What is he best known for?
The fields of study he is best known for:
Samuel G. Jacobson mostly deals with Genetics, Retinal, Retina, Retinal degeneration and Retinitis pigmentosa.
The Genetics study combines topics in areas such as Rhodopsin, Visual phototransduction and Cell biology.
His Retinal study improves the overall literature in Ophthalmology.
In his work, Morphogenesis is strongly intertwined with Optic nerve, which is a subfield of Retina.
His Retinal degeneration research is multidisciplinary, relying on both Endocrinology, Outer nuclear layer, Neurodegeneration, Retinal Rod Photoreceptor Cells and Internal medicine.
He interconnects Phenotype, Anatomy, Point mutation, Molecular biology and Visual field in the investigation of issues within Retinitis pigmentosa.
His most cited work include:
- Gene therapy restores vision in a canine model of childhood blindness. (1025 citations)
- Treatment of Leber Congenital Amaurosis Due to RPE65 Mutations by Ocular Subretinal Injection of Adeno-Associated Virus Gene Vector: Short-Term Results of a Phase I Trial (799 citations)
- Human gene therapy for RPE65 isomerase deficiency activates the retinoid cycle of vision but with slow rod kinetics (590 citations)
What are the main themes of his work throughout his whole career to date?
Samuel G. Jacobson mainly focuses on Retinal, Genetics, Retina, Ophthalmology and Retinitis pigmentosa.
His Retinal research incorporates themes from Genetic enhancement and Cell biology.
His research on Genetics frequently links to adjacent areas such as Molecular biology.
His Retina study combines topics in areas such as Rhodopsin, Anatomy and Pathology.
His biological study spans a wide range of topics, including Peripherin, Gene mutation and Exon.
Samuel G. Jacobson has included themes like Endocrinology, Degeneration, Phenotype, Internal medicine and Visual phototransduction in his Retinal degeneration study.
He most often published in these fields:
- Retinal (35.43%)
- Genetics (33.27%)
- Retina (31.89%)
What were the highlights of his more recent work (between 2013-2021)?
- Ophthalmology (32.09%)
- Retinal (35.43%)
- Retina (31.89%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Ophthalmology, Retinal, Retina, Retinal degeneration and Retinitis pigmentosa.
His study in Ophthalmology is interdisciplinary in nature, drawing from both Clinical trial and Cohort.
His Retinal research is multidisciplinary, incorporating elements of Optical coherence tomography and Genetic enhancement.
His work deals with themes such as Fovea centralis, Pathophysiology and Anatomy, which intersect with Retina.
His research in Retinal degeneration intersects with topics in Mutation, Retinal pigment epithelium, Phenotype, Pathology and Retinal regeneration.
His Retinitis pigmentosa research includes elements of Maculopathy, Allele and Transgene.
Between 2013 and 2021, his most popular works were:
- Improvement and decline in vision with gene therapy in childhood blindness. (248 citations)
- Effect of an intravitreal antisense oligonucleotide on vision in Leber congenital amaurosis due to a photoreceptor cilium defect (89 citations)
- Canine retina has a primate fovea-like bouquet of cone photoreceptors which is affected by inherited macular degenerations. (71 citations)
In his most recent research, the most cited papers focused on:
Samuel G. Jacobson mainly investigates Retina, Ophthalmology, Retinal, Retinal degeneration and Retinitis pigmentosa.
His studies in Retina integrate themes in fields like Anatomy, Foveal and Pathology.
His study in the field of Visual acuity, Electroretinography and Leber congenital amaurosis is also linked to topics like Childhood blindness.
His research brings together the fields of Genetic enhancement and Retinal.
His work carried out in the field of Retinal degeneration brings together such families of science as Retinal regeneration, Disease, Mutant and ABCA4.
His Retinitis pigmentosa study combines topics from a wide range of disciplines, such as Phenotype, Loss function and Rhodopsin, Retinal Rod Photoreceptor Cells.
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