Karl Peter Giese

What is he best known for?

The fields of study he is best known for:

• Gene
• Genetics
• Mutation

Karl Peter Giese mainly focuses on Cell biology, Neuroscience, Long-term potentiation, Hippocampal formation and Ca2+/calmodulin-dependent protein kinase. His Cell biology research incorporates elements of Embryonic stem cell, Molecular cloning, Myelin and Voltage-gated potassium channel. His study focuses on the intersection of Long-term potentiation and fields such as Kinase with connections in the field of Inhibitory postsynaptic potential, Neurotransmission, Postsynaptic density and Phosphorylation.

His biological study spans a wide range of topics, including Synaptic plasticity and Hippocampus. His Ca2+/calmodulin-dependent protein kinase research incorporates themes from Autophosphorylation and Mutant. His work deals with themes such as Calmodulin, CAMK2A and Morris water navigation task, which intersect with Autophosphorylation.

His most cited work include:

• Autophosphorylation at Thr286 of the alpha calcium-calmodulin kinase II in LTP and learning. (906 citations)
• Molecular basis of functional diversity of voltage-gated potassium channels in mammalian brain (653 citations)
• Mouse P0 gene disruption leads to hypomyelination, abnormal expression of recognition molecules, and degeneration of myelin and axons (438 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Neuroscience, Ca2+/calmodulin-dependent protein kinase, Cell biology, Long-term potentiation and Hippocampal formation. His studies in Neuroscience integrate themes in fields like Synaptic plasticity, Autophosphorylation, Kinase and Neurodegeneration. His Ca2+/calmodulin-dependent protein kinase research integrates issues from Long-term memory, CREB and Synaptic signaling.

As part of one scientific family, Karl Peter Giese deals mainly with the area of Cell biology, narrowing it down to issues related to the Myelin, and often Embryonic stem cell, Null allele and Axon. His studies deal with areas such as Endocrinology and Postsynaptic density as well as Long-term potentiation. When carried out as part of a general Hippocampal formation research project, his work on Dendritic spine is frequently linked to work in Slow afterhyperpolarization, therefore connecting diverse disciplines of study.

He most often published in these fields:

• Neuroscience (60.71%)
• Ca2+/calmodulin-dependent protein kinase (23.81%)
• Cell biology (22.62%)

What were the highlights of his more recent work (between 2013-2021)?

• Neuroscience (60.71%)
• Ca2+/calmodulin-dependent protein kinase (23.81%)
• Long-term potentiation (21.43%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Neuroscience, Ca2+/calmodulin-dependent protein kinase, Long-term potentiation, Synaptic plasticity and Hippocampal formation. His research combines Autophosphorylation and Neuroscience. His research in Ca2+/calmodulin-dependent protein kinase intersects with topics in Psychoanalysis and Synaptic signaling.

Long-term potentiation is frequently linked to Cell biology in his study. His research investigates the connection between Hippocampal formation and topics such as Stimulation that intersect with issues in Wild type, Signal transduction, Effector and NMDA receptor. His Kinase study incorporates themes from Mutation, Molecular biology, Axon and Mutant.

Between 2013 and 2021, his most popular works were:

• Memory acquisition and retrieval impact different epigenetic processes that regulate gene expression (97 citations)
• Calcium/calmodulin-dependent kinase II and Alzheimer's disease. (72 citations)
• Alzheimer-related decrease in CYFIP2 links amyloid production to tau hyperphosphorylation and memory loss. (27 citations)

In his most recent research, the most cited papers focused on:

• Gene
• Genetics
• Gene expression

Karl Peter Giese mainly investigates Neuroscience, Alzheimer's disease, Synapse, Cerebellum and Frontotemporal lobar degeneration. Neuroscience and Amyloid are frequently intertwined in his study. His Alzheimer's disease research includes elements of Synaptic plasticity, Ca2+/calmodulin-dependent protein kinase, Neurodegeneration, Dementia and Kinase.

His research on Synapse frequently connects to adjacent areas such as Calcium signaling. The Cerebellum study combines topics in areas such as Downregulation and upregulation, Neuroprotection and Tauopathy.