What is he best known for?
The fields of study he is best known for:
Michael Ittmann mostly deals with Prostate cancer, Cancer research, Cancer, Prostate and Internal medicine.
His Prostate cancer research is multidisciplinary, incorporating perspectives in Carcinogenesis, Tumor progression, Metastasis and Pathology.
His studies in Cancer research integrate themes in fields like DNA methylation, microRNA, Fibroblast growth factor, Regulation of gene expression and PTEN.
Michael Ittmann has included themes like Transcription factor and Cell growth in his Cancer study.
His Prostate research includes elements of Basal, Anatomical pathology, Genitourinary system and Adenocarcinoma.
His biological study spans a wide range of topics, including Missense mutation, Endocrinology, Lineage and Gene isoform.
His most cited work include:
- PTEN, a Putative Protein Tyrosine Phosphatase Gene Mutated in Human Brain, Breast, and Prostate Cancer (4224 citations)
- The Molecular Taxonomy of Primary Prostate Cancer (1426 citations)
- Widespread deregulation of microRNA expression in human prostate cancer. (591 citations)
What are the main themes of his work throughout his whole career to date?
Michael Ittmann mainly focuses on Cancer research, Prostate cancer, Internal medicine, Prostate and Cancer.
Michael Ittmann has researched Cancer research in several fields, including Carcinogenesis, Signal transduction, PTEN and Fibroblast growth factor.
His Prostate cancer study integrates concerns from other disciplines, such as Metastasis and Pathology.
His Internal medicine study combines topics in areas such as Endocrinology, Protein kinase B, Oncology and In vivo.
Michael Ittmann works mostly in the field of Prostate, limiting it down to concerns involving Growth factor and, occasionally, Cytokine.
His research investigates the connection between Cancer and topics such as DNA methylation that intersect with problems in Regulation of gene expression.
He most often published in these fields:
- Cancer research (74.79%)
- Prostate cancer (68.91%)
- Internal medicine (44.26%)
What were the highlights of his more recent work (between 2018-2021)?
- Cancer research (74.79%)
- Prostate cancer (68.91%)
- Internal medicine (44.26%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Cancer research, Prostate cancer, Internal medicine, Oncology and Cancer.
His Cancer research research is multidisciplinary, incorporating perspectives in Methylation, Carcinogenesis, Fusion gene, Transcription factor and PI3K/AKT/mTOR pathway.
His Prostate cancer research integrates issues from Tumor progression, Protein kinase B, Prostate and Standard of care.
His biological study deals with issues like Tumor microenvironment, which deal with fields such as Epigenomics.
When carried out as part of a general Internal medicine research project, his work on Clinical recurrence is frequently linked to work in African american, therefore connecting diverse disciplines of study.
His work on Kidney cancer is typically connected to Field based as part of general Cancer study, connecting several disciplines of science.
Between 2018 and 2021, his most popular works were:
- Pan-cancer analysis of whole genomes (538 citations)
- Integrated Proteogenomic Characterization of Clear Cell Renal Cell Carcinoma. (96 citations)
- Proteogenomic Characterization of Endometrial Carcinoma. (56 citations)
In his most recent research, the most cited papers focused on:
Michael Ittmann focuses on Cancer research, Wnt signaling pathway, Cell biology, Progenitor and Stromal cell.
His studies in Cancer research integrate themes in fields like Cancer, Gene knockdown, In vivo and Small interfering RNA.
Endometrial cancer is the focus of his Cancer research.
His study looks at the intersection of In vivo and topics like Lung squamous cell carcinoma with Cell.
He interconnects Metabolic flux analysis, Lipogenesis, Citric acid cycle and Androgen receptor in the investigation of issues within Cell biology.
His research in Progenitor intersects with topics in Transforming growth factor, Tissue homeostasis and WNT5A.
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