropathies

R Martini;J Zielasek;K V Toyka;K P Giese.
Nature Genetics (1995)

333 Citations

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