His primary areas of study are Retinal, Retina, Retinal degeneration, Genetics and Ophthalmology. Many of his studies on Retinal apply to Genetic enhancement as well. His study focuses on the intersection of Retina and fields such as Optic nerve with connections in the field of CRB1 and Morphogenesis.
His Retinal degeneration research is multidisciplinary, incorporating elements of Endocrinology, Outer nuclear layer, Anatomy, Retinal Rod Photoreceptor Cells and Molecular biology. His Anatomy research is multidisciplinary, incorporating perspectives in Degeneration and Pathology. His work carried out in the field of Genetics brings together such families of science as Electroretinography, Retinal Cone Photoreceptor Cells, Visual phototransduction and Cell biology.
His scientific interests lie mostly in Retinal, Ophthalmology, Retina, Retinal degeneration and Retinitis pigmentosa. His Retinal study integrates concerns from other disciplines, such as Genetics and Cell biology. His Ophthalmology study frequently links to other fields, such as Clinical trial.
He has researched Retina in several fields, including Mutation and Pathology. His work in Retinal degeneration tackles topics such as Molecular biology which are related to areas like Mutation. The concepts of his Stargardt disease study are interwoven with issues in Prospective cohort study and ABCA4.
His primary areas of investigation include Ophthalmology, Retinal, Clinical trial, Visual acuity and Leber congenital amaurosis. His Ophthalmology research incorporates themes from Nystagmus and Prospective cohort study. His studies deal with areas such as Retina, Optical coherence tomography and Calcium as well as Retinal.
His Retina research integrates issues from Degeneration and Pathophysiology. His work deals with themes such as BLUE CONE MONOCHROMACY and Compound heterozygosity, which intersect with Visual acuity. His Retinal degeneration study deals with Genetic enhancement intersecting with Recombinant DNA and Ciliopathy.
Ophthalmology, Retinal, Retina, Stargardt disease and Retinitis pigmentosa are his primary areas of study. His Visual acuity, Leber congenital amaurosis, Outer nuclear layer and Fovea centralis study in the realm of Ophthalmology interacts with subjects such as Childhood blindness. His study in Retinal focuses on Retinal degeneration, RPE65 and Visual phototransduction.
His study in Retina is interdisciplinary in nature, drawing from both Visual field and Transgene. Stargardt disease is a subfield of Genetics that Artur V. Cideciyan explores. The Retinitis pigmentosa study combines topics in areas such as X-linked recessive inheritance and Genetic enhancement.
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Gene therapy restores vision in a canine model of childhood blindness.
Gregory M. Acland;Gustavo D. Aguirre;Jharna Ray;Qi Zhang.
Nature Genetics (2001)
Treatment of Leber Congenital Amaurosis Due to RPE65 Mutations by Ocular Subretinal Injection of Adeno-Associated Virus Gene Vector: Short-Term Results of a Phase I Trial
William W. Hauswirth;Tomas S. Aleman;Shalesh Kaushal;Artur V. Cideciyan.
Human Gene Therapy (2008)
Human gene therapy for RPE65 isomerase deficiency activates the retinoid cycle of vision but with slow rod kinetics
Artur V. Cideciyan;Tomas S. Aleman;Sanford L. Boye;Sharon B. Schwartz.
Proceedings of the National Academy of Sciences of the United States of America (2008)
Interim Results from the International Trial of Second Sight's Visual Prosthesis
Mark S. Humayun;Jessy D. Dorn;Lyndon Da Cruz;Gislin Dagnelie.
Gene Therapy for Leber Congenital Amaurosis Caused by RPE65 Mutations: Safety and Efficacy in 15 Children and Adults Followed Up to 3 Years
Samuel G. Jacobson;Artur V. Cideciyan;Ramakrishna Ratnakaram;Elise Heon.
Archives of Ophthalmology (2012)
Long-Term Restoration of Rod and Cone Vision by Single Dose rAAV-Mediated Gene Transfer to the Retina in a Canine Model of Childhood Blindness
Gregory M. Acland;Gustavo D. Aguirre;Jean Bennett;Tomas S. Aleman.
Molecular Therapy (2005)
Mutation of a nuclear receptor gene, NR2E3 , causes enhanced S cone syndrome, a disorder of retinal cell fate
Neena B. Haider;Samuel G. Jacobson;Artur V. Cideciyan;Ruth Swiderski.
Nature Genetics (2000)
Human retinal gene therapy for Leber congenital amaurosis shows advancing retinal degeneration despite enduring visual improvement
Artur V. Cideciyan;Samuel G. Jacobson;William A. Beltran;Alexander Sumaroka.
Proceedings of the National Academy of Sciences of the United States of America (2013)
Human RPE65 Gene Therapy for Leber Congenital Amaurosis: Persistence of Early Visual Improvements and Safety at 1 Year
Artur V. Cideciyan;William W. Hauswirth;Tomas S. Aleman;Shalesh Kaushal.
Human Gene Therapy (2009)
Genetically engineered large animal model for studying cone photoreceptor survival and degeneration in retinitis pigmentosa
Petters Rm;Alexander Ca;Wells Kd;Collins Eb.
Nature Biotechnology (1997)
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