His primary areas of study are Cell biology, Neuroscience, Myelin, Tenascin-R and Tenascin C. The concepts of his Cell biology study are interwoven with issues in Immunocytochemistry, Neurite, Mutant, Retina and Transplantation. Many of his research projects under Retina are closely connected to Mesaxon with Mesaxon, tying the diverse disciplines of science together.
The Neuroscience study combines topics in areas such as Synaptic plasticity and Long-term potentiation. Udo Bartsch interconnects Extracellular matrix, Neural stem cell and Optic nerve in the investigation of issues within Myelin. His studies examine the connections between Tenascin C and genetics, as well as such issues in Cerebellar cortex, with regards to Northern blot and Golgi apparatus.
Udo Bartsch mainly focuses on Cell biology, Neuroscience, Retina, Myelin and Optic nerve. Udo Bartsch connects Cell biology with Tenascin-R in his study. His Neuroscience research is multidisciplinary, incorporating elements of Synaptic plasticity and Immunoglobulin superfamily.
His Retina research incorporates themes from Neurotrophic factors, Stem cell, Cell type and Immunology. His Myelin study combines topics in areas such as Axon and Neural stem cell. His Neurite research is multidisciplinary, incorporating perspectives in Tenascin C, Molecular biology and In situ hybridization.
His main research concerns Cell biology, Neuronal ceroid lipofuscinosis, Retinal degeneration, Astrogliosis and Pathology. Udo Bartsch is interested in Neural stem cell, which is a field of Cell biology. His work in Neuronal ceroid lipofuscinosis addresses issues such as Neuroinflammation, which are connected to fields such as Epilepsy, Neuroscience, Dementia and Stem-cell therapy.
In his research on the topic of Astrogliosis, Virology and Infectious disease is strongly related with Neurodegeneration. He has included themes like Molecular biology, Knockout mouse and Western blot in his Pathology study. When carried out as part of a general Retina research project, his work on Inner nuclear layer is frequently linked to work in Tissue Degeneration, therefore connecting diverse disciplines of study.
Udo Bartsch mainly investigates Cell biology, Neurodegeneration, Neuronal ceroid lipofuscinosis, Ciliary neurotrophic factor and Retinal ganglion. The concepts of his Cell biology study are interwoven with issues in Regulation of gene expression, Astrogliosis and Pathology. Within one scientific family, he focuses on topics pertaining to Neuroinflammation under Neurodegeneration, and may sometimes address concerns connected to Stem-cell therapy, Dementia, Neurology and Neuroscience.
His Neuronal ceroid lipofuscinosis research incorporates themes from Allograft inflammatory factor 1 and Sequestosome 1. Udo Bartsch interconnects Lesion, Retinal, Optic nerve and Neural stem cell in the investigation of issues within Ciliary neurotrophic factor. The study incorporates disciplines such as Cell, Cell culture, Ganglion, Glial cell line-derived neurotrophic factor and Transplantation in addition to Retinal ganglion.
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Disruption of the mouse L1 gene leads to malformations of the nervous system
Miriam Dahme;Udo Bartsch;Rudolf Martini;Rudolf Martini;Brigitte Anliker.
Nature Genetics (1997)
Terminal differentiation of myelin-forming oligodendrocytes depends on the transcription factor Sox10
C. Claus Stolt;Stephan Rehberg;Marius Ader;Petra Lommes.
Genes & Development (2002)
Mice deficient for the myelin-associated glycoprotein show subtle abnormalities in myelin.
Dirk Montag;Karl Peter Giese;Udo Bartsch;Rudolf Martini.
The Disintegrin/Metalloproteinase ADAM10 Is Essential for the Establishment of the Brain Cortex
Ellen Jorissen;Johannes Prox;Christian Bernreuther;Silvio Weber.
The Journal of Neuroscience (2010)
A Multicolor Panel of Novel Lentiviral “Gene Ontology” (LeGO) Vectors for Functional Gene Analysis
Kristoffer Weber;Udo Bartsch;Carol Stocking;Boris Fehse.
Molecular Therapy (2008)
Expression of tenascin in the developing and adult cerebellar cortex.
S Bartsch;U Bartsch;U Dorries;A Faissner.
The Journal of Neuroscience (1992)
Lack of evidence that myelin-associated glycoprotein is a major inhibitor of axonal regeneration in the CNS
Udo Bartsch;Christine E. Bandtlow;Lisa Schnell;Susanne Bartsch.
Enhanced expression of the developmentally regulated extracellular matrix molecule tenascin following adult brain injury
Eric D. Laywell;Ulrich Dorries;Udo Bartsch;Andreas Faissner.
Proceedings of the National Academy of Sciences of the United States of America (1992)
Mice deficient for tenascin-R display alterations of the extracellular matrix and decreased axonal conduction velocities in the CNS.
P Weber;U Bartsch;U Bartsch;M N Rasband;R Czaniera.
The Journal of Neuroscience (1999)
Immunohistological localization of the adhesion molecules L1, N-CAM, and MAG in the developing and adult optic nerve of mice.
Udo Bartsch;Frank Kirchhoff;Melitta Schachner.
The Journal of Comparative Neurology (1989)
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